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Pathogenicity and virulence of Clostridium perfringens

Virulence, 2021-12, Vol.12 (1), p.723-753

2021

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Expansion of the Clostridium perfringens toxin-based typing scheme

Anaerobe, 2018-10, Vol.53, p.5-10

2018

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Mechanisms of Action and Cell Death Associated with Clostridium perfringens Toxins

Toxins, 2018-05, Vol.10 (5), p.212

2018

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Clostridium perfringens Enterotoxin: Action, Genetics, and Translational Applications

Toxins, 2016-03, Vol.8 (3), p.73

2016

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Identification of orphan histidine kinases that impact sporulation and enterotoxin production by Clostridium perfringens type F strain SM101 in a pathophysiologically-relevant ex vivo mouse intestinal contents model

PLoS pathogens, 2023-06, Vol.19 (6), p.e1011429-e1011429

2023

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The Sialidases of Clostridium perfringens Type D Strain CN3718 Differ in Their Properties and Sensitivities to Inhibitors

Applied and Environmental Microbiology, 2014-03, Vol.80 (5), p.1701-1709

2014

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Toxin Plasmids of Clostridium perfringens

Microbiology and Molecular Biology Reviews, 2013-06, Vol.77 (2), p.208-233

2013

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Evidence That VirS Is a Receptor for the Signaling Peptide of the Clostridium perfringens Agr-like Quorum Sensing System

mBio, 2020-09, Vol.11 (5)

2020

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Contributions of NanI sialidase to Caco-2 cell adherence by Clostridium perfringens type A and C strains causing human intestinal disease

Infection and immunity, 2014-11, Vol.82 (11), p.4620-4630

2014

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Sialidases affect the host cell adherence and epsilon toxin-induced cytotoxicity of Clostridium perfringens type D strain CN3718

PLoS pathogens, 2011-12, Vol.7 (12), p.e1002429-e1002429

2011

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Role of the Agr-Like Quorum-Sensing System in Regulating Toxin Production by Clostridium perfringens Type B Strains CN1793 and CN1795

Infection and Immunity, 2012-09, Vol.80 (9), p.3008-3017

2012

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Agr-Like Quorum-Sensing System Regulates Sporulation and Production of Enterotoxin and Beta2 Toxin by Clostridium perfringens Type A Non-Food-Borne Human Gastrointestinal Disease Strain F5603

Infection and Immunity, 2011-06, Vol.79 (6), p.2451-2459

2011

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NanH Is Produced by Sporulating Cultures of Clostridium perfringens Type F Food Poisoning Strains and Enhances the Cytotoxicity of C. perfringens Enterotoxin

mSphere, 2021-04, Vol.6 (2)

2021

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The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation

mBio, 2024-04, Vol.15 (4), p.e0224823

2024

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RIP1, RIP3, and MLKL Contribute to Cell Death Caused by Clostridium perfringens Enterotoxin

mBio, 2019-12, Vol.10 (6)

2019

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Structure-Function Analysis of Peptide Signaling in the Clostridium perfringens Agr-Like Quorum Sensing System

Journal of bacteriology, 2015-05, Vol.197 (10), p.1807-1818

2015

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Evaluating the Involvement of Alternative Sigma Factors SigF and SigG in Clostridium perfringens Sporulation and Enterotoxin Synthesis

Infection and Immunity, 2010-10, Vol.78 (10), p.4286-4293

2010

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A novel small acid soluble protein variant is important for spore resistance of most Clostridium perfringens food poisoning isolates

PLoS pathogens, 2008-05, Vol.4 (5), p.e1000056

2008

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Characterization of NanR Regulation of Sialidase Production, Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strains Carrying a Chromosomal Enterotoxin Gene

Toxins, 2022-12, Vol.14 (12), p.872

2022

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Characterization of Toxin Plasmids in Clostridium perfringens Type C Isolates

