UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Defects in Galactose Metabolism and Glycoconjugate Biosynthesis in a UDP-Glucose Pyrophosphorylase-Deficient Cell Line Are Reversed by Adding Galactose to the Growth Medium

International journal of molecular sciences, 2020-03, Vol.21 (6), p.2028

2020

Volltextzugriff (PDF)

Myc is required for activation of the ATM-dependent checkpoints in response to DNA damage

PloS one, 2010-01, Vol.5 (1), p.e8924

2010

Volltextzugriff (PDF)

A bacterial cytotoxin identifies the RhoA exchange factor Net1 as a key effector in the response to DNA damage

PloS one, 2008-05, Vol.3 (5), p.e2254

2008

Volltextzugriff (PDF)

The Haemophilus ducreyi cytolethal distending toxin induces DNA double‐strand breaks and promotes ATM‐dependent activation of RhoA

Cellular microbiology, 2003-10, Vol.5 (10), p.695-707

2003

Volltextzugriff (PDF)

Cellular internalization of cytolethal distending toxin: a new end to a known pathway

Cellular microbiology, 2005-07, Vol.7 (7), p.921-934

2005

Volltextzugriff (PDF)

The Haemophilus ducreyi Cytolethal Distending Toxin Induces Cell Cycle Arrest and Apoptosis via the DNA Damage Checkpoint Pathways

The Journal of biological chemistry, 2001-02, Vol.276 (7), p.5296-5302

2001

Volltextzugriff (PDF)

The Haemophilus ducreyi cytolethal distending toxin activates sensors of DNA damage and repair complexes in proliferating and non‐proliferating cells

Cellular microbiology, 2002-02, Vol.4 (2), p.87-99

2002

Volltextzugriff (PDF)

Cellular Internalization of Cytolethal Distending Toxin from Haemophilus ducreyi

Infection and Immunity, 2000-12, Vol.68 (12), p.6903-6911

2000

Volltextzugriff (PDF)

A Cellular Deficiency of Gangliosides Causes Hypersensitivity to Clostridium perfringens Phospholipase C

The Journal of biological chemistry, 2005-07, Vol.280 (29), p.26680-26689

2005

Volltextzugriff (PDF)

R-Ras Glucosylation and Transient RhoA Activation Determine the Cytopathic Effect Produced by Toxin B Variants from Toxin A-negative Strains of Clostridium difficile

The Journal of biological chemistry, 2003-03, Vol.278 (10), p.7956-7963

2003

Volltextzugriff (PDF)

A Cellular UDP-glucose Deficiency Causes Overexpression of Glucose/Oxygen-regulated Proteins Independent of the Endoplasmic Reticulum Stress Elements

The Journal of biological chemistry, 2004-05, Vol.279 (21), p.21724-21731

2004

Volltextzugriff (PDF)

Divergent Roles for Ras and Rap in the Activation of p38 Mitogen-activated Protein Kinase by Interleukin-1

The Journal of biological chemistry, 2000-03, Vol.275 (11), p.7818-7825

2000

Volltextzugriff (PDF)

Ras, Rap, and Rac Small GTP-binding Proteins Are Targets for Clostridium sordellii Lethal Toxin Glucosylation

The Journal of biological chemistry, 1996-04, Vol.271 (17), p.10217-10224

1996

Volltextzugriff (PDF)

Effects of Clostridium perfringens phospholipase C in mammalian cells

Anaerobe, 2004-04, Vol.10 (2), p.115-123

2004

Volltextzugriff (PDF)

Cellular UDP-Glucose Deficiency Caused by a Single Point Mutation in the UDP-Glucose Pyrophosphorylase Gene

The Journal of biological chemistry, 1997-09, Vol.272 (38), p.23784-23791

1997

Volltextzugriff (PDF)

The projection structure of Perfringolysin O ( Clostridium perfringens θ-toxin)

FEBS letters, 1993-03, Vol.319 (1), p.125-127

1993

Volltextzugriff (PDF)

Cytolethal distending toxins

Reviews of physiology, biochemistry and pharmacology, 2005-01, Vol.152, p.111

2005

Volltextzugriff (PDF)

Is there a risk of cancer development after Campylobacter infection?

Scandinavian journal of gastroenterology, 2010-08, Vol.45 (7-8), p.893-897

2010

Volltextzugriff (PDF)

A novel mode of translocation for cytolethal distending toxin

Biochimica et biophysica acta, 2009-03, Vol.1793 (3), p.489-495

2009

Volltextzugriff (PDF)

UDP-glucose Deficiency Causes Hypersensitivity to the Cytotoxic Effect of Clostridium perfringens Phospholipase C

The Journal of biological chemistry, 1998-09, Vol.273 (38), p.24433-24438

1998

Volltextzugriff (PDF)

UDP-Glucose Deficiency in a Mutant Cell Line Protects against Glucosyltransferase Toxins from Clostridium difficile and Clostridium sordellii

