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TRPA1: a molecular view

Journal of neurophysiology, 2019-02, Vol.121 (2), p.427-443

2019

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Sensitization of TRPA1 by Protein Kinase A

PloS one, 2017-01, Vol.12 (1), p.e0170097

2017

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Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity

The Journal of biological chemistry, 2021-01, Vol.296, p.100227, Article 100227

2021

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The role of Nav1.7 in human nociceptors: insights from human induced pluripotent stem cell-derived sensory neurons of erythromelalgia patients

Pain (Amsterdam), 2019-06, Vol.160 (6), p.1327-1341

2019

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Correction: Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity

The Journal of biological chemistry, 2021-09, Vol.297 (3), p.101122-101122, Article 101122

2021

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Identification of aurintricarboxylic acid as a potent allosteric antagonist of P2X1 and P2X3 receptors

Neuropharmacology, 2019-11, Vol.158, p.107749, Article 107749

2019

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Agonist‐induced sensitisation of the irritant receptor ion channel TRPA1

The Journal of physiology, 2016-11, Vol.594 (22), p.6643-6660

2016

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The difficulty to model Huntington's disease in vitro using striatal medium spiny neurons differentiated from human induced pluripotent stem cells

Scientific reports, 2021-03, Vol.11 (1), p.6934-6934, Article 6934

2021

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TRPV1 in migraine pathophysiology

Trends in molecular medicine, 2010-04, Vol.16 (4), p.153-159

2010

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The opioid oxycodone use‐dependently inhibits the cardiac sodium channel NaV1.5

British journal of pharmacology, 2018-07, Vol.175 (14), p.3007-3020

2018

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Loss-of-function of Nav1.8/D1639N linked to human pain can be rescued by lidocaine

Pflügers Archiv, 2018-12, Vol.470 (12), p.1787-1801

2018

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Two TRPV1 receptor antagonists are effective in two different experimental models of migraine

Journal of headache and pain, 2015-06, Vol.16 (1), p.57-57, Article 57

2015

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Assessing the impact of pain-linked Nav1.7 variants: An example of two variants with no biophysical effect

Channels (Austin, Tex.), 2021-01, Vol.15 (1), p.208-228

2021

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The Potential Effect of Nav1.8 in Autism Spectrum Disorder: Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations

Frontiers in molecular neuroscience, 2021-07, Vol.14

2021

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Noncanonical Ion Channel Behaviour in Pain

International journal of molecular sciences, 2019-09, Vol.20 (18), p.4572

2019

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SCN10A Mutation in a Patient with Erythromelalgia Enhances C-Fiber Activity Dependent Slowing

PloS one, 2016-09, Vol.11 (9), p.e0161789-e0161789

2016

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Dataset of electrophysiological patch-clamp recordings of the effect of the compounds deltamethrin, ATx-II and β4-peptide on human cardiac Nav1.5 sodium channel gating properties

Data in brief, 2020-08, Vol.31, p.105844-105844, Article 105844

2020

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β1 subunit stabilises sodium channel Nav1.7 against mechanical stress

The Journal of physiology, 2018-06, Vol.596 (12), p.2433-2445

2018

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Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation

British journal of pharmacology, 2020-10, Vol.177 (19), p.4481-4496

2020

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Mechanism underlying hooked resurgent-like tail currents induced by an insecticide in human cardiac Nav1.5

Toxicology and applied pharmacology, 2020-06, Vol.397, p.115010, Article 115010

2020

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Functional Uncoupling of Pain-Linked Nav1.7/A1632E Dimers Partly Rescues Its Pain-Causing Phenotype

Biophysical journal, 2020-02, Vol.118 (3), p.579a-579a

2020

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5-HT1F receptor agonists: A new treatment option for migraine attacks?

Neurotherapeutics, 2010-04, Vol.7 (2), p.176-182

2010

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The opioid oxycodone use-dependently inhibits the cardiac sodium channel Na V 1.5

British journal of pharmacology, 2018-07, Vol.175 (14), p.3007-3020

2018

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The Potential Effect of Na v 1.8 in Autism Spectrum Disorder: Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations

Frontiers in molecular neuroscience, 2021-01, Vol.14, p.709228-709228

2021

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TRPA1: a molecular view

Sensitization of TRPA1 by Protein Kinase A

Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity

The role of Nav1.7 in human nociceptors: insights from human induced pluripotent stem cell-derived sensory neurons of erythromelalgia patients

Correction: Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity

Identification of aurintricarboxylic acid as a potent allosteric antagonist of P2X1 and P2X3 receptors

Agonist‐induced sensitisation of the irritant receptor ion channel TRPA1

The difficulty to model Huntington's disease in vitro using striatal medium spiny neurons differentiated from human induced pluripotent stem cells

TRPV1 in migraine pathophysiology

The opioid oxycodone use‐dependently inhibits the cardiac sodium channel NaV1.5

Loss-of-function of Nav1.8/D1639N linked to human pain can be rescued by lidocaine

Two TRPV1 receptor antagonists are effective in two different experimental models of migraine

Assessing the impact of pain-linked Nav1.7 variants: An example of two variants with no biophysical effect

Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors

The Potential Effect of Nav1.8 in Autism Spectrum Disorder: Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations

Noncanonical Ion Channel Behaviour in Pain

SCN10A Mutation in a Patient with Erythromelalgia Enhances C-Fiber Activity Dependent Slowing

Dataset of electrophysiological patch-clamp recordings of the effect of the compounds deltamethrin, ATx-II and β4-peptide on human cardiac Nav1.5 sodium channel gating properties

β1 subunit stabilises sodium channel Nav1.7 against mechanical stress

Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation

Mechanism underlying hooked resurgent-like tail currents induced by an insecticide in human cardiac Nav1.5

Functional Uncoupling of Pain-Linked Nav1.7/A1632E Dimers Partly Rescues Its Pain-Causing Phenotype

5-HT1F receptor agonists: A new treatment option for migraine attacks?

The opioid oxycodone use-dependently inhibits the cardiac sodium channel Na V 1.5

The Potential Effect of Na v 1.8 in Autism Spectrum Disorder: Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations

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