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Mutant SOD1 mediated pathogenesis of Amyotrophic Lateral Sclerosis

Gene, 2016-02, Vol.577 (2), p.109-118

2016

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Use of the comet-FISH assay to compare DNA damage and repair in p53 and hTERT genes following ionizing radiation

PloS one, 2012-11, Vol.7 (11), p.e49364

2012

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Targeting DNA topoisomerase IIα (TOP2A) in the hypoxic tumour microenvironment using unidirectional hypoxia‐activated prodrugs (uHAPs)

IUBMB life, 2023-01, Vol.75 (1), p.40-54

2023

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The potential of seaweed as a source of drugs for use in cancer chemotherapy

Journal of applied phycology, 2014-10, Vol.26 (5), p.2211-2264

2014

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Radiotherapy for benign disease; assessing the risk of radiation-induced cancer following exposure to intermediate dose radiation

British journal of radiology, 2015-01, Vol.88 (1056), p.20150405

2015

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Targeting Hypoxic Prostate Tumors Using the Novel Hypoxia-Activated Prodrug OCT1002 Inhibits Expression of Genes Associated with Malignant Progression

Clinical cancer research, 2017-04, Vol.23 (7), p.1797-1808

2017

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Nitric Oxide Up-Regulates RUNX2 in LNCaP Prostate Tumours: Implications for Tumour Growth In Vitro and In Vivo

Journal of cellular physiology, 2016-02, Vol.231 (2), p.473-482

2016

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Potential use of the comet assay in the clinical management of cancer

Mutagenesis, 2008-05, Vol.23 (3), p.183-190

2008

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Androgen deprivation results in time‐dependent hypoxia in LNCaP prostate tumours: Informed scheduling of the bioreductive drug AQ4N improves treatment response

International journal of cancer, 2013-03, Vol.132 (6), p.1323-1332

2013

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The unidirectional hypoxia‐activated prodrug OCT1002 inhibits growth and vascular development in castrate‐resistant prostate tumors

The Prostate, 2017-11, Vol.77 (15), p.1539-1547

2017

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Modification of the alkaline Comet assay to allow simultaneous evaluation of mitomycin C-induced DNA cross-link damage and repair of specific DNA sequences in RT4 cells

DNA repair, 2003-08, Vol.2 (8), p.879-890

2003

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Hypoxia selects for androgen independent LNCaP cells with a more malignant geno‐ and phenotype

International journal of cancer, 2008-08, Vol.123 (4), p.760-768

2008

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Comet assay measures of DNA damage are predictive of bladder cancer cell treatment sensitivity in vitro and outcome in vivo

International journal of cancer, 2014-03, Vol.134 (5), p.1102-1111

2014

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Estimating the Risk of Radiation-Induced Malignancy Following Radiotherapy for Benign Disease

Radiation Therapy of Benign Diseases, 2023, p.1-18

2023

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Cytochrome P450 1B1 mRNA untranslated regions interact to inhibit protein translation

Molecular carcinogenesis, 2010-02, Vol.49 (2), p.190-199

2010

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Potential of a Funalia trogii laccase enzyme as an anticancer agent

Annals of microbiology, 2015-03, Vol.65 (1), p.175-183

2015

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Cell cycle checkpoint function in Bladder cancer

JNCI : Journal of the National Cancer Institute, 2003-12, Vol.95 (24), p.1859-1868

2003

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Pericytes influence endothelial cell growth characteristics : Role of plasminogen activator inhibitor type 1 (PAI-1)

Cardiovascular research, 2006, Vol.69 (1), p.207-217

2006

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Investigation of pericytes, hypoxia, and vascularity in bladder tumors: association with clinical outcomes

Oncology research, 2008-01, Vol.17 (3), p.93-101

2008

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A cytochrome P450 2B6 meditated gene therapy strategy to enhance the effects of radiation or cyclophosphamide when combined with the bioreductive drug AQ4N

