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A novel panel of α-synuclein antibodies reveal distinctive staining profiles in synucleinopathies

PloS one, 2017-09, Vol.12 (9), p.e0184731-e0184731

2017

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Remodeling Alzheimer-amyloidosis models by seeding

Molecular neurodegeneration, 2021-02, Vol.16 (1), p.8-8, Article 8

2021

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Novel monoclonal antibodies targeting the microtubule-binding domain of human tau

PloS one, 2018-04, Vol.13 (4), p.e0195211-e0195211

2018

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Cytosolic proteins lose solubility as amyloid deposits in a transgenic mouse model of Alzheimer-type amyloidosis

Human molecular genetics, 2013-07, Vol.22 (14), p.2765-2774

2013

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Generation and characterization of new monoclonal antibodies targeting the PHF1 and AT8 epitopes on human tau

Acta neuropathologica communications, 2017-07, Vol.5 (1), p.58-58, Article 58

2017

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A C1qTNF3 collagen domain fusion chaperones diverse secreted proteins and anti-Aβ scFvs: Applications for gene therapies

Molecular therapy. Methods & clinical development, 2023-12, Vol.31, p.101146-101146, Article 101146

2023

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Aß40 displays amyloidogenic properties in the non-transgenic mouse brain but does not exacerbate Aß42 toxicity in Drosophila

Alzheimer's research & therapy, 2020-10, Vol.12 (1), p.132-12, Article 132

2020

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Divergent effects of the H50Q and G51D SNCA mutations on the aggregation of α‐synuclein

Journal of neurochemistry, 2014-12, Vol.131 (6), p.859-867

2014

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The role of cannabinoid receptor 2 in microglial clearance of human tau

Alzheimer's & dementia, 2021-12, Vol.17, p.e054361-n/a

2021

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Short Aβ peptides attenuate Aβ42 toxicity in vivo

The Journal of experimental medicine, 2018-01, Vol.215 (1), p.283-301

2018

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TLR5 decoy receptor as a novel anti-amyloid therapeutic for Alzheimer's disease

The Journal of experimental medicine, 2018-09, Vol.215 (9), p.2247-2264

2018

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Reversible pathologic and cognitive phenotypes in an inducible model of Alzheimer-amyloidosis

The Journal of neuroscience, 2013-02, Vol.33 (9), p.3765-3779

2013

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An anti-CRF antibody suppresses the HPA axis and reverses stress-induced phenotypes

The Journal of experimental medicine, 2019-11, Vol.216 (11), p.2479-2491

2019

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Amyloid-β(1−42) Rapidly Forms Protofibrils and Oligomers by Distinct Pathways in Low Concentrations of Sodium Dodecylsulfate

Biochemistry (Easton), 2007-10, Vol.46 (43), p.12451-12462

2007

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Steroids as γ‐secretase modulators

The FASEB journal, 2013-09, Vol.27 (9), p.3775-3785

2013

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Novel Monoclonal Antibody Specific toward Amyloid-β Binds to a Unique Epitope within the N-Terminal Region

Antibodies (Basel), 2024-08, Vol.13 (3), p.68

2024

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Overlapping profiles of Aβ peptides in the Alzheimer's disease and pathological aging brains

Alzheimer's research & therapy, 2012-05, Vol.4 (3), p.18-18

2012

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Biophysical Analyses of Synthetic Amyloid-β(1−42) Aggregates before and after Covalent Cross-Linking. Implications for Deducing the Structure of Endogenous Amyloid-β Oligomers

Biochemistry (Easton), 2009-12, Vol.48 (49), p.11796-11806

2009

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Secondary Structure and Interfacial Aggregation of Amyloid-β(1−40) on Sodium Dodecyl Sulfate Micelles

Biochemistry (Easton), 2006-07, Vol.45 (28), p.8639-8648

2006

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Retention in Endoplasmic Reticulum 1 (RER1) Modulates Amyloid-β (Aβ) Production by Altering Trafficking of γ-Secretase and Amyloid Precursor Protein (APP)