The Journal of biological chemistry, 1996-03, Vol.271 (12), p.6925-6932

1996

Volltextzugriff (PDF)

The cytolethal distending toxins induce DNA damage and cell cycle arrest

Toxicon (Oxford), 2001-11, Vol.39 (11), p.1729-1736

2001

Volltextzugriff (PDF)

Defects in galactose metabolism and glycoconjugate biosynthesis in UDP‐glucose pyrophosphorylase‐deficient fibroblasts are reversed by supplementing the cell growth medium with galactose

The FASEB journal, 2012-04, Vol.26 (S1)

2012

Volltextzugriff (PDF)

GTPases of the Rho Subfamily Are Required for Brucella abortus Internalization in Nonprofessional Phagocytes

The Journal of biological chemistry, 2001-11, Vol.276 (48), p.44435-44443

2001

Volltextzugriff (PDF)

Large clostridial cytotoxins — a family of glycosyltransferases modifying small GTP-binding proteins

Trends in microbiology (Regular ed.), 1996-10, Vol.4 (10), p.375-382

1996

Volltextzugriff (PDF)

1 – 25 von 104

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(99)
Mit Volltextzugriff
(90)

Publikationsform

Artikel
(103)
Buchkapitel
(1)

Sprache

Englisch
(104)
Japanisch
(16)

Erscheinungsjahr

n.n

Thema

Science & Technology
(65)
Life Sciences & Biomedicine
(63)
Animals
(44)
Humans
(42)
Medicin Och Hälsovetenskap
(40)
Biochemistry & Molecular Biology
(34)
Biological And Medical Sciences
(29)
Microbiology
(27)
Fundamental And Applied Biological Sciences. Psychology
(23)
Cell Line
(21)
Bacterial Toxins - Pharmacology
(19)
Bacterial Toxins - Toxicity
(18)
Bacteriology
(18)
Cells, Cultured
(18)
Bacterial Toxins - Metabolism
(16)
Mice
(15)
Clostridium Difficile
(13)
Cricetinae
(13)
Bacterial Proteins
(12)
Cell Biology
(12)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Defects in Galactose Metabolism and Glycoconjugate Biosynthesis in a UDP-Glucose Pyrophosphorylase-Deficient Cell Line Are Reversed by Adding Galactose to the Growth Medium

Myc is required for activation of the ATM-dependent checkpoints in response to DNA damage

A bacterial cytotoxin identifies the RhoA exchange factor Net1 as a key effector in the response to DNA damage

The Haemophilus ducreyi cytolethal distending toxin induces DNA double‐strand breaks and promotes ATM‐dependent activation of RhoA

Cellular internalization of cytolethal distending toxin: a new end to a known pathway

The Haemophilus ducreyi Cytolethal Distending Toxin Induces Cell Cycle Arrest and Apoptosis via the DNA Damage Checkpoint Pathways

The Haemophilus ducreyi cytolethal distending toxin activates sensors of DNA damage and repair complexes in proliferating and non‐proliferating cells

Cellular Internalization of Cytolethal Distending Toxin from Haemophilus ducreyi

A Cellular Deficiency of Gangliosides Causes Hypersensitivity to Clostridium perfringens Phospholipase C

R-Ras Glucosylation and Transient RhoA Activation Determine the Cytopathic Effect Produced by Toxin B Variants from Toxin A-negative Strains of Clostridium difficile

A Cellular UDP-glucose Deficiency Causes Overexpression of Glucose/Oxygen-regulated Proteins Independent of the Endoplasmic Reticulum Stress Elements

Divergent Roles for Ras and Rap in the Activation of p38 Mitogen-activated Protein Kinase by Interleukin-1

Ras, Rap, and Rac Small GTP-binding Proteins Are Targets for Clostridium sordellii Lethal Toxin Glucosylation

Effects of Clostridium perfringens phospholipase C in mammalian cells

Cellular UDP-Glucose Deficiency Caused by a Single Point Mutation in the UDP-Glucose Pyrophosphorylase Gene

The projection structure of Perfringolysin O ( Clostridium perfringens θ-toxin)

Cytolethal distending toxins

Is there a risk of cancer development after Campylobacter infection?

A novel mode of translocation for cytolethal distending toxin

UDP-glucose Deficiency Causes Hypersensitivity to the Cytotoxic Effect of Clostridium perfringens Phospholipase C

UDP-Glucose Deficiency in a Mutant Cell Line Protects against Glucosyltransferase Toxins from Clostridium difficile and Clostridium sordellii

The cytolethal distending toxins induce DNA damage and cell cycle arrest

Defects in galactose metabolism and glycoconjugate biosynthesis in UDP‐glucose pyrophosphorylase‐deficient fibroblasts are reversed by supplementing the cell growth medium with galactose

GTPases of the Rho Subfamily Are Required for Brucella abortus Internalization in Nonprofessional Phagocytes

Large clostridial cytotoxins — a family of glycosyltransferases modifying small GTP-binding proteins

Suchergebnisse filtern