The journal of gene medicine, 2005-07, Vol.7 (7), p.851-859

2005

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Bioreductive GDEPT using cytochrome P450 3A4 in combination with AQ4N

Cancer gene therapy, 2003-01, Vol.10 (1), p.40-48

2003

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Gene Expression in Normal Urothelium Depends on Location within the Bladder: A Possible Link to Bladder Carcinogenesis

European urology, 2006-08, Vol.50 (2), p.290-301

2006

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Cytochrome P450 1B1 expression in rat esophageal tumorigenesis promoted by gastric and duodenal reflux

Molecular carcinogenesis, 2009-02, Vol.48 (2), p.110-117

2009

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Use of the Comet-FISH Assay to Demonstrate Repair of the TP53 Gene Region in Two Human Bladder Carcinoma Cell Lines

Radiation research, 2003-01, Vol.159 (1), p.49-56

2003

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Abstract 4009: Hypoxia potentiates Runx2-mediated increased Bcl-2 expression in prostate cancer resulting in apoptosis resistance

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.4009-4009

2012

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Mutant SOD1 mediated pathogenesis of Amyotrophic Lateral Sclerosis

Use of the comet-FISH assay to compare DNA damage and repair in p53 and hTERT genes following ionizing radiation

Targeting DNA topoisomerase IIα (TOP2A) in the hypoxic tumour microenvironment using unidirectional hypoxia‐activated prodrugs (uHAPs)

The potential of seaweed as a source of drugs for use in cancer chemotherapy

Radiotherapy for benign disease; assessing the risk of radiation-induced cancer following exposure to intermediate dose radiation

Targeting Hypoxic Prostate Tumors Using the Novel Hypoxia-Activated Prodrug OCT1002 Inhibits Expression of Genes Associated with Malignant Progression

Nitric Oxide Up-Regulates RUNX2 in LNCaP Prostate Tumours: Implications for Tumour Growth In Vitro and In Vivo

Potential use of the comet assay in the clinical management of cancer

Androgen deprivation results in time‐dependent hypoxia in LNCaP prostate tumours: Informed scheduling of the bioreductive drug AQ4N improves treatment response

The unidirectional hypoxia‐activated prodrug OCT1002 inhibits growth and vascular development in castrate‐resistant prostate tumors

Modification of the alkaline Comet assay to allow simultaneous evaluation of mitomycin C-induced DNA cross-link damage and repair of specific DNA sequences in RT4 cells

Hypoxia selects for androgen independent LNCaP cells with a more malignant geno‐ and phenotype

Comet assay measures of DNA damage are predictive of bladder cancer cell treatment sensitivity in vitro and outcome in vivo

Estimating the Risk of Radiation-Induced Malignancy Following Radiotherapy for Benign Disease

Cytochrome P450 1B1 mRNA untranslated regions interact to inhibit protein translation

Potential of a Funalia trogii laccase enzyme as an anticancer agent

Cell cycle checkpoint function in Bladder cancer

Pericytes influence endothelial cell growth characteristics : Role of plasminogen activator inhibitor type 1 (PAI-1)

Investigation of pericytes, hypoxia, and vascularity in bladder tumors: association with clinical outcomes

A cytochrome P450 2B6 meditated gene therapy strategy to enhance the effects of radiation or cyclophosphamide when combined with the bioreductive drug AQ4N

Bioreductive GDEPT using cytochrome P450 3A4 in combination with AQ4N

Gene Expression in Normal Urothelium Depends on Location within the Bladder: A Possible Link to Bladder Carcinogenesis

Cytochrome P450 1B1 expression in rat esophageal tumorigenesis promoted by gastric and duodenal reflux

Use of the Comet-FISH Assay to Demonstrate Repair of the TP53 Gene Region in Two Human Bladder Carcinoma Cell Lines

Abstract 4009: Hypoxia potentiates Runx2-mediated increased Bcl-2 expression in prostate cancer resulting in apoptosis resistance

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