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The Initial Hepatitis B Virus-Hepatocyte Genomic Integrations and Their Role in Hepatocellular Oncogenesis

International journal of molecular sciences, 2023-10, Vol.24 (19), p.14849

Michalak, Tomasz I

2023

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Diverse Virus and Host-Dependent Mechanisms Influence the Systemic and Intrahepatic Immune Responses in the Woodchuck Model of Hepatitis B

Frontiers in immunology, 2020-05, Vol.11, p.853

Michalak, Tomasz I

2020

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Hepadnaviral Lymphotropism and Its Relevance to HBV Persistence and Pathogenesis

Frontiers in microbiology, 2021-08, Vol.12, p.695384-695384

2021

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Differential Expression of Woodchuck Toll-Like Receptors 1-10 in Distinct Forms of Infection and Stages of Hepatitis in Experimental Hepatitis B Virus Infection

Frontiers in microbiology, 2018-12, Vol.9, p.3007-3007

2018

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Identification of antibodies cross-reactive with woodchuck immune cells and activation of virus-specific and global cytotoxic T cell responses by anti-PD-1 and anti-PD-L1 in experimental chronic hepatitis B and persistent occult hepadnaviral infection

Frontiers in microbiology, 2022-12, Vol.13, p.1011070-1011070

2022

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Primary seronegative but molecularly evident hepadnaviral infection engages liver and induces hepatocarcinoma in the woodchuck model of hepatitis B

PLoS pathogens, 2014-08, Vol.10 (8), p.e1004332-e1004332

2014

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Persistent occult hepatitis B virus infection: experimental findings and clinical implications

World journal of gastroenterology : WJG, 2007-11, Vol.13 (43), p.5682

2007

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Reactivation of hepatitis B after liver transplantation: Current knowledge, molecular mechanisms and implications in management

World journal of hepatology, 2018-03, Vol.10 (3), p.352-370

2018

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Hepatitis C Virus Infection of Human T Lymphocytes Is Mediated by CD5

Journal of Virology, 2012-04, Vol.86 (7), p.3723-3735

2012

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Hepatitis C Virus Persistence after Spontaneous or Treatment-Induced Resolution of Hepatitis C

Journal of Virology, 2004-06, Vol.78 (11), p.5867-5874

2004

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Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C

PloS one, 2015-10, Vol.10 (10), p.e0140312-e0140312

2015

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Hepatitis C Virus Replicates in the Same Immune Cell Subsets in Chronic Hepatitis C and Occult Infection

Gastroenterology (New York, N.Y. 1943), 2008-03, Vol.134 (3), p.812-822

2008

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Intrahepatic Expression of Genes Affiliated with Innate and Adaptive Immune Responses Immediately after Invasion and during Acute Infection with Woodchuck Hepadnavirus

Journal of Virology, 2008-09, Vol.82 (17), p.8579-8591

2008

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Differential expression of candidate virus receptors in human T lymphocytes prone or resistant to infection with patient-derived hepatitis C virus

PloS one, 2013-04, Vol.8 (4), p.e62159-e62159

2013

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Hepatitis C virus persisting after clinically apparent sustained virological response to antiviral therapy retains infectivity in vitro

Hepatology (Baltimore, Md.), 2009-05, Vol.49 (5), p.1431-1441

2009

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Hepatocyte cytotoxicity is facilitated by asialoglycoprotein receptor

Hepatology (Baltimore, Md.), 2011-09, Vol.54 (3), p.1043-1050

2011

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Persistence of hepatitis C virus during and after otherwise clinically successful treatment of chronic hepatitis C with standard pegylated interferon α-2b and ribavirin therapy

PloS one, 2013-11, Vol.8 (11), p.e80078

2013

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Repeated Exposure to Trace Amounts of Woodchuck Hepadnavirus Induces Molecularly Evident Infection and Virus-Specific T Cell Response in the Absence of Serological Infection Markers and Hepatitis

Journal of Virology, 2013-01, Vol.87 (2), p.1035-1048

2013

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Primary Occult Hepadnavirus Infection Induces Virus-Specific T-Cell and Aberrant Cytokine Responses in the Absence of Antiviral Antibody Reactivity in the Woodchuck Model of Hepatitis B Virus Infection

Journal of Virology, 2009-04, Vol.83 (8), p.3861-3876

2009

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Occult persistence and lymphotropism of hepatitis C virus infection

