UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Cell death and differentiation, 2017-06, Vol.24 (6), p.1017-1028

2017

Volltextzugriff (PDF)

Conditional cell reprogramming for modeling host‐virus interactions and human viral diseases

Journal of medical virology, 2020-11, Vol.92 (11), p.2440-2452

2020

Volltextzugriff (PDF)

Crosstalk of moderate ROS and PARP‐1 contributes to sustainable proliferation of conditionally reprogrammed keratinocytes

Journal of biochemical and molecular toxicology, 2023-02, Vol.37 (2), p.e23262-n/a

2023

Volltextzugriff (PDF)

Delta 133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Cell death and differentiation, 2017-06, Vol.24 (6), p.1017-1028

2017

Volltextzugriff (PDF)

p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes

The Journal of clinical investigation, 2013-12, Vol.123 (12), p.5247-5257

2013

Volltextzugriff (PDF)

p53 isoforms Delta 133p53 and p53 beta are endogenous regulators of replicative cellular senescence

Nature cell biology, 2009-09, Vol.11 (9), p.1135-1142

2009

Volltextzugriff (PDF)

p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence

Nature cell biology, 2009-09, Vol.11 (9), p.1135-1142

2009

Volltextzugriff (PDF)

Fidelity of a PDX-CR model for bladder cancer

Biochemical and biophysical research communications, 2019-09, Vol.517 (1), p.49-56

2019

Volltextzugriff (PDF)

Δ133p53α, a natural p53 isoform, contributes to conditional reprogramming and long-term proliferation of primary epithelial cells

Cell death & disease, 2018-07, Vol.9 (7), p.750-14, Article 750

2018

Volltextzugriff (PDF)

Positive feedback between p53 and TRF2 during telomere-damage signalling and cellular senescence

Nature cell biology, 2010-12, Vol.12 (12), p.1205-1212

2010

Volltextzugriff (PDF)

Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescence

Nature communications, 2014-08, Vol.5 (1), p.4706-4706, Article 4706

2014

Volltextzugriff (PDF)

Cover Image, Volume 92, Number 11, November 2020

Journal of medical virology, 2020-11, Vol.92 (11), p.i-i

2020

Volltextzugriff (PDF)

Abstract A27: Fidelity of a PDX-CR model for bladder cancer

Clinical cancer research, 2020-08, Vol.26 (15_Supplement), p.A27-A27

2020

Volltextzugriff (PDF)

Abstract 922: Delta133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.922-922

2017

Volltextzugriff (PDF)

[Delta]133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Cell death and differentiation, 2017-06, Vol.24 (6), p.1017

2017

Volltextzugriff (PDF)

Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Cell death and differentiation, 2017-06, Vol.24 (6), p.1017-1028

2017

Volltextzugriff (PDF)

Abstract PR08: Autophagic degradation of Δ133p53 during replicative cellular senescence: An isoform-specific protein degradation mechanism for p53

Cancer research (Chicago, Ill.), 2013-10, Vol.73 (19_Supplement), p.PR08-PR08

2013

Volltextzugriff (PDF)

Isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes

The Journal of clinical investigation, 2013-12, Vol.123 (12), p.5247

2013

Volltextzugriff (PDF)

Abstract 3199: p53 represses TRF2 through E3 ubiquitin ligase Siah-1: Feedback regulation in telomere-initiated damage signaling

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.3199-3199

2010

Volltextzugriff (PDF)

Stability of the Age Distribution of Measles Cases Over Time During Outbreaks in Bangladesh, 2004-2006

The Journal of infectious diseases, 2011-07, Vol.204 (suppl_1), p.S414-S420

2011

Volltextzugriff (PDF)

Abstract 2915: p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.2915-2915

2010

Volltextzugriff (PDF)

p53 isoforms [Delta]133p53 and p53[beta] are endogenous regulators of replicative cellular senescence

Nature cell biology, 2009-09, Vol.11 (9), p.1135

2009

Volltextzugriff (PDF)

Autophagic degradation of the inhibitory p53 isoform [Delta]133p53[alpha] as a regulatory mechanism for p53-mediated senescence

Nature communications, 2014-08, Vol.5, p.4706

2014

Volltextzugriff (PDF)

Stability of the Age Distribution of Measles Cases Over Time During Outbreaks in Bangladesh, 2004-2006: Global Progress Toward Measles Eradication and Prevention of Rubella and Congenital Rubella Syndrome

The Journal of infectious diseases, 2011, Vol.204

2011

Volltextzugriff (PDF)

p53 isoforms Delta133p53 and p53beta are endogenous regulators of replicative cellular senescence

Nature cell biology, 2009-09, Vol.11 (9), p.1135

2009

Volltextzugriff (PDF)

1 – 25 von 88

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(64)
Mit Volltextzugriff
(73)

Publikationsform

Sprache

Englisch
(88)

Erscheinungsjahr

n.n

Thema

Science & Technology
(39)
Life Sciences & Biomedicine
(27)
Humans
(15)
Animals
(13)
Female
(9)
Male
(9)
Bangladesh
(8)
Life Sciences
(7)
Technology
(7)
Biomedical And Life Sciences
(6)
Cell Biology
(6)
Physical Sciences
(6)
Tropical Medicine
(6)
Adult
(5)
Bangladesh - Epidemiology
(5)
Infectious Diseases
(5)
Mortality
(5)
Public, Environmental & Occupational Health
(5)
Senescence
(5)
Child, Preschool
(4)

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)
... seit 90 Tagen
(3)

Menü

Weitere Dienste

Einstellungen

Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Conditional cell reprogramming for modeling host‐virus interactions and human viral diseases

Crosstalk of moderate ROS and PARP‐1 contributes to sustainable proliferation of conditionally reprogrammed keratinocytes

Delta 133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes

p53 isoforms Delta 133p53 and p53 beta are endogenous regulators of replicative cellular senescence

p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence

Fidelity of a PDX-CR model for bladder cancer

Δ133p53α, a natural p53 isoform, contributes to conditional reprogramming and long-term proliferation of primary epithelial cells

Positive feedback between p53 and TRF2 during telomere-damage signalling and cellular senescence

Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescence

Cover Image, Volume 92, Number 11, November 2020

Abstract A27: Fidelity of a PDX-CR model for bladder cancer

Abstract 922: Delta133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

[Delta]133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells

Abstract PR08: Autophagic degradation of Δ133p53 during replicative cellular senescence: An isoform-specific protein degradation mechanism for p53

Isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes

Abstract 3199: p53 represses TRF2 through E3 ubiquitin ligase Siah-1: Feedback regulation in telomere-initiated damage signaling

Stability of the Age Distribution of Measles Cases Over Time During Outbreaks in Bangladesh, 2004-2006

Abstract 2915: p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence

p53 isoforms [Delta]133p53 and p53[beta] are endogenous regulators of replicative cellular senescence

Autophagic degradation of the inhibitory p53 isoform [Delta]133p53[alpha] as a regulatory mechanism for p53-mediated senescence

Stability of the Age Distribution of Measles Cases Over Time During Outbreaks in Bangladesh, 2004-2006: Global Progress Toward Measles Eradication and Prevention of Rubella and Congenital Rubella Syndrome

p53 isoforms Delta133p53 and p53beta are endogenous regulators of replicative cellular senescence

Suchergebnisse filtern