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Deficiency in CCR2 increases susceptibility of mice to infection with an intracellular pathogen, Francisella tularensis LVS, but does not impair development of protective immunity
PloS one, 2021-03, Vol.16 (3), p.e0249142-e0249142
2021
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Control of Francisella tularensis Intracellular Growth by Pulmonary Epithelial Cells
PloS one, 2015-09, Vol.10 (9), p.e0138565-e0138565
2015
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A Francisella tularensis Pathogenicity Island Required for Intramacrophage Growth
Journal of Bacteriology, 2004-10, Vol.186 (19), p.6430-6436
2004
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MAIT cells promote inflammatory monocyte differentiation into dendritic cells during pulmonary intracellular infection
The Journal of experimental medicine, 2016-11, Vol.213 (12), p.2793-2809
2016
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T Cells from Lungs and Livers of Francisella tularensis-Immune Mice Control the Growth of Intracellular Bacteria
Infection and Immunity, 2009-05, Vol.77 (5), p.2010-2021
2009
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Lung CD4-CD8- double-negative T cells are prominent producers of IL-17A and IFN-gamma during primary respiratory murine infection with Francisella tularensis live vaccine strain
The Journal of immunology (1950), 2010-05, Vol.184 (10), p.5791-5801
2010
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Myeloid differentiation factor-88 (MyD88) is essential for control of primary in vivo Francisella tularensis LVS infection, but not for control of intramacrophage bacterial replication
Microbes and infection, 2006-03, Vol.8 (3), p.779-790
2006
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Survival of secondary lethal systemic Francisella LVS challenge depends largely on interferon gamma
Microbes and infection, 2010, Vol.12 (1), p.28-36
2010
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Control of Francisella tularensis Intracellular Growth by Pulmonary Epithelial Cells: e0138565
PloS one, 2015-09, Vol.10 (9)
2015
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Tumor necrosis factor-alpha: central regulatory cytokine in the induction of macrophage antimicrobial activities
Pathobiology (Basel), 1991, Vol.59 (3), p.182
1991
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Myeloid differentiation factor-88 (MyD88) is essential for control of primary in vivo Francisella tularensis LVS infection, but not for control of intra-macrophage bacterial replication
Microbes and infection, 2006-03, Vol.8 (3), p.779-790
2006
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MAIT cells are critical for optimal mucosal immune responses during in vivo pulmonary bacterial infection
Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (33), p.E3119-E3128
2013
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Interleukin-18 Is Critical for Mucosa-Associated Invariant T Cell Gamma Interferon Responses to Francisella Species In Vitro but Not In Vivo
Infection and immunity, 2018-05, Vol.86 (5)
2018
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MAIT cells are critical for optimal mucosal immune responses during in vivo pulmonary bacterial infection
Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (33), p.13243-13243
2013
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