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Characterising effects of impact velocity on brain and behaviour in a model of diffuse traumatic axonal injury

Neuroscience, 2013-09, Vol.248, p.17-29

2013

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Animal models of traumatic brain injury: Is there an optimal model to reproduce human brain injury in the laboratory?

Injury, 2010-07, Vol.41, p.S10-S13

2010

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The duality of the inflammatory response to traumatic brain injury

Molecular neurobiology, 2001-08, Vol.24 (1-3), p.169-181

2001

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Neurotransmitters in cerebrospinal fluid reflect pathological activity

European journal of clinical investigation, 1997-12, Vol.27 (12), p.1038-1043

1997

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Experimental Closed Head Injury: Analysis of Neurological Outcome, Blood-Brain Barrier Dysfunction, Intracranial Neutrophil Infiltration, and Neuronal Cell Death in Mice Deficient in Genes for Pro-Inflammatory Cytokines

Journal of cerebral blood flow and metabolism, 2000-02, Vol.20 (2), p.369-380

2000

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Cell activation and inflammatory response following traumatic axonal injury in the rat

Neuroreport, 2000-08, Vol.11 (11), p.2587-2590

2000

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TGF-β is elevated in the CSF of patients with severe traumatic brain injuries and parallels blood-brain barrier function

Journal of neurotrauma, 1999-07, Vol.16 (7), p.617-628

1999

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Role of cerebral inflammation after traumatic brain injury : A revisited concept

Shock (Augusta, Ga.), 2001-09, Vol.16 (3), p.165-177

2001

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Experimental Axonal Injury Triggers Interleukin-6 mRNA, Protein Synthesis and Release Into Cerebrospinal Fluid

Journal of cerebral blood flow and metabolism, 1999-02, Vol.19 (2), p.184-194

1999

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Traumatic Brain Injury Increases β‐Amyloid Peptide 1‐42 in Cerebrospinal Fluid

Journal of neurochemistry, 1998-12, Vol.71 (6), p.2505-2509

1998

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Early management of severe traumatic brain injury

The Lancet (British edition), 2012-09, Vol.380 (9847), p.1088-1098

2012

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The role of markers of inflammation in traumatic brain injury

Frontiers in neurology, 2013-01, Vol.4, p.18-18

2013

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Involvement of Pro- and Anti-Inflammatory Cytokines and Chemokines in the Pathophysiology of Traumatic Brain Injury

Neurotherapeutics, 2010-01, Vol.7 (1), p.22-30

2010

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IL-10 levels in cerebrospinal fluid and serum of patients with severe traumatic brain injury: relationship to IL-6, TNF- alpha , TGF- beta 1 and blood-brain barrier function

Journal of neuroimmunology, 1999-11, Vol.101 (2), p.211-221

1999

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Interleukin-6 and its soluble receptor in serum and cerebrospinal fluid after cerebral trauma

Neuroreport, 1999-02, Vol.10 (2), p.409-412

1999

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S-100β reflects the extent of injury and outcome, whereas neuronal specific enolase is a better indicator of neuroinflammation in patients with severe traumatic brain injury

Journal of neurotrauma, 2001-05, Vol.18 (5), p.491-498

2001

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Cytokines and neuropathology

Trends in pharmacological sciences (Regular ed.), 1992, Vol.13 (7), p.286-291

1992

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Macrophage- and astrocyte-derived transforming growth factor β as a mediator of central nervous system dysfunction in acquired immune deficiency syndrome

The Journal of experimental medicine, 1991-04, Vol.173 (4), p.981-991

1991

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Soluble ICAM-1 in CSF coincides with the extent of cerebral damage in patients with severe traumatic brain injury

Journal of neurotrauma, 1998-06, Vol.15 (6), p.399-409

1998

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Intrathecal levels of complement-derived soluble membrane attack complex (sc5b-9) correlate with blood-brain barrier dysfunction in patients with traumatic brain injury

