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The Replication Checkpoint Prevents Two Types of Fork Collapse without Regulating Replisome Stability

Molecular cell, 2015-09, Vol.59 (6), p.998-1010

2015

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Protection of abasic sites during DNA replication by a stable thiazolidine protein-DNA cross-link

Nature structural & molecular biology, 2019-07, Vol.26 (7), p.613-618

2019

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HMCES Maintains Genome Integrity by Shielding Abasic Sites in Single-Strand DNA

Cell, 2019-01, Vol.176 (1-2), p.144-153.e13

2019

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Functional Analysis of the Replication Fork Proteome Identifies BET Proteins as PCNA Regulators

Cell reports (Cambridge), 2019-09, Vol.28 (13), p.3497-3509.e4

2019

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A Synthetic Lethal Screen Identifies DNA Repair Pathways that Sensitize Cancer Cells to Combined ATR Inhibition and Cisplatin Treatments

PloS one, 2015-05, Vol.10 (5), p.e0125482

2015

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The HSV-1 exonuclease, UL12, stimulates recombination by a single strand annealing mechanism

PLoS pathogens, 2012-08, Vol.8 (8), p.e1002862-e1002862

2012

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An Intrinsically Disordered Region of the DNA Repair Protein Nbs1 Is a Species-Specific Barrier to Herpes Simplex Virus 1 in Primates

Cell host & microbe, 2016-08, Vol.20 (2), p.178-188

2016

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Efficient Herpes Simplex Virus 1 Replication Requires Cellular ATR Pathway Proteins

Journal of Virology, 2013-01, Vol.87 (1), p.531-542

2013

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Herpes simplex virus type 1 single strand DNA binding protein and helicase/primase complex disable cellular ATR signaling

PLoS pathogens, 2013-10, Vol.9 (10), p.e1003652-e1003652

2013

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DNA Mismatch Repair Proteins Are Required for Efficient Herpes Simplex Virus 1 Replication

Journal of Virology, 2011-12, Vol.85 (23), p.12241-12253

2011

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ATR Pathway Inhibition Is Synthetically Lethal in Cancer Cells with ERCC1 Deficiency

Cancer research (Chicago, Ill.), 2014-05, Vol.74 (10), p.2835-2845

2014

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ATR and ATRIP Are Recruited to Herpes Simplex Virus Type 1 Replication Compartments Even though ATR Signaling Is Disabled

Journal of Virology, 2010-12, Vol.84 (23), p.12152-12164

2010

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Structure of the herpes simplex virus 1 genome: manipulation of nicks and gaps can abrogate infectivity and alter the cellular DNA damage response

Journal of virology, 2014-09, Vol.88 (17), p.10146-10156

2014

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Structural Characterization of Interaction between Human Ubiquitin-specific Protease 7 and Immediate-Early Protein ICP0 of Herpes Simplex Virus-1

The Journal of biological chemistry, 2015-09, Vol.290 (38), p.22907-22918

2015

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Enhancer of Rudimentary Homolog Affects the Replication Stress Response through Regulation of RNA Processing

Molecular and cellular biology, 2015-09, Vol.35 (17), p.2979-2990

2015

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A whole genome RNAi screen identifies replication stress response genes

DNA repair, 2015-11, Vol.35, p.55-62

2015

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Lethal H5N1 influenza viruses are not resistant to interferon action in human, simian, porcine or chicken cells

Nature medicine, 2012-10, Vol.18 (10), p.1456-1457

2012

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Influenza virus subpopulations: exchange of lethal H5N1 virus NS for H1N1 virus NS triggers de novo generation of defective-interfering particles and enhances interferon-inducing particle efficiency

Journal of interferon & cytokine research, 2013-03, Vol.33 (3), p.99-107

2013

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Protection of abasic sites during DNA replication by a stable thiazolidine protein–DNA crosslink

Acta crystallographica. Section A, Foundations and advances, 2019-07, Vol.75 (a1), p.a205-a205

2019

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Herpes Simplex Virus type 1 replication proteins disable ATR signaling by binding to substrates that would normally recruit 9–1‐1 and topBP1 to activate ATR

