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The duality of the inflammatory response to traumatic brain injury

Molecular neurobiology, 2001-08, Vol.24 (1-3), p.169-181

2001

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Sensory cortex underpinnings of traumatic brain injury deficits

PloS one, 2012-12, Vol.7 (12), p.e52169-e52169

2012

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The role of markers of inflammation in traumatic brain injury

Frontiers in neurology, 2013-01, Vol.4, p.18-18

2013

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Involvement of Pro- and Anti-Inflammatory Cytokines and Chemokines in the Pathophysiology of Traumatic Brain Injury

Neurotherapeutics, 2010-01, Vol.7 (1), p.22-30

2010

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Role of chemokines in CNS health and pathology: a focus on the CCL2 CCR2 and CXCL8 CXCR2 networks

Journal of Cerebral Blood Flow & Metabolism, 2010-03, Vol.30 (3), p.459-473

2010

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Modulation of immune response by head injury

Injury, 2007-12, Vol.38 (12), p.1392-1400

2007

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Interleukin-13 and its receptor are synaptic proteins involved in plasticity and neuroprotection

Nature communications, 2023-01, Vol.14 (1), p.200-200, Article 200

2023

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Monitoring the Neuroinflammatory Response Following Acute Brain Injury

Frontiers in neurology, 2017-07, Vol.8, p.351-351

2017

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The scavenging chemokine receptor ACKR2 has a significant impact on acute mortality rate and early lesion development after traumatic brain injury

PloS one, 2017-11, Vol.12 (11), p.e0188305

2017

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Neuronal nuclear calcium signaling suppression of microglial reactivity is mediated by osteoprotegerin after traumatic brain injury

Journal of neuroinflammation, 2022-11, Vol.19 (1), p.1-279, Article 279

2022

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EPO treatment does not alter acute serum profiles of GFAP and S100B after TBI: A brief report on the Australian EPO-TBI clinical trial

Journal of clinical neuroscience, 2020-06, Vol.76, p.5-8

2020

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Guilty Molecules, Guilty Minds? The Conflicting Roles of the Innate Immune Response to Traumatic Brain Injury

Mediators of Inflammation, 2012-01, Vol.2012 (2012), p.603-620

2012

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Role of cerebral inflammation after traumatic brain injury : A revisited concept

Shock (Augusta, Ga.), 2001-09, Vol.16 (3), p.165-177

2001

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Measurement of serum melatonin in intensive care unit patients: changes in traumatic brain injury, trauma, and medical conditions

Frontiers in neurology, 2014-01, Vol.5, p.237-237

2014

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Interleukin-6 and its soluble receptor in serum and cerebrospinal fluid after cerebral trauma

Neuroreport, 1999-02, Vol.10 (2), p.409-412

1999

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In situ detection of inflammatory mediators in post mortem human brain tissue after traumatic injury

Journal of neurotrauma, 2010-03, Vol.27 (3), p.497-507

2010

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Interleukin-6 released in human cerebrospinal fluid following traumatic brain injury may trigger nerve growth factor production in astrocytes

Brain research, 1996-03, Vol.713 (1), p.143-152

1996

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The complexity of neuroinflammation consequent to traumatic brain injury: from research evidence to potential treatments

Acta neuropathologica, 2019-05, Vol.137 (5), p.731-755

2019

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Anti-lysophosphatidic acid antibodies improve traumatic brain injury outcomes

Journal of neuroinflammation, 2014-02, Vol.11 (1), p.37-37

2014

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Prolonged intrathecal release of soluble Fas following severe traumatic brain injury in humans

Journal of neuroimmunology, 2002, Vol.122 (1), p.167-174

2002

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sICAM-1 and TNF-α induce MIP-2 with distinct kinetics in astrocytes and brain microvascular endothelial cells

Journal of neuroscience research, 2000-06, Vol.60 (6), p.733-742

2000

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Met/HGFR triggers detrimental reactive microglia in TBI

Cell reports (Cambridge), 2022-12, Vol.41 (13), p.111867, Article 111867

2022

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Therapies negating neuroinflammation after brain trauma

Brain research, 2016-06, Vol.1640 (Pt A), p.36-56

2016

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Inflammatory response in acute traumatic brain injury: a double-edged sword

Current opinion in critical care, 2002-04, Vol.8 (2), p.101-105

2002

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The duality of the inflammatory response to traumatic brain injury

Sensory cortex underpinnings of traumatic brain injury deficits

The role of markers of inflammation in traumatic brain injury

Involvement of Pro- and Anti-Inflammatory Cytokines and Chemokines in the Pathophysiology of Traumatic Brain Injury

Role of chemokines in CNS health and pathology: a focus on the CCL2 CCR2 and CXCL8 CXCR2 networks

Modulation of immune response by head injury

Interleukin-13 and its receptor are synaptic proteins involved in plasticity and neuroprotection

Monitoring the Neuroinflammatory Response Following Acute Brain Injury

The scavenging chemokine receptor ACKR2 has a significant impact on acute mortality rate and early lesion development after traumatic brain injury

Neuronal nuclear calcium signaling suppression of microglial reactivity is mediated by osteoprotegerin after traumatic brain injury

EPO treatment does not alter acute serum profiles of GFAP and S100B after TBI: A brief report on the Australian EPO-TBI clinical trial

Guilty Molecules, Guilty Minds? The Conflicting Roles of the Innate Immune Response to Traumatic Brain Injury

Role of cerebral inflammation after traumatic brain injury : A revisited concept

Measurement of serum melatonin in intensive care unit patients: changes in traumatic brain injury, trauma, and medical conditions

Interleukin-6 and its soluble receptor in serum and cerebrospinal fluid after cerebral trauma

In situ detection of inflammatory mediators in post mortem human brain tissue after traumatic injury

Interleukin-6 released in human cerebrospinal fluid following traumatic brain injury may trigger nerve growth factor production in astrocytes

The complexity of neuroinflammation consequent to traumatic brain injury: from research evidence to potential treatments

Anti-lysophosphatidic acid antibodies improve traumatic brain injury outcomes

Prolonged intrathecal release of soluble Fas following severe traumatic brain injury in humans

sICAM-1 and TNF-α induce MIP-2 with distinct kinetics in astrocytes and brain microvascular endothelial cells

Met/HGFR triggers detrimental reactive microglia in TBI

Therapies negating neuroinflammation after brain trauma

Inflammatory response in acute traumatic brain injury: a double-edged sword

Targeted therapeutic mild hypercapnia after cardiac arrest: A phase II multi-centre randomised controlled trial (the CCC trial)

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