World journal of gastroenterology : WJG, 2008-05, Vol.14 (18), p.2789-2793

2008

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Immune cell subsets and their gene expression profiles from human PBMC isolated by Vacutainer Cell Preparation Tube (CPT™) and standard density gradient

BMC immunology, 2015-08, Vol.16 (1), p.48-48, Article 48

2015

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Authentic Patient-Derived Hepatitis C Virus Infects and Productively Replicates in Primary CD4 + and CD8 + T Lymphocytes In Vitro

Journal of virology, 2018-02, Vol.92 (3)

2018

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Kinetics of DNA damage repair response accompanying initial hepadnavirus-host genomic integration in woodchuck hepatitis virus infection of hepatocyte

Cancer genetics, 2020-06, Vol.244, p.1-10

2020

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Low Doses of Hepadnavirus Induce Infection of the Lymphatic System That Does Not Engage the Liver

Journal of Virology, 2004-02, Vol.78 (4), p.1730-1738

2004

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Repeated Passage of Wild-Type Woodchuck Hepatitis Virus in Lymphoid Cells Does Not Generate Cell Type-Specific Variants or Alter Virus Infectivity

Journal of Virology, 2008-08, Vol.82 (15), p.7540-7550

2008

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The Initial Hepatitis B Virus-Hepatocyte Genomic Integrations and Their Role in Hepatocellular Oncogenesis

Diverse Virus and Host-Dependent Mechanisms Influence the Systemic and Intrahepatic Immune Responses in the Woodchuck Model of Hepatitis B

Hepadnaviral Lymphotropism and Its Relevance to HBV Persistence and Pathogenesis

Differential Expression of Woodchuck Toll-Like Receptors 1-10 in Distinct Forms of Infection and Stages of Hepatitis in Experimental Hepatitis B Virus Infection

Identification of antibodies cross-reactive with woodchuck immune cells and activation of virus-specific and global cytotoxic T cell responses by anti-PD-1 and anti-PD-L1 in experimental chronic hepatitis B and persistent occult hepadnaviral infection

Primary seronegative but molecularly evident hepadnaviral infection engages liver and induces hepatocarcinoma in the woodchuck model of hepatitis B

Persistent occult hepatitis B virus infection: experimental findings and clinical implications

Reactivation of hepatitis B after liver transplantation: Current knowledge, molecular mechanisms and implications in management

Hepatitis C Virus Infection of Human T Lymphocytes Is Mediated by CD5

Hepatitis C Virus Persistence after Spontaneous or Treatment-Induced Resolution of Hepatitis C

Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C

Hepatitis C Virus Replicates in the Same Immune Cell Subsets in Chronic Hepatitis C and Occult Infection

Intrahepatic Expression of Genes Affiliated with Innate and Adaptive Immune Responses Immediately after Invasion and during Acute Infection with Woodchuck Hepadnavirus

Differential expression of candidate virus receptors in human T lymphocytes prone or resistant to infection with patient-derived hepatitis C virus

Hepatitis C virus persisting after clinically apparent sustained virological response to antiviral therapy retains infectivity in vitro

Hepatocyte cytotoxicity is facilitated by asialoglycoprotein receptor

Persistence of hepatitis C virus during and after otherwise clinically successful treatment of chronic hepatitis C with standard pegylated interferon α-2b and ribavirin therapy

Repeated Exposure to Trace Amounts of Woodchuck Hepadnavirus Induces Molecularly Evident Infection and Virus-Specific T Cell Response in the Absence of Serological Infection Markers and Hepatitis

Primary Occult Hepadnavirus Infection Induces Virus-Specific T-Cell and Aberrant Cytokine Responses in the Absence of Antiviral Antibody Reactivity in the Woodchuck Model of Hepatitis B Virus Infection

Occult persistence and lymphotropism of hepatitis C virus infection

Immune cell subsets and their gene expression profiles from human PBMC isolated by Vacutainer Cell Preparation Tube (CPT™) and standard density gradient

Authentic Patient-Derived Hepatitis C Virus Infects and Productively Replicates in Primary CD4 + and CD8 + T Lymphocytes In Vitro

Kinetics of DNA damage repair response accompanying initial hepadnavirus-host genomic integration in woodchuck hepatitis virus infection of hepatocyte

Low Doses of Hepadnavirus Induce Infection of the Lymphatic System That Does Not Engage the Liver

Repeated Passage of Wild-Type Woodchuck Hepatitis Virus in Lymphoid Cells Does Not Generate Cell Type-Specific Variants or Alter Virus Infectivity

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