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γ-Tocotrienol Protects against Mitochondrial Dysfunction, Energy Deficits, Morphological Damage, and Decreases in Renal Functions after Renal Ischemia

International journal of molecular sciences, 2021-11, Vol.22 (23), p.12674

2021

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Sulforaphane protects the heart from doxorubicin-induced toxicity

Free radical biology & medicine, 2015-09, Vol.86, p.90-101

2015

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Formoterol Restores Mitochondrial and Renal Function after Ischemia-Reperfusion Injury

Journal of the American Society of Nephrology, 2014-06, Vol.25 (6), p.1157-1162

2014

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Mitochondrial biogenesis induced by the β2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury

Kidney international, 2019-09, Vol.96 (3), p.656-673

2019

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Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease

American journal of physiology. Renal physiology, 2018-07, Vol.315 (1), p.F161-F172

2018

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Repeated administration of low-dose cisplatin in mice induces fibrosis

American journal of physiology. Renal physiology, 2016-03, Vol.310 (6), p.F560-F568

2016

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Evidence Suggesting a Role of Iron in a Mouse Model of Nephrogenic Systemic Fibrosis

PloS one, 2015-08, Vol.10 (8), p.e0136563

2015

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Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury

Journal of lipid research, 2017-07, Vol.58 (7), p.1439-1452

2017

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Pericytes and immune cells contribute to complement activation in tubulointerstitial fibrosis

American journal of physiology. Renal physiology, 2017-03, Vol.312 (3), p.F516-F532

2017

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5-HT1F receptor regulates mitochondrial homeostasis and its loss potentiates acute kidney injury and impairs renal recovery

American journal of physiology. Renal physiology, 2018-10, Vol.315 (4), p.F1119-F1128

2018

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Activation and involvement of p53 in cisplatin-induced nephrotoxicity

American journal of physiology. Renal physiology, 2007-10, Vol.293 (4), p.F1282-F1291

2007

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Transgenic expression of proximal tubule peroxisome proliferator–activated receptor-α in mice confers protection during acute kidney injury

Kidney international, 2009-11, Vol.76 (10), p.1049-1062

2009

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Deletion of VDAC1 Hinders Recovery of Mitochondrial and Renal Functions After Acute Kidney Injury

Biomolecules (Basel, Switzerland), 2020-04, Vol.10 (4), p.585

2020

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Suramin: a potential therapy for diabetic nephropathy

PloS one, 2013-09, Vol.8 (9), p.e73655-e73655

2013

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Neutral ceramidase deficiency protects against cisplatin-induced acute kidney injury

Journal of lipid research, 2022-03, Vol.63 (3), p.100179, Article 100179

2022

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Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells

American journal of physiology. Renal physiology, 2004-09, Vol.287 (3), p.F543-F549

2004

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Gender differences control the susceptibility to ER stress-induced acute kidney injury

American journal of physiology. Renal physiology, 2013-04, Vol.304 (7), p.F875-F882

2013

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Suramin protects from cisplatin-induced acute kidney injury

American journal of physiology. Renal physiology, 2016-02, Vol.310 (3), p.F248-F258

2016

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Disruption of renal peritubular blood flow in lipopolysaccharide-induced renal failure: role of nitric oxide and caspases

American journal of physiology. Renal physiology, 2005-12, Vol.289 (6), p.F1324-F1332

2005

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Protein kinase Cα mediates recovery of renal and mitochondrial functions following acute injury

The FEBS journal, 2020-05, Vol.287 (9), p.1830-1849

2020

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Alteration of renal respiratory Complex-III during experimental type-1 diabetes

BMC endocrine disorders, 2009-01, Vol.9 (1), p.2-2, Article 2

2009

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Activation of ERK or inhibition of JNK ameliorates H2O2 cytotoxicity in mouse renal proximal tubule cells

Kidney international, 2004-04, Vol.65 (4), p.1231-1239

2004

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Reduced kidney lipoprotein lipase and renal tubule triglyceride accumulation in cisplatin-mediated acute kidney injury

American Journal of Physiology - Renal Physiology, 2012-08, Vol.303 (3), p.F437-F448

2012

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Protection of renal cells from cisplatin toxicity by cell cycle inhibitors

American journal of physiology. Renal physiology, 2004-02, Vol.286 (2), p.F378-F384

2004

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Involvement of the CDK2-E2F1 pathway in cisplatin cytotoxicity in vitro and in vivo

American journal of physiology. Renal physiology, 2007-07, Vol.293 (1), p.F52-F59

2007

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γ-Tocotrienol Protects against Mitochondrial Dysfunction, Energy Deficits, Morphological Damage, and Decreases in Renal Functions after Renal Ischemia

Sulforaphane protects the heart from doxorubicin-induced toxicity

Formoterol Restores Mitochondrial and Renal Function after Ischemia-Reperfusion Injury

Mitochondrial biogenesis induced by the β2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury

Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease

Repeated administration of low-dose cisplatin in mice induces fibrosis

Evidence Suggesting a Role of Iron in a Mouse Model of Nephrogenic Systemic Fibrosis

Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury

Pericytes and immune cells contribute to complement activation in tubulointerstitial fibrosis

5-HT1F receptor regulates mitochondrial homeostasis and its loss potentiates acute kidney injury and impairs renal recovery

Activation and involvement of p53 in cisplatin-induced nephrotoxicity

Transgenic expression of proximal tubule peroxisome proliferator–activated receptor-α in mice confers protection during acute kidney injury

Deletion of VDAC1 Hinders Recovery of Mitochondrial and Renal Functions After Acute Kidney Injury

Suramin: a potential therapy for diabetic nephropathy

Neutral ceramidase deficiency protects against cisplatin-induced acute kidney injury

Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells

Gender differences control the susceptibility to ER stress-induced acute kidney injury

Suramin protects from cisplatin-induced acute kidney injury

Disruption of renal peritubular blood flow in lipopolysaccharide-induced renal failure: role of nitric oxide and caspases

Protein kinase Cα mediates recovery of renal and mitochondrial functions following acute injury

Alteration of renal respiratory Complex-III during experimental type-1 diabetes

Activation of ERK or inhibition of JNK ameliorates H2O2 cytotoxicity in mouse renal proximal tubule cells

Reduced kidney lipoprotein lipase and renal tubule triglyceride accumulation in cisplatin-mediated acute kidney injury

Protection of renal cells from cisplatin toxicity by cell cycle inhibitors

Involvement of the CDK2-E2F1 pathway in cisplatin cytotoxicity in vitro and in vivo

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