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Inflammation‐induced lymphatic architecture and bone turnover changes are ameliorated by irisin treatment in chronic inflammatory bowel disease

The FASEB journal, 2018-09, Vol.32 (9), p.4848-4861

2018

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Adenine-induced chronic kidney disease induces a similar skeletal phenotype in male and female C57BL/6 mice with more severe deficits in cortical bone properties of male mice

PloS one, 2021-04, Vol.16 (4), p.e0250438-e0250438

2021

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The Role of Osteocytes in Inflammatory Bone Loss

Frontiers in endocrinology (Lausanne), 2019-05, Vol.10, p.285-285

2019

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Inflammatory Bowel Disease in a Rodent Model Alters Osteocyte Protein Levels Controlling Bone Turnover

Journal of bone and mineral research, 2017-04, Vol.32 (4), p.802-813

2017

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Effect of Advanced Glycation End‐Products (AGE) Lowering Drug ALT‐711 on Biochemical, Vascular, and Bone Parameters in a Rat Model of CKD‐MBD

Journal of bone and mineral research, 2020-03, Vol.35 (3), p.608-617

2020

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Cortical porosity development and progression is mitigated after etelcalcetide treatment in an animal model of chronic kidney disease

Bone (New York, N.Y.), 2022-04, Vol.157, p.116340-116340, Article 116340

2022

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Age and sex effects on FGF23-mediated response to mild phosphate challenge

Bone (New York, N.Y.), 2021-05, Vol.146, p.115885-115885, Article 115885

2021

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Exercise training modifies the bone and endocrine response to graded reductions in energy availability in skeletally mature female rodents

Frontiers in endocrinology (Lausanne), 2023-06, Vol.14, p.1141906-1141906

2023

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Single cell cortical bone transcriptomics define novel osteolineage gene sets altered in chronic kidney disease

Frontiers in endocrinology (Lausanne), 2023-01, Vol.14, p.1063083-1063083

2023

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Romosozumab rescues impaired bone mass and strength in a murine model of diabetic kidney disease

Bone Reports, 2024-06, Vol.21, p.101774-101774, Article 101774

2024

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A moderate spinal contusion injury in rats alters bone turnover both below and above the level of injury with sex-based differences apparent in long-term recovery

Bone Reports, 2024-06, Vol.21, p.101761-101761, Article 101761

2024

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Fat and Lean Mass Predict Bone Mass During Energy Restriction in Sedentary and Exercising Rodents

Frontiers in physiology, 2018-09, Vol.9, p.1346-1346

2018

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Cortical porosity occurs at varying degrees throughout the skeleton in rats with chronic kidney disease

Bone Reports, 2022-12, Vol.17, p.101612-101612, Article 101612

2022

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The Effect of Single Versus Group μCT on the Detection of Trabecular and Cortical Disease Phenotypes in Mouse Bones

JBMR plus, 2021-04, Vol.5 (4), p.e10473-n/a

2021

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The Dietary Fiber Inulin Slows Progression of Chronic Kidney Disease–Mineral Bone Disorder (CKD‐MBD) in a Rat Model of CKD

JBMR plus, 2023-12, Vol.7 (12), p.e10837-n/a

2023

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Cortical porosity is elevated after a single dose of zoledronate in two rodent models of chronic kidney disease

Bone Reports, 2022-06, Vol.16, p.101174-101174, Article 101174

2022

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NMUR1 in the NMU-Mediated Regulation of Bone Remodeling

Life (Basel, Switzerland), 2021-09, Vol.11 (10), p.1028

2021

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Non‐Additive Effects of Combined NOX1/4 Inhibition and Calcimimetic Treatment on a Rat Model of Chronic Kidney Disease‐Mineral and Bone Disorder (CKD‐MBD)

JBMR plus, 2022-03, Vol.6 (3), p.e10600-n/a

2022

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Elevations in Cortical Porosity Occur Prior to Significant Rise in Serum Parathyroid Hormone in Young Female Mice with Adenine-Induced CKD

Calcified tissue international, 2020-04, Vol.106 (4), p.392-400

2020

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Strain-specific alterations in the skeletal response to adenine-induced chronic kidney disease are associated with differences in parathyroid hormone levels

