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Current knowledge on the genetics of incident venous thrombosis

Journal of thrombosis and haemostasis, 2013-06, Vol.11, p.111-121

2013

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High post‐treatment platelet reactivity identified low‐responders to dual antiplatelet therapy at increased risk of recurrent cardiovascular events after stenting for acute coronary syndrome

Journal of thrombosis and haemostasis, 2006-03, Vol.4 (3), p.542-549

2006

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Endothelial cell markers and the risk of coronary heart disease: The prospective epidemiological study of myocardial infarction (PRIME) study

Circulation (New York, N.Y.), 2004-03, Vol.109 (11), p.1343-1348

2004

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Single nucleotide polymorphisms in an intergenic chromosome 2q region associated with tissue factor pathway inhibitor plasma levels and venous thromboembolism

Journal of thrombosis and haemostasis, 2016-10, Vol.14 (10), p.1960-1970

2016

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Genetic risk factors for venous thrombosis in women using combined oral contraceptives: update of the PILGRIM study

Clinical genetics, 2017-01, Vol.91 (1), p.131-136

2017

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Plasminogen activator inhibitor‐1, inflammation, obesity, insulin resistance and vascular risk

Journal of thrombosis and haemostasis, 2003-07, Vol.1 (7), p.1575-1579

2003

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Is there still room for additional common susceptibility alleles for venous thromboembolism?

Journal of thrombosis and haemostasis, 2016-09, Vol.14 (9), p.1798-1802

2016

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Impact on venous thrombosis risk of newly discovered gene variants associated with FVIII and VWF plasma levels

Journal of thrombosis and haemostasis, 2011-01, Vol.9 (1), p.229-231

2011

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Haemostatic factors and the risk of cardiovascular death in patients with coronary artery disease: the AtheroGene study

Arteriosclerosis, thrombosis, and vascular biology, 2006-12, Vol.26 (12), p.2793

2006

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Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Arteriosclerosis, thrombosis, and vascular biology, 2007-10, Vol.27 (10), p.2250-2257

2007

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Low-grade inflammation may play a role in the etiology of the metabolic syndrome in patients with coronary heart disease: the HIFMECH study

Metabolism, clinical and experimental, 2004-07, Vol.53 (7), p.852-857

2004

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Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Arteriosclerosis, thrombosis, and vascular biology, 2002-05, Vol.22 (5), p.867-873

2002

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Polymorphisms of the tumor necrosis factor-alpha (TNF) and the TNF-alpha converting enzyme (TACE/ADAM17) genes in relation to cardiovascular mortality: the AtheroGene study

Journal of molecular medicine (Berlin, Germany), 2008-10, Vol.86 (10), p.1153-1161

2008

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Lack of association of non‐synonymous FUT2 and ALPL polymorphisms with venous thrombosis

Journal of thrombosis and haemostasis, 2012-08, Vol.10 (8), p.1693-1695

2012

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A multi‐stage multi‐design strategy provides strong evidence that the BAI3 locus is associated with early‐onset venous thromboembolism

Journal of thrombosis and haemostasis, 2010-12, Vol.8 (12), p.2671-2679

2010

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Influence of PAI-1 on adipose tissue growth and metabolic parameters in a murine model of diet-induced obesity

Arteriosclerosis, thrombosis, and vascular biology, 2000-04, Vol.20 (4), p.1150-1154

2000

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Genetic variation in F3 (tissue factor) and the risk of incident venous thrombosis: meta‐analysis of eight studies

Journal of thrombosis and haemostasis, 2012-04, Vol.10 (4), p.719-722

2012

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Biological and genetic factors influencing plasma factor VIII levels in a healthy family population: results from the Stanislas cohort

British journal of haematology, 2005-01, Vol.128 (1), p.91-99

2005

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The missing heritability of venous thrombosis: what about factor V Leiden heterogeneity?

Journal of thrombosis and haemostasis, 2018-11, Vol.16 (11), p.2125-2127

2018

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Plasma TAFI antigen variations in healthy subjects

Thrombosis and haemostasis, 2000-06, Vol.83 (6), p.902-905

2000

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Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Arteriosclerosis, thrombosis, and vascular biology, 2000-09, Vol.20 (9), p.2156-2161

2000

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Heparin-induced thrombocytopenia: Update

La revue de medecine interne, 2022-01, Vol.43 (1), p.18-25

2022

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TLR4 Asp299Gly, CD14 C-260T, plasma levels of the soluble receptor CD14 and the risk of coronary heart disease: The PRIME Study

European journal of human genetics : EJHG, 2004-12, Vol.12 (12), p.1041-1049

2004

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Genetic polymorphisms and coronary artery disease in the south of France

Thrombosis and haemostasis, 2000-02, Vol.83 (2), p.212-216

2000

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PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

Arteriosclerosis, thrombosis, and vascular biology, 1999-05, Vol.19 (5), p.1361-1365

1999

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Current knowledge on the genetics of incident venous thrombosis

High post‐treatment platelet reactivity identified low‐responders to dual antiplatelet therapy at increased risk of recurrent cardiovascular events after stenting for acute coronary syndrome

Endothelial cell markers and the risk of coronary heart disease: The prospective epidemiological study of myocardial infarction (PRIME) study

Single nucleotide polymorphisms in an intergenic chromosome 2q region associated with tissue factor pathway inhibitor plasma levels and venous thromboembolism

Genetic risk factors for venous thrombosis in women using combined oral contraceptives: update of the PILGRIM study

Plasminogen activator inhibitor‐1, inflammation, obesity, insulin resistance and vascular risk

Is there still room for additional common susceptibility alleles for venous thromboembolism?

Impact on venous thrombosis risk of newly discovered gene variants associated with FVIII and VWF plasma levels

Haemostatic factors and the risk of cardiovascular death in patients with coronary artery disease: the AtheroGene study

Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Low-grade inflammation may play a role in the etiology of the metabolic syndrome in patients with coronary heart disease: the HIFMECH study

Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Polymorphisms of the tumor necrosis factor-alpha (TNF) and the TNF-alpha converting enzyme (TACE/ADAM17) genes in relation to cardiovascular mortality: the AtheroGene study

Lack of association of non‐synonymous FUT2 and ALPL polymorphisms with venous thrombosis

A multi‐stage multi‐design strategy provides strong evidence that the BAI3 locus is associated with early‐onset venous thromboembolism

Influence of PAI-1 on adipose tissue growth and metabolic parameters in a murine model of diet-induced obesity

Genetic variation in F3 (tissue factor) and the risk of incident venous thrombosis: meta‐analysis of eight studies

Biological and genetic factors influencing plasma factor VIII levels in a healthy family population: results from the Stanislas cohort

The missing heritability of venous thrombosis: what about factor V Leiden heterogeneity?

Plasma TAFI antigen variations in healthy subjects

Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Heparin-induced thrombocytopenia: Update

TLR4 Asp299Gly, CD14 C-260T, plasma levels of the soluble receptor CD14 and the risk of coronary heart disease: The PRIME Study

Genetic polymorphisms and coronary artery disease in the south of France

PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

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