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Insulin Receptor Substrate-2 Proteasomal Degradation Mediated by a Mammalian Target of Rapamycin (mTOR)-induced Negative Feedback Down-regulates Protein Kinase B-mediated Signaling Pathway in β-Cells

The Journal of biological chemistry, 2005-01, Vol.280 (3), p.2282-2293

2005

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A cis-Element in the 5′ Untranslated Region of the Preproinsulin mRNA (ppIGE) Is Required for Glucose Regulation of Proinsulin Translation

Cell metabolism, 2007-03, Vol.5 (3), p.221-227

2007

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Activation of IRS-2—Mediated Signal Transduction by IGF-1, but not TGF-α or EGF, Augments Pancreatic β-Cell Proliferation

Diabetes (New York, N.Y.), 2002-04, Vol.51 (4), p.966-976

2002

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Specific Regulation of IRS-2 Expression by Glucose in Rat Primary Pancreatic Islet β-Cells

The Journal of biological chemistry, 2006-06, Vol.281 (23), p.15884-15892

2006

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Decreasing IRS-2 expression in pancreatic β-cells (INS-1) promotes apoptosis, which can be compensated for by introduction of IRS-4 expression

Molecular and cellular endocrinology, 2003-11, Vol.209 (1), p.17-31

2003

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FoxO feedback control of basal IRS-2 expression in pancreatic β-cells is distinct from that in hepatocytes

Diabetes (New York, N.Y.), 2011-11, Vol.60 (11), p.2883-2891

2011

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IRS-3 inhibits IRS-2-mediated signaling in pancreatic β-cells

Molecular and cellular endocrinology, 2003-06, Vol.204 (1), p.85-99

2003

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FoxO feedback control of basal IRS-2 expression in pancreatic [beta]-cells is distinct from that in hepatocytes.(ORIGINAL ARTICLE)

Diabetes (New York, N.Y.), 2011-11, Vol.60 (11), p.2883

2011

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FoxO Feedback Control of Basal IRS-2 Expression in Pancreatic β-Cells Is Distinct From That in Hepatocytes

Diabetes (New York, N.Y.), 2011, Vol.60 (11), p.2283-2291

2011

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Sequencing and expression analysis of the serine protease gene cluster located in chromosome 19q13 region

Gene, 2000-10, Vol.257 (1), p.119-130

2000

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FXYD6 Directs [beta]-Granule Transport to Active Zones of Exocytosis

Diabetes (New York, N.Y.), 2005-06, Vol.54, p.A44

2005

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Specific regulation of IRS-2 expression by glucose in rat primary pancreatic islet β-cells. VOLUME 281 (2006) PAGES 15884-15892

The Journal of biological chemistry, 2006-08, Vol.281 (32), p.23296-23296

2006

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Transcriptional Regulation of IRS-2 Expression by Both Glucose and Inhibition of PI3K-Signaling in Pancreatic [beta]-Cells

Diabetes (New York, N.Y.), 2005-06, Vol.54, p.A78

2005

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Activation of IRS-2--mediated signal transduction by IGF-1, but not TGF-[alpha] or EGF, augments pancreatic [beta]-cell proliferation

Diabetes (New York, N.Y.), 2002-04, Vol.51 (4), p.966

2002

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Specific Regulation of IRS-2 Expression by Glucose in Rat Primary Pancreatic Islet Î²-Cells

The Journal of biological chemistry, 2006-06, Vol.281 (23), p.15884

2006

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Differential Activation of Protein Kinase B and p70S6K by Glucose and Insulin-like Growth Factor 1 in Pancreatic β-Cells (INS-1)

The Journal of biological chemistry, 2001-06, Vol.276 (24), p.21110-21120

2001

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Differential Activation of Protein Kinase B and p70S6K by Glucose and Insulin-like Growth Factor 1 in Pancreatic Î²-Cells (INS-1)

The Journal of biological chemistry, 2001-06, Vol.276 (24), p.21110

2001

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Insulin Receptor Substrate-2 Proteasomal Degradation Mediated by a Mammalian Target of Rapamycin (mTOR)-induced Negative Feedback Down-regulates Protein Kinase B-mediated Signaling Pathway in Î²-Cells

The Journal of biological chemistry, 2005-01, Vol.280 (3), p.2282

2005

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Making Team Working Work - A Real Life Experience

International journal of language & communication disorders, 1998, Vol.33 (S1), p.570-574

1998

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Making Team Working Work-A Real Life Experience

International journal of language & communication disorders, 1998-01, Vol.33 (supplement), p.570-574

1998

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Insulin Receptor Substrate-2 Proteasomal Degradation Mediated by a Mammalian Target of Rapamycin (mTOR)-induced Negative Feedback Down-regulates Protein Kinase B-mediated Signaling Pathway in β-Cells

A cis-Element in the 5′ Untranslated Region of the Preproinsulin mRNA (ppIGE) Is Required for Glucose Regulation of Proinsulin Translation

Activation of IRS-2—Mediated Signal Transduction by IGF-1, but not TGF-α or EGF, Augments Pancreatic β-Cell Proliferation

Specific Regulation of IRS-2 Expression by Glucose in Rat Primary Pancreatic Islet β-Cells

Decreasing IRS-2 expression in pancreatic β-cells (INS-1) promotes apoptosis, which can be compensated for by introduction of IRS-4 expression

FoxO feedback control of basal IRS-2 expression in pancreatic β-cells is distinct from that in hepatocytes

IRS-3 inhibits IRS-2-mediated signaling in pancreatic β-cells

FoxO feedback control of basal IRS-2 expression in pancreatic [beta]-cells is distinct from that in hepatocytes.(ORIGINAL ARTICLE)

FoxO Feedback Control of Basal IRS-2 Expression in Pancreatic β-Cells Is Distinct From That in Hepatocytes

Sequencing and expression analysis of the serine protease gene cluster located in chromosome 19q13 region

FXYD6 Directs [beta]-Granule Transport to Active Zones of Exocytosis

Specific regulation of IRS-2 expression by glucose in rat primary pancreatic islet β-cells. VOLUME 281 (2006) PAGES 15884-15892

Transcriptional Regulation of IRS-2 Expression by Both Glucose and Inhibition of PI3K-Signaling in Pancreatic [beta]-Cells

Activation of IRS-2--mediated signal transduction by IGF-1, but not TGF-[alpha] or EGF, augments pancreatic [beta]-cell proliferation

Specific Regulation of IRS-2 Expression by Glucose in Rat Primary Pancreatic Islet Î²-Cells

Differential Activation of Protein Kinase B and p70S6K by Glucose and Insulin-like Growth Factor 1 in Pancreatic β-Cells (INS-1)

Differential Activation of Protein Kinase B and p70S6K by Glucose and Insulin-like Growth Factor 1 in Pancreatic Î²-Cells (INS-1)

Insulin Receptor Substrate-2 Proteasomal Degradation Mediated by a Mammalian Target of Rapamycin (mTOR)-induced Negative Feedback Down-regulates Protein Kinase B-mediated Signaling Pathway in Î²-Cells

Making Team Working Work - A Real Life Experience

Making Team Working Work-A Real Life Experience

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