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Herpes Simplex Virus: Manipulating DNA Damage Response Pathways

The FASEB journal, 2012-04, Vol.26 (S1)

2012

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Herpes Simplex Virus type 1 infection disrupts the ATR‐mediated damage response

The FASEB journal, 2010-04, Vol.24 (S1), p.492.12-492.12

2010

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Herpes Simplex Virus Type 1 Single Strand DNA Binding Protein and Helicase/Primase Complex Disable Cellular ATR Signaling: e1003652

PLoS pathogens, 2013-10, Vol.9 (10)

2013

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A Synthetic Lethal Screen Identifies DNA Repair Pathways that Sensitize Cancer Cells to Combined ATR Inhibition and Cisplatin Treatments: e0125482

PloS one, 2015-05, Vol.10 (5)

2015

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EFFECTS OF COSTIMULATION ON INFLUENZA SPECIFIC CD8+ T CELLS DURING LATE ANTIGEN PRESENTATION

The FASEB journal, 2008-03, Vol.22 (S1)

2008

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The Replication Checkpoint Prevents Two Types of Fork Collapse without Regulating Replisome Stability

Protection of abasic sites during DNA replication by a stable thiazolidine protein-DNA cross-link

HMCES Maintains Genome Integrity by Shielding Abasic Sites in Single-Strand DNA

Functional Analysis of the Replication Fork Proteome Identifies BET Proteins as PCNA Regulators

A Synthetic Lethal Screen Identifies DNA Repair Pathways that Sensitize Cancer Cells to Combined ATR Inhibition and Cisplatin Treatments

The HSV-1 exonuclease, UL12, stimulates recombination by a single strand annealing mechanism

An Intrinsically Disordered Region of the DNA Repair Protein Nbs1 Is a Species-Specific Barrier to Herpes Simplex Virus 1 in Primates

Efficient Herpes Simplex Virus 1 Replication Requires Cellular ATR Pathway Proteins

Herpes simplex virus type 1 single strand DNA binding protein and helicase/primase complex disable cellular ATR signaling

DNA Mismatch Repair Proteins Are Required for Efficient Herpes Simplex Virus 1 Replication

ATR Pathway Inhibition Is Synthetically Lethal in Cancer Cells with ERCC1 Deficiency

ATR and ATRIP Are Recruited to Herpes Simplex Virus Type 1 Replication Compartments Even though ATR Signaling Is Disabled

Structure of the herpes simplex virus 1 genome: manipulation of nicks and gaps can abrogate infectivity and alter the cellular DNA damage response

Structural Characterization of Interaction between Human Ubiquitin-specific Protease 7 and Immediate-Early Protein ICP0 of Herpes Simplex Virus-1

Enhancer of Rudimentary Homolog Affects the Replication Stress Response through Regulation of RNA Processing

A whole genome RNAi screen identifies replication stress response genes

Lethal H5N1 influenza viruses are not resistant to interferon action in human, simian, porcine or chicken cells

Influenza virus subpopulations: exchange of lethal H5N1 virus NS for H1N1 virus NS triggers de novo generation of defective-interfering particles and enhances interferon-inducing particle efficiency

Protection of abasic sites during DNA replication by a stable thiazolidine protein–DNA crosslink

Herpes Simplex Virus type 1 replication proteins disable ATR signaling by binding to substrates that would normally recruit 9–1‐1 and topBP1 to activate ATR

Herpes Simplex Virus: Manipulating DNA Damage Response Pathways

Herpes Simplex Virus type 1 infection disrupts the ATR‐mediated damage response

Herpes Simplex Virus Type 1 Single Strand DNA Binding Protein and Helicase/Primase Complex Disable Cellular ATR Signaling: e1003652

A Synthetic Lethal Screen Identifies DNA Repair Pathways that Sensitize Cancer Cells to Combined ATR Inhibition and Cisplatin Treatments: e0125482

EFFECTS OF COSTIMULATION ON INFLUENZA SPECIFIC CD8+ T CELLS DURING LATE ANTIGEN PRESENTATION

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