Journal of neurotrauma, 2001-08, Vol.18 (8), p.773-781

2001

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The role of the complement system in traumatic brain injury

Brain Research Reviews, 1998-08, Vol.27 (3), p.243-256

1998

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Interleukin-8 released into the cerebrospinal fluid after brain injury is associated with blood-brain barrier dysfunction and nerve growth factor production

Journal of cerebral blood flow and metabolism, 1997-03, Vol.17 (3), p.280-289

1997

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Interleukin-6 released in human cerebrospinal fluid following traumatic brain injury may trigger nerve growth factor production in astrocytes

Brain research, 1996-03, Vol.713 (1), p.143-152

1996

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Thiopental attenuates energetic impairment but fails to normalize cerebrospinal fluid glutamate in brain-injured patients

Critical care medicine, 1999-07, Vol.27 (7), p.1351-1357

1999

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Erythropoietin improves motor and cognitive deficit, axonal pathology, and neuroinflammation in a combined model of diffuse traumatic brain injury and hypoxia, in association with upregulation of the erythropoietin receptor

Journal of neuroinflammation, 2013-12, Vol.10 (1), p.156-156, Article 926

2013

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Characterising effects of impact velocity on brain and behaviour in a model of diffuse traumatic axonal injury

Animal models of traumatic brain injury: Is there an optimal model to reproduce human brain injury in the laboratory?

The duality of the inflammatory response to traumatic brain injury

Neurotransmitters in cerebrospinal fluid reflect pathological activity

Experimental Closed Head Injury: Analysis of Neurological Outcome, Blood-Brain Barrier Dysfunction, Intracranial Neutrophil Infiltration, and Neuronal Cell Death in Mice Deficient in Genes for Pro-Inflammatory Cytokines

Cell activation and inflammatory response following traumatic axonal injury in the rat

TGF-β is elevated in the CSF of patients with severe traumatic brain injuries and parallels blood-brain barrier function

Role of cerebral inflammation after traumatic brain injury : A revisited concept

Experimental Axonal Injury Triggers Interleukin-6 mRNA, Protein Synthesis and Release Into Cerebrospinal Fluid

Traumatic Brain Injury Increases β‐Amyloid Peptide 1‐42 in Cerebrospinal Fluid

Early management of severe traumatic brain injury

The role of markers of inflammation in traumatic brain injury

Involvement of Pro- and Anti-Inflammatory Cytokines and Chemokines in the Pathophysiology of Traumatic Brain Injury

IL-10 levels in cerebrospinal fluid and serum of patients with severe traumatic brain injury: relationship to IL-6, TNF- alpha , TGF- beta 1 and blood-brain barrier function

Interleukin-6 and its soluble receptor in serum and cerebrospinal fluid after cerebral trauma

S-100β reflects the extent of injury and outcome, whereas neuronal specific enolase is a better indicator of neuroinflammation in patients with severe traumatic brain injury

Cytokines and neuropathology

Macrophage- and astrocyte-derived transforming growth factor β as a mediator of central nervous system dysfunction in acquired immune deficiency syndrome

Soluble ICAM-1 in CSF coincides with the extent of cerebral damage in patients with severe traumatic brain injury

Intrathecal levels of complement-derived soluble membrane attack complex (sc5b-9) correlate with blood-brain barrier dysfunction in patients with traumatic brain injury

The role of the complement system in traumatic brain injury

Interleukin-8 released into the cerebrospinal fluid after brain injury is associated with blood-brain barrier dysfunction and nerve growth factor production

Interleukin-6 released in human cerebrospinal fluid following traumatic brain injury may trigger nerve growth factor production in astrocytes

Thiopental attenuates energetic impairment but fails to normalize cerebrospinal fluid glutamate in brain-injured patients

Erythropoietin improves motor and cognitive deficit, axonal pathology, and neuroinflammation in a combined model of diffuse traumatic brain injury and hypoxia, in association with upregulation of the erythropoietin receptor

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