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Coronavirus Proteins as Ion Channels: Current and Potential Research

Frontiers in immunology, 2020-10, Vol.11, p.573339-573339

2020

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Rapid Characterization of the Functional and Pharmacological Consequences of Cantú Syndrome K ATP Channel Mutations in Intact Cells

The Journal of pharmacology and experimental therapeutics, 2023-09, Vol.386 (3), p.298-309

2023

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ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9

Nature communications, 2019-10, Vol.10 (1), p.1-19, Article 4457

2019

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Skeletal muscle delimited myopathy and verapamil toxicity in SUR2 mutant mouse models of AIMS

EMBO molecular medicine, 2023-06, Vol.15 (6), p.e16883-n/a

2023

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Pathophysiological Consequences of KATP Channel Overactivity and Pharmacological Response to Glibenclamide in Skeletal Muscle of a Murine Model of Cantù Syndrome

Frontiers in pharmacology, 2020-11, Vol.11, p.604885-604885

2020

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A Unique High-Output Cardiac Hypertrophy Phenotype Arising From Low Systemic Vascular Resistance in Cantu Syndrome

Journal of the American Heart Association, 2022-12, Vol.11 (24), p.e027363-e027363

2022

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Zoledronic Acid Blocks Overactive Kir6.1/SUR2-Dependent KATP Channels in Skeletal Muscle and Osteoblasts in a Murine Model of Cantú Syndrome

Cells (Basel, Switzerland), 2023-03, Vol.12 (6), p.928

2023

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Consequences of SUR2[A478V] Mutation in Skeletal Muscle of Murine Model of Cantu Syndrome

Cells (Basel, Switzerland), 2021-07, Vol.10 (7), p.1791

2021

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Complex consequences of Cantu syndrome SUR2 variant R1154Q in genetically modified mice

JCI insight, 2021-03, Vol.6 (5)

2021

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Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets

Physiological reports, 2019-06, Vol.7 (11), p.e14101-n/a

2019

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Kir6.1 and SUR2B in Cantú syndrome

American Journal of Physiology: Cell Physiology, 2022-09, Vol.323 (3), p.C920-C935

2022

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GPR39 is coupled to TMEM16A in intestinal fibroblast-like cells

PloS one, 2012-10, Vol.7 (10), p.e47686-e47686

2012

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Pulmonary Hypertension and ATP-Sensitive Potassium Channels: Paradigms and Paradoxes

Hypertension (Dallas, Tex. 1979), 2019-07, Vol.74 (1), p.14-22

2019

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Mitochondrial Ca 2+ -coupled generation of reactive oxygen species causes endothelial dysfunction in Cantú syndrome

Physiology (Bethesda, Md.), 2024-05, Vol.39 (S1)

2024

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Polymodal activation of the TREK-2 K2P channel produces structurally distinct open states

The Journal of general physiology, 2016-06, Vol.147 (6), p.497-505

2016

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Endogenous currents in HEK 293 cells are inhibited by memantine

Nature chemical biology, 2023-11, Vol.19 (11), p.1303-1305

2023

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Glibenclamide reverses cardiovascular abnormalities of Cantu syndrome driven by KATP channel overactivity

The Journal of clinical investigation, 2020-03, Vol.130 (3), p.1116-1121

2020

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Conserved functional consequences of disease-associated mutations in the slide helix of Kir6.1 and Kir6.2 subunits of the ATP-sensitive potassium channel

The Journal of biological chemistry, 2017-10, Vol.292 (42), p.17387-17398

2017

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K2P channel gating mechanisms revealed by structures of TREK-2 and a complex with Prozac

Science (American Association for the Advancement of Science), 2015-03, Vol.347 (6227), p.1256-1259

2015

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Lymphedema as first clinical presentation of Cantu Syndrome: reversed phenotyping after identification of gain-of-function variant in ABCC9

European journal of human genetics : EJHG, 2023-02, Vol.31 (2), p.188-194

2023

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Biophysical and Pharmacological Characterization of Atp-Sensitive Potassium Channels in Mice Kir6.1WT/V65M Mirroring the Human Cantu′ Syndrome

Biophysical journal, 2020-02, Vol.118 (3), p.589a-590a

2020

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Mechanisms of TREK-2 Potassium Channel Gating

Biophysical journal, 2015-01, Vol.108 (2), p.437a-437a

2015

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KATP Channels in Zebrafish Cardiovascular System: A Model to Study Cantú Syndrome

Biophysical journal, 2020-02, Vol.118 (3), p.259a-259a

2020

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Pharmacological Approaches for Targeting Cardiovascular and Skeletal Muscle KATP Channelopathies

Biophysical journal, 2020-02, Vol.118 (3), p.590a-590a

2020

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Modulation of TMEM16B channel activity by the calcium-activated chloride channel regulator 4 (CLCA4) in human cells

The Journal of biological chemistry, 2024-07, Vol.300 (7), p.107432, Article 107432

2024

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Coronavirus Proteins as Ion Channels: Current and Potential Research

Rapid Characterization of the Functional and Pharmacological Consequences of Cantú Syndrome K ATP Channel Mutations in Intact Cells

ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9

Skeletal muscle delimited myopathy and verapamil toxicity in SUR2 mutant mouse models of AIMS

Pathophysiological Consequences of KATP Channel Overactivity and Pharmacological Response to Glibenclamide in Skeletal Muscle of a Murine Model of Cantù Syndrome

A Unique High-Output Cardiac Hypertrophy Phenotype Arising From Low Systemic Vascular Resistance in Cantu Syndrome

Zoledronic Acid Blocks Overactive Kir6.1/SUR2-Dependent KATP Channels in Skeletal Muscle and Osteoblasts in a Murine Model of Cantú Syndrome

Consequences of SUR2[A478V] Mutation in Skeletal Muscle of Murine Model of Cantu Syndrome

Complex consequences of Cantu syndrome SUR2 variant R1154Q in genetically modified mice

Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets

Kir6.1 and SUR2B in Cantú syndrome

GPR39 is coupled to TMEM16A in intestinal fibroblast-like cells

Pulmonary Hypertension and ATP-Sensitive Potassium Channels: Paradigms and Paradoxes

Mitochondrial Ca 2+ -coupled generation of reactive oxygen species causes endothelial dysfunction in Cantú syndrome

Polymodal activation of the TREK-2 K2P channel produces structurally distinct open states

Endogenous currents in HEK 293 cells are inhibited by memantine

Glibenclamide reverses cardiovascular abnormalities of Cantu syndrome driven by KATP channel overactivity

Conserved functional consequences of disease-associated mutations in the slide helix of Kir6.1 and Kir6.2 subunits of the ATP-sensitive potassium channel

K2P channel gating mechanisms revealed by structures of TREK-2 and a complex with Prozac

Lymphedema as first clinical presentation of Cantu Syndrome: reversed phenotyping after identification of gain-of-function variant in ABCC9

Biophysical and Pharmacological Characterization of Atp-Sensitive Potassium Channels in Mice Kir6.1WT/V65M Mirroring the Human Cantu′ Syndrome

Mechanisms of TREK-2 Potassium Channel Gating

KATP Channels in Zebrafish Cardiovascular System: A Model to Study Cantú Syndrome

Pharmacological Approaches for Targeting Cardiovascular and Skeletal Muscle KATP Channelopathies

Modulation of TMEM16B channel activity by the calcium-activated chloride channel regulator 4 (CLCA4) in human cells

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