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B Cells Produce the Tissue-Protective Protein RELMα during Helminth Infection, which Inhibits IL-17 Expression and Limits Emphysema

Cell reports (Cambridge), 2018-12, Vol.25 (10), p.2775-2783.e3

2018

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Natural killer cells, glutathione, cytokines, and innate immunity against Mycobacterium tuberculosis

Journal of interferon & cytokine research, 2008-03, Vol.28 (3), p.153

2008

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Glutathione and growth inhibition of Mycobacterium tuberculosis in healthy and HIV infected subjects

AIDS research and therapy, 2006-02, Vol.3 (1), p.5-5, Article 5

2006

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An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infection

Nature medicine, 2012-01, Vol.18 (2), p.260-266

2012

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Neutrophils prime a long-lived effector macrophage phenotype that mediates accelerated helminth expulsion

Nature immunology, 2014-10, Vol.15 (10), p.938

2014

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Sterile particle-induced inflammation is mediated by macrophages releasing IL-33 through a Bruton's tyrosine kinase-dependent pathway

Nature materials, 2019-03, Vol.18 (3), p.289-297

2019

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B lymphocytes limit the severity of emphysema caused by N. brasiliensis infection in mice (INC4P.332)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.125-125.11

2015

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Helminth-induced B lymphocytes mediate control of emphysema through production of RELMa

The Journal of immunology (1950), 2017-05, Vol.198 (1_Supplement), p.138-138.9

2017

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Helminth-induced B lymphocytes mediate control of IL-17 and emphysema through their production of RELMα

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.52-52.26

2018

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A2B Adenosine Receptor Induces Protective Antihelminth Type 2 Immune Responses

Cell host & microbe, 2014-03, Vol.15 (3), p.339-350

2014

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IL-4R signaling in B cells mediates control of IL-17A dependent emphysema triggered by N. brasiliensis infection

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.196-196.7

2016

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Sterile type 2 inflammation triggered by microparticles is abrogated by inhibition of IL33 through blockade of Bruton’s tyrosine kinase

The Journal of immunology (1950), 2017-05, Vol.198 (1_Supplement), p.70-70.3

2017

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CoCr micro-particles induce a robust type 2 inflammation in knee synovial tissues leading to fibrosis and tissue damage

The Journal of immunology (1950), 2017-05, Vol.198 (1_Supplement), p.70-70.2

2017

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Primed effector macrophages mediate rapid worm expulsion after N. brasiliensis infection (P4247)

The Journal of immunology (1950), 2013-05, Vol.190 (1_Supplement), p.130-130.23

2013

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Blockade of either SYK or BTK signaling pathways inhibits microparticle-induced type 2 inflammatory response (IRM11P.628)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.132-132.7

2015

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Repeated low dose injection of cobalt chromium microparticles in a mouse knee model induces robust inflammation with cartilage and bone loss (INC1P.345)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.54-54.2

2015

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CoCr microparticles induce a robust type 2 innate immune response that is dependent on SYK and BTK signaling

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.203-203.21

2016

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Neutrophils prime a long-lived effector macrophage phenotype that mediates accelerated helminth expulsion (INC7P.423)

The Journal of immunology (1950), 2014-05, Vol.192 (1_Supplement), p.186-186.24

2014

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Cobalt-chrome micro-particles induce a robust MyD88-independent type 2 immune response (IRM7P.495)

The Journal of immunology (1950), 2014-05, Vol.192 (1_Supplement), p.126-126.20

2014

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Cobalt-chrome micro-particles induce a robust MyD88-independent, Caspase1-dependent type 2 immune response (P1266)

The Journal of immunology (1950), 2013-05, Vol.190 (1_Supplement), p.116-116.11

2013

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Glutathione levels and immune responses in tuberculosis patients

Microbial pathogenesis, 2008-03, Vol.44 (3), p.255-261

2008

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IL-4Rα signaling may enhance resolution of parasite-induced lung damage by inhibiting inflammation and mediating acute wound healing (37.8)

The Journal of immunology (1950), 2010-04, Vol.184 (1_Supplement), p.37-37.8

2010

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An essential role for [T.sub.H] 2-type responses in limiting acute tissue damage during experimental helminth infection

Nature medicine, 2012-02, Vol.18 (2), p.260

2012

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An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infection

Nature medicine, 2012-02, Vol.18 (2), p.260

2012

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An essential role for the Th2-type response in limiting tissue damage during helminth infection

Nature medicine, 2012-01, Vol.18 (2), p.260-266

2012

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B Cells Produce the Tissue-Protective Protein RELMα during Helminth Infection, which Inhibits IL-17 Expression and Limits Emphysema

Natural killer cells, glutathione, cytokines, and innate immunity against Mycobacterium tuberculosis

Glutathione and growth inhibition of Mycobacterium tuberculosis in healthy and HIV infected subjects

An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infection

Neutrophils prime a long-lived effector macrophage phenotype that mediates accelerated helminth expulsion

Sterile particle-induced inflammation is mediated by macrophages releasing IL-33 through a Bruton's tyrosine kinase-dependent pathway

B lymphocytes limit the severity of emphysema caused by N. brasiliensis infection in mice (INC4P.332)

Helminth-induced B lymphocytes mediate control of emphysema through production of RELMa

Helminth-induced B lymphocytes mediate control of IL-17 and emphysema through their production of RELMα

A2B Adenosine Receptor Induces Protective Antihelminth Type 2 Immune Responses

IL-4R signaling in B cells mediates control of IL-17A dependent emphysema triggered by N. brasiliensis infection

Sterile type 2 inflammation triggered by microparticles is abrogated by inhibition of IL33 through blockade of Bruton’s tyrosine kinase

CoCr micro-particles induce a robust type 2 inflammation in knee synovial tissues leading to fibrosis and tissue damage

Primed effector macrophages mediate rapid worm expulsion after N. brasiliensis infection (P4247)

Blockade of either SYK or BTK signaling pathways inhibits microparticle-induced type 2 inflammatory response (IRM11P.628)

Repeated low dose injection of cobalt chromium microparticles in a mouse knee model induces robust inflammation with cartilage and bone loss (INC1P.345)

CoCr microparticles induce a robust type 2 innate immune response that is dependent on SYK and BTK signaling

Neutrophils prime a long-lived effector macrophage phenotype that mediates accelerated helminth expulsion (INC7P.423)

Cobalt-chrome micro-particles induce a robust MyD88-independent type 2 immune response (IRM7P.495)

Cobalt-chrome micro-particles induce a robust MyD88-independent, Caspase1-dependent type 2 immune response (P1266)

Glutathione levels and immune responses in tuberculosis patients

IL-4Rα signaling may enhance resolution of parasite-induced lung damage by inhibiting inflammation and mediating acute wound healing (37.8)

An essential role for [T.sub.H] 2-type responses in limiting acute tissue damage during experimental helminth infection

An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infection

An essential role for the Th2-type response in limiting tissue damage during helminth infection

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