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Common exposure to STL polyomavirus during childhood

Emerging infectious diseases, 2014-09, Vol.20 (9), p.1559-1561

2014

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A Single Dose, Thermostable, Trivalent Human Papillomavirus Vaccine Formulated Using Atomic Layer Deposition

Journal of pharmaceutical sciences, 2023-08, Vol.112 (8), p.2223-2229

2023

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Dynamics of urinary polyomavirus shedding in healthy adult women

Journal of medical virology, 2012-09, Vol.84 (9), p.1459-1463

2012

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Thermostability of a trivalent, capsomere-based vaccine for human papillomavirus infection

European journal of pharmaceutics and biopharmaceutics, 2021-11, Vol.168, p.131-138

2021

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Dynamics of pregnancy-associated polyomavirus urinary excretion: A prospective longitudinal study

Journal of medical virology, 2012-08, Vol.84 (8), p.1312-1322

2012

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Nicotinic agonists stimulate acetylcholine release from mouse interpeduncular nucleus: a function mediated by a different nAChR than dopamine release from striatum

Journal of neurochemistry, 2001-01, Vol.76 (1), p.258-268

2001

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Development of a highly thermostable, adjuvanted human papillomavirus vaccine

European journal of pharmaceutics and biopharmaceutics, 2015-08, Vol.94, p.220-228

2015

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Single-administration, thermostable human papillomavirus vaccines prepared with atomic layer deposition technology

npj vaccines, 2020, Vol.5 (1), p.45, Article 45

2020

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86Rb + efflux mediated by α4β2-nicotinic acetylcholine receptors with high and low-sensitivity to stimulation by acetylcholine display similar agonist-induced desensitization

Biochemical pharmacology, 2010-10, Vol.80 (8), p.1238-1251

2010

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Single-administration, thermostable human papillomavirus vaccines prepared with atomic layer deposition technology

npj vaccines, 2020-06, Vol.5 (1), p.45

2020

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super(86)Rb super(+) efflux mediated by a4b2-nicotinic acetylcholine receptors with high and low-sensitivity to stimulation by acetylcholine display similar agonist-induced desensitization

Biochemical pharmacology, 2010-10, Vol.80 (8), p.1238-1251

2010

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Partial Deletion of the Nicotinic Cholinergic Receptor Î±4 or Î²2 Subunit Genes Changes the Acetylcholine Sensitivity of Receptor-Mediated 86Rb+ Efflux in Cortex and Thalamus and Alters Relative Expression of Î±4 and Î²2 Subunits

Molecular pharmacology, 2008-06, Vol.73 (6), p.1796

2008

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Partial Deletion of the Nicotinic Cholinergic Receptor α4 or β2 Subunit Genes Changes the Acetylcholine Sensitivity of Receptor-Mediated 86 Rb + Efflux in Cortex and Thalamus and Alters Relative Expression of α4 and β2 Subunits

Molecular pharmacology, 2008-06, Vol.73 (6), p.1796-1807

2008

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Partial Deletion of the Nicotinic Cholinergic Receptor alpha 4 or beta 2 Subunit Genes Changes the Acetylcholine Sensitivity of Receptor-Mediated super(86)Rb super(+) Efflux in Cortex and Thalamus and Alters Relative Expression of alpha 4 and beta 2 Subunits

Molecular pharmacology, 2008-06, Vol.73 (6), p.1796-1807

2008

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Gene targeting demonstrates that alpha4 nicotinic acetylcholine receptor subunits contribute to expression of diverse [3H]epibatidine binding sites and components of biphasic 86Rb+ efflux with high and low sensitivity to stimulation by acetylcholine

Neuropharmacology, 2007-09, Vol.53 (3), p.390

2007

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Gene targeting demonstrates that α4 nicotinic acetylcholine receptor subunits contribute to expression of diverse [ 3H]epibatidine binding sites and components of biphasic 86Rb + efflux with high and low sensitivity to stimulation by acetylcholine

Neuropharmacology, 2007-09, Vol.53 (3), p.390-405

2007

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86Rb+ efflux mediated by alpha4beta2-nicotinic acetylcholine receptors with high and low-sensitivity to stimulation by acetylcholine display similar agonist-induced desensitization

Biochemical pharmacology, 2010-10, Vol.80 (8), p.1238

2010

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Partial deletion of the nicotinic cholinergic receptor alpha 4 or beta 2 subunit genes changes the acetylcholine sensitivity of receptor-mediated 86Rb+ efflux in cortex and thalamus and alters relative expression of alpha 4 and beta 2 subunits

Molecular pharmacology, 2008-06, Vol.73 (6), p.1796-1807

2008

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Common exposure to STL polyomavirus during childhood

A Single Dose, Thermostable, Trivalent Human Papillomavirus Vaccine Formulated Using Atomic Layer Deposition

Dynamics of urinary polyomavirus shedding in healthy adult women

Thermostability of a trivalent, capsomere-based vaccine for human papillomavirus infection

Dynamics of pregnancy-associated polyomavirus urinary excretion: A prospective longitudinal study

Nicotinic agonists stimulate acetylcholine release from mouse interpeduncular nucleus: a function mediated by a different nAChR than dopamine release from striatum

Development of a highly thermostable, adjuvanted human papillomavirus vaccine

Single-administration, thermostable human papillomavirus vaccines prepared with atomic layer deposition technology

86Rb + efflux mediated by α4β2-nicotinic acetylcholine receptors with high and low-sensitivity to stimulation by acetylcholine display similar agonist-induced desensitization

Single-administration, thermostable human papillomavirus vaccines prepared with atomic layer deposition technology

super(86)Rb super(+) efflux mediated by a4b2-nicotinic acetylcholine receptors with high and low-sensitivity to stimulation by acetylcholine display similar agonist-induced desensitization

Partial Deletion of the Nicotinic Cholinergic Receptor Î±4 or Î²2 Subunit Genes Changes the Acetylcholine Sensitivity of Receptor-Mediated 86Rb+ Efflux in Cortex and Thalamus and Alters Relative Expression of Î±4 and Î²2 Subunits

Partial Deletion of the Nicotinic Cholinergic Receptor α4 or β2 Subunit Genes Changes the Acetylcholine Sensitivity of Receptor-Mediated 86 Rb + Efflux in Cortex and Thalamus and Alters Relative Expression of α4 and β2 Subunits

Partial Deletion of the Nicotinic Cholinergic Receptor alpha 4 or beta 2 Subunit Genes Changes the Acetylcholine Sensitivity of Receptor-Mediated super(86)Rb super(+) Efflux in Cortex and Thalamus and Alters Relative Expression of alpha 4 and beta 2 Subunits

Gene targeting demonstrates that alpha4 nicotinic acetylcholine receptor subunits contribute to expression of diverse [3H]epibatidine binding sites and components of biphasic 86Rb+ efflux with high and low sensitivity to stimulation by acetylcholine

Gene targeting demonstrates that α4 nicotinic acetylcholine receptor subunits contribute to expression of diverse [ 3H]epibatidine binding sites and components of biphasic 86Rb + efflux with high and low sensitivity to stimulation by acetylcholine

86Rb+ efflux mediated by alpha4beta2-nicotinic acetylcholine receptors with high and low-sensitivity to stimulation by acetylcholine display similar agonist-induced desensitization

Partial deletion of the nicotinic cholinergic receptor alpha 4 or beta 2 subunit genes changes the acetylcholine sensitivity of receptor-mediated 86Rb+ efflux in cortex and thalamus and alters relative expression of alpha 4 and beta 2 subunits

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