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Functional role of voltage gated Ca2+ channels in heart automaticity

Frontiers in physiology, 2015-02, Vol.6

2015

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Nfix: a transcription factor with an important functional role in cardiac automaticity

Acta Physiologica, 2023-10, Vol.239 (2), p.e14034-e14034

Mesirca, Pietro

2023

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Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity

Scientific reports, 2020-11, Vol.10 (1), p.18906-18906, Article 18906

2020

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A synthetic peptide that prevents cAMP regulation in mammalian hyperpolarization-activated cyclic nucleotide-gated (HCN) channels

eLife, 2018-06, Vol.7

2018

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State-of-the-Art Differentiation Protocols for Patient-Derived Cardiac Pacemaker Cells

International journal of molecular sciences, 2024-03, Vol.25 (6), p.3387

2024

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Correction to: Channelopathies of voltage-gated L-type Cav1.3/α1D and T-type Cav3.1/α1G Ca2+ channels in dysfunction of heart automaticity

Pflügers Archiv, 2020-08, Vol.472 (8), p.1103-1104

2020

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Inhibition of G protein-gated K + channels by tertiapin-Q rescues sinus node dysfunction and atrioventricular conduction in mouse models of primary bradycardia

Scientific reports, 2020-06, Vol.10 (1), p.9835-9835, Article 9835

2020

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Selective blockade of Cav1.2 (α1C) versus Cav1.3 (α1D) L-type calcium channels by the black mamba toxin calciseptine

Nature communications, 2024-01, Vol.15 (1), p.54-54, Article 54

2024

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Intrinsic Electrical Remodeling Underlies Atrioventricular Block in Athletes

Circulation research, 2021-06, Vol.129 (1), p.e1-e20

2021

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Ion channel-kinase TRPM7 is required for maintaining cardiac automaticity

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (32), p.E3037-E3046

2013

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Channelopathies of voltage-gated L-type Cav1.3/α1D and T-type Cav3.1/α1G Ca2+ channels in dysfunction of heart automaticity

Pflügers Archiv, 2020-07, Vol.472 (7), p.817-830

2020

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CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current

Scientific reports, 2017-08, Vol.7 (1), p.1-12, Article 7869

2017

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Genetic Complexity of Sinoatrial Node Dysfunction

Frontiers in genetics, 2021-04, Vol.12, p.654925-654925

2021

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Characterization of sinoatrial automaticity in Microcebus murinus to study the effect of aging on cardiac activity and the correlation with longevity

Scientific reports, 2023-02, Vol.13 (1), p.3054-3054, Article 3054

2023

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Regulation of sinus node pacemaking and atrioventricular node conduction by HCN channels in health and disease

Progress in biophysics and molecular biology, 2021-11, Vol.166, p.61-85

2021

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G protein-gated IKACh channels as therapeutic targets for treatment of sick sinus syndrome and heart block

Proceedings of the National Academy of Sciences - PNAS, 2016-02, Vol.113 (7), p.E932-E941

2016

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Editorial: Cardiac Pacemaking in Health and Disease: From Genes to Function

Frontiers in physiology, 2022-05, Vol.13, p.913506-913506

2022

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Control of heart rate by cAMP sensitivity of HCN channels

Proceedings of the National Academy of Sciences - PNAS, 2009-07, Vol.106 (29), p.12189-12194

2009

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Paradoxical Effect of Increased Diastolic Ca2+ Release and Decreased Sinoatrial Node Activity in a Mouse Model of Catecholaminergic Polymorphic Ventricular Tachycardia

Circulation (New York, N.Y.), 2012-07, Vol.126 (4), p.392-401

2012

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Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation

Nature communications, 2014-08, Vol.5 (1), p.4664-4664, Article 4664

2014

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Clock-dependent and system-driven oscillators interact in the suprachiasmatic nuclei to pace mammalian circadian rhythms

PloS one, 2017-10, Vol.12 (10), p.e0187001-e0187001

2017

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The G-protein-gated K+ channel, IKACh, is required for regulation of pacemaker activity and recovery of resting heart rate after sympathetic stimulation

The Journal of general physiology, 2013-08, Vol.142 (2), p.113-126

2013

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Distinct localization and modulation of Cav1.2 and Cav1.3 L‐type Ca2+ channels in mouse sinoatrial node

The Journal of physiology, 2012-12, Vol.590 (24), p.6327-6341

2012

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L-Type Cav1.3 Calcium Channels Are Required for Beta-Adrenergic Triggered Automaticity in Dormant Mouse Sinoatrial Pacemaker Cells

Cells (Basel, Switzerland), 2022-03, Vol.11 (7), p.1114

2022

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Functional role of voltage gated Ca2+ channels in heart automaticity

Nfix: a transcription factor with an important functional role in cardiac automaticity

Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity

A synthetic peptide that prevents cAMP regulation in mammalian hyperpolarization-activated cyclic nucleotide-gated (HCN) channels

State-of-the-Art Differentiation Protocols for Patient-Derived Cardiac Pacemaker Cells

Correction to: Channelopathies of voltage-gated L-type Cav1.3/α1D and T-type Cav3.1/α1G Ca2+ channels in dysfunction of heart automaticity

Inhibition of G protein-gated K + channels by tertiapin-Q rescues sinus node dysfunction and atrioventricular conduction in mouse models of primary bradycardia

Selective blockade of Cav1.2 (α1C) versus Cav1.3 (α1D) L-type calcium channels by the black mamba toxin calciseptine

A circadian clock in the sinus node mediates day-night rhythms in Hcn4 and heart rate

Intrinsic Electrical Remodeling Underlies Atrioventricular Block in Athletes

Ion channel-kinase TRPM7 is required for maintaining cardiac automaticity

Channelopathies of voltage-gated L-type Cav1.3/α1D and T-type Cav3.1/α1G Ca2+ channels in dysfunction of heart automaticity

CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current

Genetic Complexity of Sinoatrial Node Dysfunction

Characterization of sinoatrial automaticity in Microcebus murinus to study the effect of aging on cardiac activity and the correlation with longevity

Regulation of sinus node pacemaking and atrioventricular node conduction by HCN channels in health and disease

G protein-gated IKACh channels as therapeutic targets for treatment of sick sinus syndrome and heart block

Editorial: Cardiac Pacemaking in Health and Disease: From Genes to Function

Control of heart rate by cAMP sensitivity of HCN channels

Paradoxical Effect of Increased Diastolic Ca2+ Release and Decreased Sinoatrial Node Activity in a Mouse Model of Catecholaminergic Polymorphic Ventricular Tachycardia

Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation

Clock-dependent and system-driven oscillators interact in the suprachiasmatic nuclei to pace mammalian circadian rhythms

The G-protein-gated K+ channel, IKACh, is required for regulation of pacemaker activity and recovery of resting heart rate after sympathetic stimulation

Distinct localization and modulation of Cav1.2 and Cav1.3 L‐type Ca2+ channels in mouse sinoatrial node

L-Type Cav1.3 Calcium Channels Are Required for Beta-Adrenergic Triggered Automaticity in Dormant Mouse Sinoatrial Pacemaker Cells

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