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Indoleamine 2,3-Dioxygenase and Tolerance: Where Are We Now?

Frontiers in immunology, 2017-10, Vol.8, p.1360-1360

2017

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STING Promotes the Growth of Tumors Characterized by Low Antigenicity via IDO Activation

Cancer research (Chicago, Ill.), 2016-04, Vol.76 (8), p.2076-2081

2016

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Ido expression by dendritic cells: tolerance and tryptophan catabolism

Nature reviews. Immunology, 2004-10, Vol.4 (10), p.762-774

2004

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CD73 on cancer-associated fibroblasts enhanced by the A2B-mediated feedforward circuit enforces an immune checkpoint

Nature communications, 2020-01, Vol.11 (1), p.1-17, Article 515

2020

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Genipin‐crosslinked chitosan hydrogels: Preliminary evaluation of the in vitro biocompatibility and biodegradation

Journal of applied polymer science, 2021-09, Vol.138 (34), p.n/a

2021

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An Inherently Bifunctional Subset of Foxp3+ T Helper Cells Is Controlled by the Transcription Factor Eos

Immunity (Cambridge, Mass.), 2013-05, Vol.38 (5), p.998-1012

2013

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Amino acid catabolism: a pivotal regulator of innate and adaptive immunity

Immunological reviews, 2012-09, Vol.249 (1), p.135-157

2012

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Indoleamine 2,3-dioxygenase controls conversion of Foxp3+ Tregs to TH17-like cells in tumor-draining lymph nodes

Blood, 2009-06, Vol.113 (24), p.6102-6111

2009

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The tumor-draining lymph node as an immune-privileged site

Immunological reviews, 2006-10, Vol.213 (1), p.146-158

2006

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Plasmacytoid dendritic cells from mouse tumor-draining lymph nodes directly activate mature Tregs via indoleamine 2,3-dioxygenase

The Journal of clinical investigation, 2007-09, Vol.117 (9), p.2570-2582

2007

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Tolerance to apoptotic cells is regulated by indoleamine 2,3-dioxygenase

Proceedings of the National Academy of Sciences - PNAS, 2012-03, Vol.109 (10), p.3909-3914

2012

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Canonical wnt signaling in dendritic cells regulates Th1/Th17 responses and suppresses autoimmune neuroinflammation

The Journal of immunology (1950), 2015-04, Vol.194 (7), p.3295-3304

2015

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IDO Immune Status after Chemoradiation May Predict Survival in Lung Cancer Patients

Cancer research (Chicago, Ill.), 2018-02, Vol.78 (3), p.809-816

2018

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IDO2 is critical for IDO1-mediated T-cell regulation and exerts a non-redundant function in inflammation

International immunology, 2014-07, Vol.26 (7), p.357-367

2014

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Cutting edge: DNA sensing via the STING adaptor in myeloid dendritic cells induces potent tolerogenic responses

The Journal of immunology (1950), 2013-10, Vol.191 (7), p.3509-3513

2013

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Marginal zone CD169+macrophages coordinate apoptotic cell-driven cellular recruitment and tolerance

Proceedings of the National Academy of Sciences - PNAS, 2014-03, Vol.111 (11), p.4215-4220

2014

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Infectious tolerance via the consumption of essential amino acids and mTOR signaling

Proceedings of the National Academy of Sciences - PNAS, 2009-07, Vol.106 (29), p.12055-12060

2009

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Virus Infections Incite Pain Hypersensitivity by Inducing Indoleamine 2,3 Dioxygenase

PLoS pathogens, 2016-05, Vol.12 (5), p.e1005615

2016

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Activation of the STING adaptor attenuates experimental autoimmune encephalitis

The Journal of immunology (1950), 2014-06, Vol.192 (12), p.5571-5578

2014

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Indoleamine 2,3 dioxygenase and metabolic control of immune responses

Trends in immunology, 2013-03, Vol.34 (3), p.137-143

2013

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GCN2-Dependent Metabolic Stress Is Essential for Endotoxemic Cytokine Induction and Pathology

Molecular and Cellular Biology, 2014-02, Vol.34 (3), p.428-438

2014

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Chronic inflammation that facilitates tumor progression creates local immune suppression by inducing indoleamine 2,3 dioxygenase

Proceedings of the National Academy of Sciences - PNAS, 2008-11, Vol.105 (44), p.17073-17078

2008

Volltextzugriff (PDF)

Inducing the tryptophan catabolic pathway, indoleamine 2,3-dioxygenase (IDO), for suppression of graft-versus-host disease (GVHD) lethality

Blood, 2009-12, Vol.114 (24), p.5062-5070

2009

Volltextzugriff (PDF)

Opposing Biological Functions of Tryptophan Catabolizing Enzymes During Intracellular Infection

The Journal of infectious diseases, 2012-01, Vol.205 (1), p.152-161

2012

Volltextzugriff (PDF)

Induction and role of indoleamine 2,3 dioxygenase in mouse models of influenza a virus infection

PloS one, 2013-06, Vol.8 (6), p.e66546-e66546

2013

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Indoleamine 2,3-Dioxygenase and Tolerance: Where Are We Now?

STING Promotes the Growth of Tumors Characterized by Low Antigenicity via IDO Activation

Ido expression by dendritic cells: tolerance and tryptophan catabolism

CD73 on cancer-associated fibroblasts enhanced by the A2B-mediated feedforward circuit enforces an immune checkpoint

Genipin‐crosslinked chitosan hydrogels: Preliminary evaluation of the in vitro biocompatibility and biodegradation

An Inherently Bifunctional Subset of Foxp3+ T Helper Cells Is Controlled by the Transcription Factor Eos

Amino acid catabolism: a pivotal regulator of innate and adaptive immunity

Indoleamine 2,3-dioxygenase controls conversion of Foxp3+ Tregs to TH17-like cells in tumor-draining lymph nodes

The tumor-draining lymph node as an immune-privileged site

Plasmacytoid dendritic cells from mouse tumor-draining lymph nodes directly activate mature Tregs via indoleamine 2,3-dioxygenase

Tolerance to apoptotic cells is regulated by indoleamine 2,3-dioxygenase

Canonical wnt signaling in dendritic cells regulates Th1/Th17 responses and suppresses autoimmune neuroinflammation

IDO Immune Status after Chemoradiation May Predict Survival in Lung Cancer Patients

IDO2 is critical for IDO1-mediated T-cell regulation and exerts a non-redundant function in inflammation

Cutting edge: DNA sensing via the STING adaptor in myeloid dendritic cells induces potent tolerogenic responses

Marginal zone CD169+macrophages coordinate apoptotic cell-driven cellular recruitment and tolerance

Infectious tolerance via the consumption of essential amino acids and mTOR signaling

Virus Infections Incite Pain Hypersensitivity by Inducing Indoleamine 2,3 Dioxygenase

Activation of the STING adaptor attenuates experimental autoimmune encephalitis

Indoleamine 2,3 dioxygenase and metabolic control of immune responses

GCN2-Dependent Metabolic Stress Is Essential for Endotoxemic Cytokine Induction and Pathology

Chronic inflammation that facilitates tumor progression creates local immune suppression by inducing indoleamine 2,3 dioxygenase

Inducing the tryptophan catabolic pathway, indoleamine 2,3-dioxygenase (IDO), for suppression of graft-versus-host disease (GVHD) lethality

Opposing Biological Functions of Tryptophan Catabolizing Enzymes During Intracellular Infection

Induction and role of indoleamine 2,3 dioxygenase in mouse models of influenza a virus infection

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