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Timing hypothesis for postmenopausal hormone therapy: its origin, current status, and future

Menopause (New York, N.Y.), 2013-03, Vol.20 (3), p.342-353

2013

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Long-term QT prolongation in monkeys after doxorubicin administration at doses similar to breast cancer therapy

Frontiers in cardiovascular medicine, 2023-12, Vol.10, p.1247273-1247273

2023

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Is Myocardial Fibrosis a New Frontier for Discovery in Cardiotoxicity Related to the Administration of Anthracyclines?

Circulation. Cardiovascular imaging, 2016-12, Vol.9 (12)

2016

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CMR-Derived Regional Strain and Radiation-Induced Cardiotoxicity: The Importance of Myocardial Inflammation

JACC CardioOncology, 2021-03, Vol.3 (1), p.131-133

Meléndez, Giselle C.

2021

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Premature Cardiac Senescence: An Unsung Hero in Chemotherapy-Induced Cardiotoxicity?

JACC CardioOncology, 2023-06, Vol.5 (3), p.316-317

2023

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New Insight Into Cardioprotection From Anthracyclines: Still Converging on the Mitochondria

JACC CardioOncology, 2023-06, Vol.5 (3), p.374-376

2023

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Interleukin 6 Mediates Myocardial Fibrosis, Concentric Hypertrophy, and Diastolic Dysfunction in Rats

Hypertension (Dallas, Tex. 1979), 2010-08, Vol.56 (2), p.225-231

2010

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The Role of Cardiac MRI in Animal Models of Cardiotoxicity: Hopes and Challenges

Journal of cardiovascular translational research, 2020-06, Vol.13 (3), p.367-376

2020

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Targeting substance P and relaxin: A future combination therapy approach for heart failure?

International journal of cardiology, 2016-02, Vol.204, p.154-155

2016

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Prevention of adverse cardiac remodeling to volume overload in female rats is the result of an estrogen-altered mast cell phenotype

American journal of physiology. Heart and circulatory physiology, 2012-02, Vol.302 (3), p.H811-H817

2012

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TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload

American journal of physiology. Heart and circulatory physiology, 2009-10, Vol.297 (4), p.H1462-H1468

2009

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Stem cell factor is responsible for the rapid response in mature mast cell density in the acutely stressed heart

Journal of molecular and cellular cardiology, 2012-10, Vol.53 (4), p.469-474

2012

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Eicosapentaenoic Acid Ameliorates Cardiac Fibrosis and Tissue Inflammation in Spontaneously Hypertensive Rats

Journal of lipid research, 2022-11, Vol.63 (11), p.100292-100292, Article 100292

2022

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Automated assessments of circumferential strain from cine CMR correlate with LVEF declines in cancer patients early after receipt of cardio-toxic chemotherapy

Journal of cardiovascular magnetic resonance, 2017-08, Vol.19 (1), p.59-12, Article 59

2017

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Anthracycline-Associated T1 Mapping Characteristics Are Elevated Independent of the Presence of Cardiovascular Comorbidities in Cancer Survivors

Circulation. Cardiovascular imaging, 2016-08, Vol.9 (8)

2016

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Progressive 3-Month Increase in LV Myocardial ECV After Anthracycline-Based Chemotherapy

JACC. Cardiovascular imaging, 2017-06, Vol.10 (6), p.708-709

2017

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Cardiac mast cells: the centrepiece in adverse myocardial remodelling

Cardiovascular research, 2011-01, Vol.89 (1), p.12-19

2011

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Tryptase/Protease-Activated Receptor 2 Interactions Induce Selective Mitogen-Activated Protein Kinase Signaling and Collagen Synthesis by Cardiac Fibroblasts

Hypertension (Dallas, Tex. 1979), 2011-08, Vol.58 (2), p.264-270

2011

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Replacement substance P reduces cardiac fibrosis in monkeys with type 2 diabetes

Biomedicine & pharmacotherapy, 2023-04, Vol.160, p.114365, Article 114365

2023

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Left Ventricular Mass Change After Anthracycline Chemotherapy

Circulation. Heart failure, 2018-07, Vol.11 (7), p.e004560-e004560

2018

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Early Myocardial Strain Changes During Potentially Cardiotoxic Chemotherapy May Occur as a Result of Reductions in Left Ventricular End-Diastolic Volume: The Need to Interpret Left Ventricular Strain With Volumes

