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Advances in the Evaluation of Gastrointestinal Absorption Considering the Mucus Layer

Pharmaceutics, 2023-11, Vol.15 (12), p.2714

2023

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CD44 Facilitates Epithelial-to-Mesenchymal Transition Phenotypic Change at Acquisition of Resistance to EGFR Kinase Inhibitors in Lung Cancer

Molecular cancer therapeutics, 2018-10, Vol.17 (10), p.2257-2265

2018

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Small cell lung cancer transformation and T790M mutation: complimentary roles in acquired resistance to kinase inhibitors in lung cancer

Scientific reports, 2015-09, Vol.5 (1), p.14447-14447, Article 14447

2015

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MET gene exon 14 deletion created using the CRISPR/Cas9 system enhances cellular growth and sensitivity to a MET inhibitor

Lung cancer (Amsterdam, Netherlands), 2015-12, Vol.90 (3), p.590-597

2015

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Oncogene swap as a novel mechanism of acquired resistance to epidermal growth factor receptor‐tyrosine kinase inhibitor in lung cancer

Cancer science, 2016-04, Vol.107 (4), p.461-468

2016

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Heterogeneity in resistance mechanisms causes shorter duration of epidermal growth factor receptor kinase inhibitor treatment in lung cancer

Lung cancer (Amsterdam, Netherlands), 2016-01, Vol.91, p.36-40

2016

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Effect of dasatinib on EMT-mediated-mechanism of resistance against EGFR inhibitors in lung cancer cells

Lung cancer (Amsterdam, Netherlands), 2017-02, Vol.104, p.85-90

2017

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MEK inhibitors against MET-amplified non-small cell lung cancer

International journal of oncology, 2016-12, Vol.49 (6), p.2236-2244

2016

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Clinical, Pathological, and Molecular Features of Lung Adenocarcinomas with AXL Expression

PloS one, 2016-04, Vol.11 (4), p.e0154186-e0154186

2016

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Liver metastases 2 years after resection of a very-low-risk duodenal gastrointestinal stromal tumor: a case report

Surgical case reports, 2022-10, Vol.8 (1), p.195-195, Article 195

2022

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Collateral chemoresistance to anti-microtubule agents in a lung cancer cell line with acquired resistance to erlotinib

PloS one, 2015-04, Vol.10 (4), p.e0123901-e0123901

2015

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Acquired resistance mechanisms to tyrosine kinase inhibitors in lung cancer with activating epidermal growth factor receptor mutation—diversity, ductility, and destiny

Cancer and metastasis reviews, 2012-12, Vol.31 (3-4), p.807-814

2012

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Recent evidence, advances, and current practices in surgical treatment of lung cancer

Respiratory investigation, 2014-11, Vol.52 (6), p.322-329

2014

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EGFR Exon 18 Mutations in Lung Cancer: Molecular Predictors of Augmented Sensitivity to Afatinib or Neratinib as Compared with First- or Third-Generation TKIs

Clinical cancer research, 2015-12, Vol.21 (23), p.5305-5313

2015

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The insulin‐like growth factor 1 receptor causes acquired resistance to erlotinib in lung cancer cells with the wild‐type epidermal growth factor receptor

International journal of cancer, 2014-08, Vol.135 (4), p.1002-1006

2014

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Impact of bevacizumab in combination with erlotinib on EGFR‐mutated non–small cell lung cancer xenograft models with T790M mutation or MET amplification

International journal of cancer, 2016-02, Vol.138 (4), p.1024-1032

2016

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Diphenhydramine transport by pH-dependent tertiary amine transport system in Caco-2 cells

American journal of physiology: Gastrointestinal and liver physiology, 2000-04, Vol.278 (4), p.G563-G569

2000

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Comparison of various EGFR tyrosine kinase inhibitors (TKIs) for tumors with exon 18 mutations of the EGFR gene

Annals of oncology, 2015-11, Vol.26, p.vii83

2015

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Functional Analyses of Mutations in Receptor Tyrosine Kinase Genes in Non-Small Cell Lung Cancer: Double-Edged Sword of DDR2

Clinical cancer research, 2016-07, Vol.22 (14), p.3663-3671

2016

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Transport characteristics of diphenhydramine in human intestinal epithelial Caco-2 cells: contribution of pH-dependent transport system

The Journal of pharmacology and experimental therapeutics, 1999-07, Vol.290 (1), p.388