Bone (New York, N.Y.), 2021-07, Vol.148, p.115963-115963, Article 115963

2021

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A novel murine model of combined insulin-dependent diabetes and chronic kidney disease has greater skeletal detriments than either disease individually

Bone (New York, N.Y.), 2022-12, Vol.165, p.116559-116559, Article 116559

2022

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Ex vivo exposure to calcitonin or raloxifene improves mechanical properties of diseased bone through non-cell mediated mechanisms

Bone (New York, N.Y.), 2023-08, Vol.173, p.116805-116805, Article 116805

2023

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Osteocytes reflect a pro-inflammatory state following spinal cord injury in a rodent model

Bone (New York, N.Y.), 2019-03, Vol.120, p.465-475

2019

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Two Weeks of Continuous Opioid Treatment in an Adenine-Induced Mouse Model of Chronic Kidney Disease Exacerbates the Bone Inflammatory State and Increases Osteoclasts

Calcified tissue international, 2024-08, Vol.115 (2), p.174-184

2024

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Inflammation‐induced lymphatic architecture and bone turnover changes are ameliorated by irisin treatment in chronic inflammatory bowel disease

Adenine-induced chronic kidney disease induces a similar skeletal phenotype in male and female C57BL/6 mice with more severe deficits in cortical bone properties of male mice

The Role of Osteocytes in Inflammatory Bone Loss

Inflammatory Bowel Disease in a Rodent Model Alters Osteocyte Protein Levels Controlling Bone Turnover

Effect of Advanced Glycation End‐Products (AGE) Lowering Drug ALT‐711 on Biochemical, Vascular, and Bone Parameters in a Rat Model of CKD‐MBD

Cortical porosity development and progression is mitigated after etelcalcetide treatment in an animal model of chronic kidney disease

Age and sex effects on FGF23-mediated response to mild phosphate challenge

Prenatal methadone exposure disrupts behavioral development and alters motor neuron intrinsic properties and local circuitry

Exercise training modifies the bone and endocrine response to graded reductions in energy availability in skeletally mature female rodents

Single cell cortical bone transcriptomics define novel osteolineage gene sets altered in chronic kidney disease

Romosozumab rescues impaired bone mass and strength in a murine model of diabetic kidney disease

A moderate spinal contusion injury in rats alters bone turnover both below and above the level of injury with sex-based differences apparent in long-term recovery

Fat and Lean Mass Predict Bone Mass During Energy Restriction in Sedentary and Exercising Rodents

Cortical porosity occurs at varying degrees throughout the skeleton in rats with chronic kidney disease

The Effect of Single Versus Group μCT on the Detection of Trabecular and Cortical Disease Phenotypes in Mouse Bones

The Dietary Fiber Inulin Slows Progression of Chronic Kidney Disease–Mineral Bone Disorder (CKD‐MBD) in a Rat Model of CKD

Cortical porosity is elevated after a single dose of zoledronate in two rodent models of chronic kidney disease

NMUR1 in the NMU-Mediated Regulation of Bone Remodeling

Non‐Additive Effects of Combined NOX1/4 Inhibition and Calcimimetic Treatment on a Rat Model of Chronic Kidney Disease‐Mineral and Bone Disorder (CKD‐MBD)

Elevations in Cortical Porosity Occur Prior to Significant Rise in Serum Parathyroid Hormone in Young Female Mice with Adenine-Induced CKD

Strain-specific alterations in the skeletal response to adenine-induced chronic kidney disease are associated with differences in parathyroid hormone levels

A novel murine model of combined insulin-dependent diabetes and chronic kidney disease has greater skeletal detriments than either disease individually

Ex vivo exposure to calcitonin or raloxifene improves mechanical properties of diseased bone through non-cell mediated mechanisms

Osteocytes reflect a pro-inflammatory state following spinal cord injury in a rodent model

Two Weeks of Continuous Opioid Treatment in an Adenine-Induced Mouse Model of Chronic Kidney Disease Exacerbates the Bone Inflammatory State and Increases Osteoclasts

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