Infection and Immunity, 2010-11, Vol.78 (11), p.4860-4869

2010

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Retraction for Chen et al., "Epsilon-Toxin Production by Clostridium perfringens Type D Strain CN3718 Is Dependent upon the agr Operon but Not the VirS/VirR Two-Component Regulatory System"

mBio, 2022-04, Vol.13 (2), p.e0049522-e0049522

2022

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Genotypic and Phenotypic Characterization of Clostridium perfringens Isolates from Darmbrand Cases in Post-World War II Germany

Infection and Immunity, 2012-12, Vol.80 (12), p.4354-4363

2012

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Overexpressing the cpr1953 Orphan Histidine Kinase Gene in the Absence of cpr1954 Orphan Histidine Kinase Gene Expression, or Vice Versa, Is Sufficient to Obtain Significant Sporulation and Strong Production of Clostridium perfringens Enterotoxin or Spo0A by Clostridium perfringens Type F Strain SM101

Toxins, 2024-04, Vol.16 (4), p.195

2024

Volltextzugriff (PDF)

Clostridium perfringens Sialidases: Potential Contributors to Intestinal Pathogenesis and Therapeutic Targets

Toxins, 2016-11, Vol.8 (11), p.341

2016

Volltextzugriff (PDF)

Human claudin-8 and -14 are receptors capable of conveying the cytotoxic effects of Clostridium perfringens enterotoxin

mBio, 2013-03, Vol.4 (1)

2013

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Pathogenicity and virulence of Clostridium perfringens

Expansion of the Clostridium perfringens toxin-based typing scheme

Mechanisms of Action and Cell Death Associated with Clostridium perfringens Toxins

Clostridium perfringens Enterotoxin: Action, Genetics, and Translational Applications

Identification of orphan histidine kinases that impact sporulation and enterotoxin production by Clostridium perfringens type F strain SM101 in a pathophysiologically-relevant ex vivo mouse intestinal contents model

The Sialidases of Clostridium perfringens Type D Strain CN3718 Differ in Their Properties and Sensitivities to Inhibitors

Toxin Plasmids of Clostridium perfringens

Evidence That VirS Is a Receptor for the Signaling Peptide of the Clostridium perfringens Agr-like Quorum Sensing System

Contributions of NanI sialidase to Caco-2 cell adherence by Clostridium perfringens type A and C strains causing human intestinal disease

Sialidases affect the host cell adherence and epsilon toxin-induced cytotoxicity of Clostridium perfringens type D strain CN3718

Role of the Agr-Like Quorum-Sensing System in Regulating Toxin Production by Clostridium perfringens Type B Strains CN1793 and CN1795

Agr-Like Quorum-Sensing System Regulates Sporulation and Production of Enterotoxin and Beta2 Toxin by Clostridium perfringens Type A Non-Food-Borne Human Gastrointestinal Disease Strain F5603

NanH Is Produced by Sporulating Cultures of Clostridium perfringens Type F Food Poisoning Strains and Enhances the Cytotoxicity of C. perfringens Enterotoxin

The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation

RIP1, RIP3, and MLKL Contribute to Cell Death Caused by Clostridium perfringens Enterotoxin

Structure-Function Analysis of Peptide Signaling in the Clostridium perfringens Agr-Like Quorum Sensing System

Evaluating the Involvement of Alternative Sigma Factors SigF and SigG in Clostridium perfringens Sporulation and Enterotoxin Synthesis

A novel small acid soluble protein variant is important for spore resistance of most Clostridium perfringens food poisoning isolates

Characterization of NanR Regulation of Sialidase Production, Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strains Carrying a Chromosomal Enterotoxin Gene

Characterization of Toxin Plasmids in Clostridium perfringens Type C Isolates

Retraction for Chen et al., "Epsilon-Toxin Production by Clostridium perfringens Type D Strain CN3718 Is Dependent upon the agr Operon but Not the VirS/VirR Two-Component Regulatory System"

Genotypic and Phenotypic Characterization of Clostridium perfringens Isolates from Darmbrand Cases in Post-World War II Germany

Clostridium perfringens Sialidases: Potential Contributors to Intestinal Pathogenesis and Therapeutic Targets

Human claudin-8 and -14 are receptors capable of conveying the cytotoxic effects of Clostridium perfringens enterotoxin

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