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Recent Patents in Allergy/Immunology: Use of arginase inhibitors in the treatment of asthma and allergic rhinitis

Allergy (Copenhagen), 2019-06, Vol.74 (6), p.1206-1208

2019

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD

Respiratory research, 2006-05, Vol.7 (1), p.73-73, Article 73

2006

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Arginase: a key enzyme in the pathophysiology of allergic asthma opening novel therapeutic perspectives

British journal of pharmacology, 2009-10, Vol.158 (3), p.652-664

2009

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Regulation of airway inflammation and remodeling by muscarinic receptors: Perspectives on anticholinergic therapy in asthma and COPD

Life sciences (1973), 2012-11, Vol.91 (21-22), p.1126-1133

2012

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Bronchoconstriction Induces TGF-β Release and Airway Remodelling in Guinea Pig Lung Slices

PloS one, 2013-06, Vol.8 (6), p.e65580

2013

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Protective Effects of Tiotropium Bromide in the Progression of Airway Smooth Muscle Remodeling

American journal of respiratory and critical care medicine, 2005-05, Vol.171 (10), p.1096-1102

2005

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Muscarinic M₃ receptors contribute to allergen-induced airway remodeling in mice

American journal of respiratory cell and molecular biology, 2014-04, Vol.50 (4), p.690-698

2014

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Combination therapy of tiotropium and ciclesonide attenuates airway inflammation and remodeling in a guinea pig model of chronic asthma

Respiratory research, 2016-02, Vol.17 (14), p.13-13, Article 13

2016

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Cigarette smoke and lipopolysaccharide induce a proliferative airway smooth muscle phenotype

Respiratory research, 2010-04, Vol.11 (1), p.48-48, Article 48

2010

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Anti-inflammatory role of the cAMP effectors Epac and PKA: implications in chronic obstructive pulmonary disease

PloS one, 2012-02, Vol.7 (2), p.e31574-e31574

2012

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Epac1 and Epac2 are differentially involved in inflammatory and remodeling processes induced by cigarette smoke

The FASEB journal, 2014-11, Vol.28 (11), p.4617-4628

2014

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TAK1 plays a major role in growth factor-induced phenotypic modulation of airway smooth muscle

American journal of physiology. Lung cellular and molecular physiology, 2011-11, Vol.301 (5), p.L822-L828

2011

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The non-neuronal cholinergic system: Basic science, therapeutic implications and new perspectives

Life sciences (1973), 2012-11, Vol.91 (21-22), p.969-972

2012

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Muscarinic receptors on airway mesenchymal cells: Novel findings for an ancient target

Pulmonary pharmacology & therapeutics, 2013-02, Vol.26 (1), p.145-155

2013

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A synthetic peptide as an allosteric inhibitor of human arginase I and II

Molecular biology reports, 2021-02, Vol.48 (2), p.1959-1966

2021

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Pro-inflammatory mechanisms of muscarinic receptor stimulation in airway smooth muscle

Respiratory research, 2010-09, Vol.11 (1), p.130-130, Article 130

2010

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Arginase Inhibition Protects against Allergen-induced Airway Obstruction, Hyperresponsiveness, and Inflammation

American journal of respiratory and critical care medicine, 2008-09, Vol.178 (6), p.565-573

2008

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Muscarinic receptor stimulation augments TGF-β1-induced contractile protein expression by airway smooth muscle cells

American journal of physiology. Lung cellular and molecular physiology, 2012-10, Vol.303 (7), p.L589-L597

2012

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Epac as a novel effector of airway smooth muscle relaxation

Journal of cellular and molecular medicine, 2011-07, Vol.15 (7), p.1551-1563

2011

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Extracellular matrix proteins differentially regulate airway smooth muscle phenotype and function

American journal of physiology. Lung cellular and molecular physiology, 2007-06, Vol.292 (6), p.L1405-L1413