1999

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Topics on transporter-mediated renal excretion and drug–drug interaction

Folia Pharmacologica Japonica, 2010, Vol.135(1), pp.30-33

Mizuuchi, Hiroshi

2010

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P1.02-047 Effect of Dasatinib on EMT-Mediated-Mechanism of Resistance against EGFR Inhibitors in Lung Cancer Cells: Topic: Driver Genes in NSCLC, Resistance, and Other

Journal of thoracic oncology, 2017-01, Vol.12 (1), p.S515-S516

2017

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P1.02-047 Effect of Dasatinib on EMT-Mediated-Mechanism of Resistance against EGFR Inhibitors in Lung Cancer Cells

Journal of thoracic oncology, 2017-01, Vol.12 (1), p.S515-S516

2017

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An activating ALK gene mutation in ALK IHC-positive/FISH-negative non-small cell lung cancer

Annals of oncology, 2015-11, Vol.26, p.vii73-vii73

2015

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CRKL amplification is rare as a mechanism for acquired resistance to kinase inhibitors in lung cancers with epidermal growth factor receptor mutation

Lung cancer (Amsterdam, Netherlands), 2014-08, Vol.85 (2), p.147-151

2014

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Advances in the Evaluation of Gastrointestinal Absorption Considering the Mucus Layer

CD44 Facilitates Epithelial-to-Mesenchymal Transition Phenotypic Change at Acquisition of Resistance to EGFR Kinase Inhibitors in Lung Cancer

Small cell lung cancer transformation and T790M mutation: complimentary roles in acquired resistance to kinase inhibitors in lung cancer

MET gene exon 14 deletion created using the CRISPR/Cas9 system enhances cellular growth and sensitivity to a MET inhibitor

Oncogene swap as a novel mechanism of acquired resistance to epidermal growth factor receptor‐tyrosine kinase inhibitor in lung cancer

Heterogeneity in resistance mechanisms causes shorter duration of epidermal growth factor receptor kinase inhibitor treatment in lung cancer

Effect of dasatinib on EMT-mediated-mechanism of resistance against EGFR inhibitors in lung cancer cells

MEK inhibitors against MET-amplified non-small cell lung cancer

Clinical, Pathological, and Molecular Features of Lung Adenocarcinomas with AXL Expression

Liver metastases 2 years after resection of a very-low-risk duodenal gastrointestinal stromal tumor: a case report

Collateral chemoresistance to anti-microtubule agents in a lung cancer cell line with acquired resistance to erlotinib

Acquired resistance mechanisms to tyrosine kinase inhibitors in lung cancer with activating epidermal growth factor receptor mutation—diversity, ductility, and destiny

Recent evidence, advances, and current practices in surgical treatment of lung cancer

EGFR Exon 18 Mutations in Lung Cancer: Molecular Predictors of Augmented Sensitivity to Afatinib or Neratinib as Compared with First- or Third-Generation TKIs

The insulin‐like growth factor 1 receptor causes acquired resistance to erlotinib in lung cancer cells with the wild‐type epidermal growth factor receptor

Impact of bevacizumab in combination with erlotinib on EGFR‐mutated non–small cell lung cancer xenograft models with T790M mutation or MET amplification

Diphenhydramine transport by pH-dependent tertiary amine transport system in Caco-2 cells

Comparison of various EGFR tyrosine kinase inhibitors (TKIs) for tumors with exon 18 mutations of the EGFR gene

Functional Analyses of Mutations in Receptor Tyrosine Kinase Genes in Non-Small Cell Lung Cancer: Double-Edged Sword of DDR2

Transport characteristics of diphenhydramine in human intestinal epithelial Caco-2 cells: contribution of pH-dependent transport system

Topics on transporter-mediated renal excretion and drug–drug interaction

P1.02-047 Effect of Dasatinib on EMT-Mediated-Mechanism of Resistance against EGFR Inhibitors in Lung Cancer Cells: Topic: Driver Genes in NSCLC, Resistance, and Other

P1.02-047 Effect of Dasatinib on EMT-Mediated-Mechanism of Resistance against EGFR Inhibitors in Lung Cancer Cells

An activating ALK gene mutation in ALK IHC-positive/FISH-negative non-small cell lung cancer

CRKL amplification is rare as a mechanism for acquired resistance to kinase inhibitors in lung cancers with epidermal growth factor receptor mutation

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