Circulation (New York, N.Y.), 2017-06, Vol.135 (25), p.2575-2577

2017

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Hypertension Severity and Declines in Left Ventricular Ejection Fraction Among Women Receiving Adjuvant Chemotherapy for Breast Cancer (WF-97415 UPBEAT)

Hypertension (Dallas, Tex. 1979), 2024-06, Vol.81 (6), p.1365-1373

2024

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Frequency of Left Ventricular End-Diastolic Volume Mediated Declines in Ejection Fraction in Patients Receiving Potentially Cardiotoxic Cancer Treatment

The American journal of cardiology, 2017-05, Vol.119 (10), p.1637-1642

2017

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Exogenous Growth Hormone Exacerbates Post-Irradiation Atherosclerosis in Susceptible Epicardial Coronary Arteries

Toxicologic pathology, 2024-09, p.1926233241277454

2024

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Simultaneous Left Ventricular Volume and Strain Changes During Chemotherapy Associate With 2-Year Postchemotherapy Measures of Left Ventricular Ejection Fraction

Journal of the American Heart Association, 2020-01, Vol.9 (2), p.e015400

2020

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Timing hypothesis for postmenopausal hormone therapy: its origin, current status, and future

Long-term QT prolongation in monkeys after doxorubicin administration at doses similar to breast cancer therapy

Is Myocardial Fibrosis a New Frontier for Discovery in Cardiotoxicity Related to the Administration of Anthracyclines?

CMR-Derived Regional Strain and Radiation-Induced Cardiotoxicity: The Importance of Myocardial Inflammation

Premature Cardiac Senescence: An Unsung Hero in Chemotherapy-Induced Cardiotoxicity?

New Insight Into Cardioprotection From Anthracyclines: Still Converging on the Mitochondria

Interleukin 6 Mediates Myocardial Fibrosis, Concentric Hypertrophy, and Diastolic Dysfunction in Rats

The Role of Cardiac MRI in Animal Models of Cardiotoxicity: Hopes and Challenges

Targeting substance P and relaxin: A future combination therapy approach for heart failure?

Prevention of adverse cardiac remodeling to volume overload in female rats is the result of an estrogen-altered mast cell phenotype

TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload

Stem cell factor is responsible for the rapid response in mature mast cell density in the acutely stressed heart

Eicosapentaenoic Acid Ameliorates Cardiac Fibrosis and Tissue Inflammation in Spontaneously Hypertensive Rats

Automated assessments of circumferential strain from cine CMR correlate with LVEF declines in cancer patients early after receipt of cardio-toxic chemotherapy

Anthracycline-Associated T1 Mapping Characteristics Are Elevated Independent of the Presence of Cardiovascular Comorbidities in Cancer Survivors

Progressive 3-Month Increase in LV Myocardial ECV After Anthracycline-Based Chemotherapy

Cardiac mast cells: the centrepiece in adverse myocardial remodelling

Tryptase/Protease-Activated Receptor 2 Interactions Induce Selective Mitogen-Activated Protein Kinase Signaling and Collagen Synthesis by Cardiac Fibroblasts

Replacement substance P reduces cardiac fibrosis in monkeys with type 2 diabetes

Left Ventricular Mass Change After Anthracycline Chemotherapy

Early Myocardial Strain Changes During Potentially Cardiotoxic Chemotherapy May Occur as a Result of Reductions in Left Ventricular End-Diastolic Volume: The Need to Interpret Left Ventricular Strain With Volumes

Hypertension Severity and Declines in Left Ventricular Ejection Fraction Among Women Receiving Adjuvant Chemotherapy for Breast Cancer (WF-97415 UPBEAT)

Frequency of Left Ventricular End-Diastolic Volume Mediated Declines in Ejection Fraction in Patients Receiving Potentially Cardiotoxic Cancer Treatment

Exogenous Growth Hormone Exacerbates Post-Irradiation Atherosclerosis in Susceptible Epicardial Coronary Arteries

Simultaneous Left Ventricular Volume and Strain Changes During Chemotherapy Associate With 2-Year Postchemotherapy Measures of Left Ventricular Ejection Fraction

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