2007

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Role for TAK1 in cigarette smoke-induced proinflammatory signaling and IL-8 release by human airway smooth muscle cells

American journal of physiology. Lung cellular and molecular physiology, 2012-08, Vol.303 (3), p.L272-L278

2012

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Arginase strongly impairs neuronal nitric oxide-mediated airway smooth muscle relaxation in allergic asthma

Respiratory research, 2006-01, Vol.7 (1), p.6-6, Article 6

2006

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Effects of (a Combination of) the Beta2-Adrenoceptor Agonist Indacaterol and the Muscarinic Receptor Antagonist Glycopyrrolate on Intrapulmonary Airway Constriction

Cells (Basel, Switzerland), 2021-05, Vol.10 (5), p.1237

2021

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The inhaled Rho kinase inhibitor Y-27632 protects against allergen-induced acute bronchoconstriction, airway hyperresponsiveness, and inflammation

American journal of physiology. Lung cellular and molecular physiology, 2008-07, Vol.295 (1), p.L214-L219

2008

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cAMP inhibits modulation of airway smooth muscle phenotype via the exchange protein activated by cAMP (Epac) and protein kinase A

British journal of pharmacology, 2011-01, Vol.162 (1), p.193-209

2011

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Muscle, Smooth - Drug Effects
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Recent Patents in Allergy/Immunology: Use of arginase inhibitors in the treatment of asthma and allergic rhinitis

Muscarinic receptor signaling in the pathophysiology of asthma and COPD

Arginase: a key enzyme in the pathophysiology of allergic asthma opening novel therapeutic perspectives

Regulation of airway inflammation and remodeling by muscarinic receptors: Perspectives on anticholinergic therapy in asthma and COPD

Bronchoconstriction Induces TGF-β Release and Airway Remodelling in Guinea Pig Lung Slices

Protective Effects of Tiotropium Bromide in the Progression of Airway Smooth Muscle Remodeling

Muscarinic M₃ receptors contribute to allergen-induced airway remodeling in mice

Combination therapy of tiotropium and ciclesonide attenuates airway inflammation and remodeling in a guinea pig model of chronic asthma

Cigarette smoke and lipopolysaccharide induce a proliferative airway smooth muscle phenotype

Anti-inflammatory role of the cAMP effectors Epac and PKA: implications in chronic obstructive pulmonary disease

Epac1 and Epac2 are differentially involved in inflammatory and remodeling processes induced by cigarette smoke

TAK1 plays a major role in growth factor-induced phenotypic modulation of airway smooth muscle

The non-neuronal cholinergic system: Basic science, therapeutic implications and new perspectives

Muscarinic receptors on airway mesenchymal cells: Novel findings for an ancient target

A synthetic peptide as an allosteric inhibitor of human arginase I and II

Pro-inflammatory mechanisms of muscarinic receptor stimulation in airway smooth muscle

Arginase Inhibition Protects against Allergen-induced Airway Obstruction, Hyperresponsiveness, and Inflammation

Muscarinic receptor stimulation augments TGF-β1-induced contractile protein expression by airway smooth muscle cells

Epac as a novel effector of airway smooth muscle relaxation

Extracellular matrix proteins differentially regulate airway smooth muscle phenotype and function

Role for TAK1 in cigarette smoke-induced proinflammatory signaling and IL-8 release by human airway smooth muscle cells

Arginase strongly impairs neuronal nitric oxide-mediated airway smooth muscle relaxation in allergic asthma

Effects of (a Combination of) the Beta2-Adrenoceptor Agonist Indacaterol and the Muscarinic Receptor Antagonist Glycopyrrolate on Intrapulmonary Airway Constriction

The inhaled Rho kinase inhibitor Y-27632 protects against allergen-induced acute bronchoconstriction, airway hyperresponsiveness, and inflammation

cAMP inhibits modulation of airway smooth muscle phenotype via the exchange protein activated by cAMP (Epac) and protein kinase A

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