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Mutational signatures are jointly shaped by DNA damage and repair

Nature communications, 2020-05, Vol.11 (1), p.2169-2169, Article 2169

2020

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Protection of the C. elegans germ cell genome depends on diverse DNA repair pathways during normal proliferation

PloS one, 2021-04, Vol.16 (4), p.e0250291

2021

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Analysis of mutational signatures in C. elegans: Implications for cancer genome analysis

DNA repair, 2020-11, Vol.95, p.102957, Article 102957

2020

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Having a direct look: Analysis of DNA damage and repair mechanisms by next generation sequencing

Experimental cell research, 2014-11, Vol.329 (1), p.35-41

2014

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LEM-3 is a midbody-tethered DNA nuclease that resolves chromatin bridges during late mitosis

Nature communications, 2018-02, Vol.9 (1), p.728-11, Article 728

2018

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Combinatorial regulation of meiotic holliday junction resolution in C. elegans by HIM-6 (BLM) helicase, SLX-4, and the SLX-1, MUS-81 and XPF-1 nucleases

PLoS genetics, 2013-07, Vol.9 (7), p.e1003591-e1003591

2013

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Clinical manifestations in primary ciliary dyskinesia: systematic review and meta-analysis

The European respiratory journal, 2016-10, Vol.48 (4), p.1081-1095

2016

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The SMC-5/6 Complex and the HIM-6 (BLM) Helicase Synergistically Promote Meiotic Recombination Intermediate Processing and Chromosome Maturation during Caenorhabditis elegans Meiosis

PLoS genetics, 2016-03, Vol.12 (3), p.e1005872-e1005872

2016

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Caenorhabditis elegans BUB-3 and SAN-1/MAD3 Spindle Assembly Checkpoint Components Are Required for Genome Stability in Response to Treatment with Ionizing Radiation

G3 : genes - genomes - genetics, 2017-12, Vol.7 (12), p.3875-3885

2017

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trt-1 is the Caenorhabditis elegans catalytic subunit of telomerase

PLoS genetics, 2006-02, Vol.2 (2), p.e18-e18

2006

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Correction: Combinatorial Regulation of Meiotic Holliday Junction Resolution in C. elegans by HIM-6 (BLM) Helicase, SLX-4, and the SLX-1, MUS-81 and XPF-1 Nucleases

PLoS genetics, 2013-08, Vol.9 (8)

2013

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Kapital und Kompetenz: Veränderungen der Arbeitswelt und Ihre Auswirkungen Aus Erziehungswissenschaftlicher Sicht

1, 2004

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Separation-of-function Mutants of Yeast Ku80 Reveal a Yku80p-Sir4p Interaction Involved in Telomeric Silencing

The Journal of biological chemistry, 2004-01, Vol.279 (1), p.86-94

2004

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Mutational signatures of DNA mismatch repair deficiency in C. elegans and human cancers

Genome research, 2018-05, Vol.28 (5), p.666-675

2018

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C. elegans whole-genome sequencing reveals mutational signatures related to carcinogens and DNA repair deficiency

Genome research, 2014-10, Vol.24 (10), p.1624-1636

2014

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Checkpoints: Chromosome pairing takes an unexpected twist

Current biology, 2001-10, Vol.11 (21), p.R865-R868

2001

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Corrigendum: Mutational signatures of DNA mismatch repair deficiency in C. elegans and human cancers

Genome research, 2019-09, Vol.29 (9), p.1566-1566_2

2019

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The SMC-5/6 Complex and the HIM-6

PLoS genetics, 2016-03, Vol.12 (3)

2016

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HDF2, the Second Subunit of the Ku Homologue from Saccharomyces cerevisiae

The Journal of biological chemistry, 1996-11, Vol.271 (44), p.27765-27769

1996

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An optimized synthetic route for the preparation of the versatile chiral building block 1,4-di-O-benzylthreitol

Monatshefte für Chemie, 2014-02, Vol.145 (2), p.305-309

2014

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Kapital und Kompetenz: Veranderungen der Arbeitswelt und ihre Auswirkungen aus erziehungswissenschaftlicher Sicht

2004 edition., 2013

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New Function of CDC13 in Positive Telomere Length Regulation

Molecular and Cellular Biology, 2001-07, Vol.21 (13), p.4233-4245

2001

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Abstract 6011: Pharmacological inhibition of PMS2 increases tumor mutational burden, induces microsatellite instability and elicits immune mediated rejection in vivo

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.6011-6011

2024

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Mutational signatures are jointly shaped by DNA damage and repair

Protection of the C. elegans germ cell genome depends on diverse DNA repair pathways during normal proliferation

Analysis of mutational signatures in C. elegans: Implications for cancer genome analysis

Having a direct look: Analysis of DNA damage and repair mechanisms by next generation sequencing

LEM-3 is a midbody-tethered DNA nuclease that resolves chromatin bridges during late mitosis

Combinatorial regulation of meiotic holliday junction resolution in C. elegans by HIM-6 (BLM) helicase, SLX-4, and the SLX-1, MUS-81 and XPF-1 nucleases

Clinical manifestations in primary ciliary dyskinesia: systematic review and meta-analysis

The SMC-5/6 Complex and the HIM-6 (BLM) Helicase Synergistically Promote Meiotic Recombination Intermediate Processing and Chromosome Maturation during Caenorhabditis elegans Meiosis

Caenorhabditis elegans BUB-3 and SAN-1/MAD3 Spindle Assembly Checkpoint Components Are Required for Genome Stability in Response to Treatment with Ionizing Radiation

trt-1 is the Caenorhabditis elegans catalytic subunit of telomerase

Correction: Combinatorial Regulation of Meiotic Holliday Junction Resolution in C. elegans by HIM-6 (BLM) Helicase, SLX-4, and the SLX-1, MUS-81 and XPF-1 Nucleases

Kapital und Kompetenz: Veränderungen der Arbeitswelt und Ihre Auswirkungen Aus Erziehungswissenschaftlicher Sicht

Separation-of-function Mutants of Yeast Ku80 Reveal a Yku80p-Sir4p Interaction Involved in Telomeric Silencing

Mutational signatures of DNA mismatch repair deficiency in C. elegans and human cancers

C. elegans whole-genome sequencing reveals mutational signatures related to carcinogens and DNA repair deficiency

Checkpoints: Chromosome pairing takes an unexpected twist

Corrigendum: Mutational signatures of DNA mismatch repair deficiency in C. elegans and human cancers

The SMC-5/6 Complex and the HIM-6

HDF2, the Second Subunit of the Ku Homologue from Saccharomyces cerevisiae

An optimized synthetic route for the preparation of the versatile chiral building block 1,4-di-O-benzylthreitol

Kapital und Kompetenz: Veranderungen der Arbeitswelt und ihre Auswirkungen aus erziehungswissenschaftlicher Sicht

New Function of CDC13 in Positive Telomere Length Regulation

Abstract 6011: Pharmacological inhibition of PMS2 increases tumor mutational burden, induces microsatellite instability and elicits immune mediated rejection in vivo

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