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c-Jun, at the crossroad of the signaling network

Protein & cell, 2011-11, Vol.2 (11), p.889-898

2011

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Targeting Interactions between Fibroblasts and Macrophages to Treat Cardiac Fibrosis

Cells (Basel, Switzerland), 2024-05, Vol.13 (9), p.764

2024

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Identification of Underwater Structural Bridge Damage and BIM-Based Bridge Damage Management

Applied sciences, 2023-02, Vol.13 (3), p.1348

2023

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Eyelid Closure in Embryogenesis Is Required for Ocular Adnexa Development

Investigative ophthalmology & visual science, 2014-11, Vol.55 (11), p.7652

2014

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Corneal Wound Healing Requires IKB kinase β Signaling in Keratocytes

PloS one, 2016-03, Vol.11 (3), p.e0151869-e0151869

2016

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A BIM technology-based underwater structure damage identification and management method

Bulletin of the Polish Academy of Sciences. Technical sciences, 2023, Vol.71 (2)

2023

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Cardiac Fibrosis in Proteotoxic Cardiac Disease is Dependent Upon Myofibroblast TGF -β Signaling

Journal of the American Heart Association, 2018-10, Vol.7 (20), p.e010013-e010013

2018

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Activation of AMPK by berberine promotes adiponectin multimerization in 3T3-L1 adipocytes

FEBS letters, 2011-06, Vol.585 (12), p.1735-1740

2011

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IκB kinase β regulates epithelium migration during corneal wound healing

PloS one, 2011-01, Vol.6 (1), p.e16132

2011

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Mitogen-activated protein kinase kinase kinase 1 (MAP3K1) integrates developmental signals for eyelid closure

Proceedings of the National Academy of Sciences - PNAS, 2011-10, Vol.108 (42), p.17349-17354

2011

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Col1a2 -Deleted Mice Have Defective Type I Collagen and Secondary Reactive Cardiac Fibrosis with Altered Hypertrophic Dynamics

Cells (Basel, Switzerland), 2023-08, Vol.12 (17), p.2174

2023

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Expression of signaling components in embryonic eyelid epithelium

PloS one, 2014-02, Vol.9 (2), p.e87038

2014

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MAP3K1 function is essential for cytoarchitecture of the mouse organ of Corti and survival of auditory hair cells

Disease models & mechanisms, 2015-12, Vol.8 (12), p.1543-1553

2015

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MMI‐0100 Inhibits Cardiac Fibrosis in a Mouse Model Overexpressing Cardiac Myosin Binding Protein C

Journal of the American Heart Association, 2017-09, Vol.6 (9), p.n/a

2017

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Deciphering gene expression program of MAP3K1 in mouse eyelid morphogenesis

Developmental biology, 2013-02, Vol.374 (1), p.96-107

2013

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Loss of IκB kinase β promotes myofibroblast transformation and senescence through activation of the ROS-TGFβ autocrine loop

Protein & cell, 2016-05, Vol.7 (5), p.338-350

2016

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Effects of operating conditions and geometries on the performance of nitrogen ejectors for Joule–Thomson cooling

Applied thermal engineering, 2022-07, Vol.212, p.118557, Article 118557

2022

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Insights into the crystallization and vitrification of cryopreserved cells

Cryobiology, 2022-06, Vol.106, p.13-23

2022

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Temperature stability of a Joule–Thomson microcooler operating with pure and mixed gases

Applied thermal engineering, 2024-02, Vol.238, p.121995, Article 121995

2024

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A closed-cycle miniature Joule–Thomson cooler for cooling cold electronics

Applied thermal engineering, 2022-06, Vol.209, p.118237, Article 118237

2022

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Genetic and Pharmacologic Inhibition of JAK1/2 Antagonizes Cardiac Fibrosis

Circulation (New York, N.Y.), 2024-09, Vol.150 (11), p.899-901

2024

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Repression of MAP3K1 expression and JNK activity by canonical Wnt signaling

Developmental biology, 2018-08, Vol.440 (2), p.129-136

2018

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A method of underwater bridge structure damage detection method based on a lightweight deep convolutional network

IET image processing, 2022-12, Vol.16 (14), p.3893-3909

2022

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Abstract P2042: Linking Extracellular Matrix Stiffness With Cardiomyocyte Structure And Function

Circulation research, 2023-08, Vol.133 (Suppl_1)

2023

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Myofibroblast-Specific TGFβ Receptor II Signaling in the Fibrotic Response to Cardiac Myosin Binding Protein C-Induced Cardiomyopathy

Circulation research, 2018-12, Vol.123 (12), p.1285-1297

2018

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c-Jun, at the crossroad of the signaling network

Targeting Interactions between Fibroblasts and Macrophages to Treat Cardiac Fibrosis

Identification of Underwater Structural Bridge Damage and BIM-Based Bridge Damage Management

Eyelid Closure in Embryogenesis Is Required for Ocular Adnexa Development

Corneal Wound Healing Requires IKB kinase β Signaling in Keratocytes

A BIM technology-based underwater structure damage identification and management method

Cardiac Fibrosis in Proteotoxic Cardiac Disease is Dependent Upon Myofibroblast TGF -β Signaling

Activation of AMPK by berberine promotes adiponectin multimerization in 3T3-L1 adipocytes

IκB kinase β regulates epithelium migration during corneal wound healing

Mitogen-activated protein kinase kinase kinase 1 (MAP3K1) integrates developmental signals for eyelid closure

Col1a2 -Deleted Mice Have Defective Type I Collagen and Secondary Reactive Cardiac Fibrosis with Altered Hypertrophic Dynamics

Expression of signaling components in embryonic eyelid epithelium

MAP3K1 function is essential for cytoarchitecture of the mouse organ of Corti and survival of auditory hair cells

MMI‐0100 Inhibits Cardiac Fibrosis in a Mouse Model Overexpressing Cardiac Myosin Binding Protein C

Deciphering gene expression program of MAP3K1 in mouse eyelid morphogenesis

Loss of IκB kinase β promotes myofibroblast transformation and senescence through activation of the ROS-TGFβ autocrine loop

Effects of operating conditions and geometries on the performance of nitrogen ejectors for Joule–Thomson cooling

Insights into the crystallization and vitrification of cryopreserved cells

Temperature stability of a Joule–Thomson microcooler operating with pure and mixed gases

A closed-cycle miniature Joule–Thomson cooler for cooling cold electronics

Genetic and Pharmacologic Inhibition of JAK1/2 Antagonizes Cardiac Fibrosis

Repression of MAP3K1 expression and JNK activity by canonical Wnt signaling

A method of underwater bridge structure damage detection method based on a lightweight deep convolutional network

Abstract P2042: Linking Extracellular Matrix Stiffness With Cardiomyocyte Structure And Function

Myofibroblast-Specific TGFβ Receptor II Signaling in the Fibrotic Response to Cardiac Myosin Binding Protein C-Induced Cardiomyopathy

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