The Journal of biological chemistry, 2012-11, Vol.287 (48), p.40629-40640

2012

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Soluble brain homogenates from diverse human and mouse sources preferentially seed diffuse Aβ plaque pathology when injected into newborn mouse hosts

Free Neuropathology, 2022-01, Vol.3 (9)

2022

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EVIDENCE TO SUPPORT γ-SECRETASE MODULATORS: SHORT Aβ PEPTIDES ARE PROTECTIVE IN VIVO

Alzheimer's & dementia, 2016-07, Vol.12 (7), p.P466-P466

2016

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Rationally designed dehydroalanine (ΔAla)-containing peptides inhibit amyloid-β (Aβ) peptide aggregation

Biopolymers, 2009-06, Vol.91 (6), p.456-465

2009

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Chimeric Phagocytic Receptors (CPR) to target Alzheimer’s disease pathology

Alzheimer's & dementia, 2022-12, Vol.18 (S10), p.n/a

2022

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A novel panel of α-synuclein antibodies reveal distinctive staining profiles in synucleinopathies

Remodeling Alzheimer-amyloidosis models by seeding

Novel monoclonal antibodies targeting the microtubule-binding domain of human tau

Cytosolic proteins lose solubility as amyloid deposits in a transgenic mouse model of Alzheimer-type amyloidosis

Generation and characterization of new monoclonal antibodies targeting the PHF1 and AT8 epitopes on human tau

A C1qTNF3 collagen domain fusion chaperones diverse secreted proteins and anti-Aβ scFvs: Applications for gene therapies

Aß40 displays amyloidogenic properties in the non-transgenic mouse brain but does not exacerbate Aß42 toxicity in Drosophila

Divergent effects of the H50Q and G51D SNCA mutations on the aggregation of α‐synuclein

The role of cannabinoid receptor 2 in microglial clearance of human tau

Short Aβ peptides attenuate Aβ42 toxicity in vivo

TLR5 decoy receptor as a novel anti-amyloid therapeutic for Alzheimer's disease

Reversible pathologic and cognitive phenotypes in an inducible model of Alzheimer-amyloidosis

An anti-CRF antibody suppresses the HPA axis and reverses stress-induced phenotypes

Amyloid-β(1−42) Rapidly Forms Protofibrils and Oligomers by Distinct Pathways in Low Concentrations of Sodium Dodecylsulfate

Steroids as γ‐secretase modulators

Novel Monoclonal Antibody Specific toward Amyloid-β Binds to a Unique Epitope within the N-Terminal Region

Overlapping profiles of Aβ peptides in the Alzheimer's disease and pathological aging brains

Biophysical Analyses of Synthetic Amyloid-β(1−42) Aggregates before and after Covalent Cross-Linking. Implications for Deducing the Structure of Endogenous Amyloid-β Oligomers

Secondary Structure and Interfacial Aggregation of Amyloid-β(1−40) on Sodium Dodecyl Sulfate Micelles

Retention in Endoplasmic Reticulum 1 (RER1) Modulates Amyloid-β (Aβ) Production by Altering Trafficking of γ-Secretase and Amyloid Precursor Protein (APP)

Integrative proteomics identifies a conserved Aβ amyloid responsome, novel plaque proteins, and pathology modifiers in Alzheimer’s disease

Soluble brain homogenates from diverse human and mouse sources preferentially seed diffuse Aβ plaque pathology when injected into newborn mouse hosts

EVIDENCE TO SUPPORT γ-SECRETASE MODULATORS: SHORT Aβ PEPTIDES ARE PROTECTIVE IN VIVO

Rationally designed dehydroalanine (ΔAla)-containing peptides inhibit amyloid-β (Aβ) peptide aggregation

Chimeric Phagocytic Receptors (CPR) to target Alzheimer’s disease pathology

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