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Metal-induced delayed type hypersensitivity responses potentiate particle induced osteolysis in a sex and age dependent manner

PloS one, 2021-05, Vol.16 (5), p.e0251885-e0251885

2021

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Cobalt Alloy Implant Debris Induces Inflammation and Bone Loss Primarily through Danger Signaling, Not TLR4 Activation: Implications for DAMP-ening Implant Related Inflammation

PloS one, 2016-07, Vol.11 (7), p.e0160141

2016

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Soluble and particulate Co-Cr-Mo alloy implant metals activate the inflammasome danger signaling pathway in human macrophages: A novel mechanism for implant debris reactivity

Journal of orthopaedic research, 2009-07, Vol.27 (7), p.847-854

2009

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Increasing both CoCrMo-alloy particle size and surface irregularity induces increased macrophage inflammasome activation in vitro potentially through lysosomal destabilization mechanisms

Journal of orthopaedic research, 2013-10, Vol.31 (10), p.1633-1642

2013

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Do Battlefield Injury-acquired Indwelling Metal Fragments Induce Metal Immunogenicity?

Clinical orthopaedics and related research, 2020-04, Vol.478 (4), p.752-766

2020

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Orthopedic implant cobalt-alloy particles produce greater toxicity and inflammatory cytokines than titanium alloy and zirconium alloy-based particles in vitro, in human osteoblasts, fibroblasts, and macrophages

Journal of biomedical materials research. Part A, 2012-08, Vol.100A (8), p.2147-2158

2012

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Osteoclasts lose innate inflammatory reactivity to metal and polymer implant debris compared to monocytes/macrophages

The open orthopaedics journal, 2013-10, Vol.7 (1), p.605-613

2013

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TLR4 (not TLR2) dominate cognate TLR activity associated with CoCrMo implant particles

Journal of orthopaedic research, 2017-05, Vol.35 (5), p.1007-1017

2017

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Soluble ions more than particulate cobalt-alloy implant debris induce monocyte costimulatory molecule expression and release of proinflammatory cytokines critical to metal-induced lymphocyte reactivity

Journal of biomedical materials research. Part A, 2010-06, Vol.93A (4), p.1312-1321

2010

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Macrophage reactivity to different polymers demonstrates particle size- and material-specific reactivity: PEEK-OPTIMA® particles versus UHMWPE particles in the submicron, micron, and 10 micron size ranges

Journal of biomedical materials research. Part B, Applied biomaterials, 2012-02, Vol.100B (2), p.480-492

2012

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TLR4 (not TLR2) dominate cognate TLR activity associated with CoCrMo implant particles: PATHOGEN RECEPTOR DEPENDENT IMMUNE REACTIVITY TO IMPLANT PARTICLES

Journal of orthopaedic research, 2017-05, Vol.35 (5), p.1007-1017

2017

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In vitro reactivity to implant metals demonstrates a person-dependent association with both T-cell and B-cell activation

Journal of biomedical materials research. Part A, 2010-02, Vol.92A (2), p.667-682

2010

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Asymptomatic prospective and retrospective cohorts with metal-on-metal hip arthroplasty indicate acquired lymphocyte reactivity varies with metal ion levels on a group basis

Journal of orthopaedic research, 2013-02, Vol.31 (2), p.173-182

2013

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Do Battlefield Injury-acquired Indwelling Metal Fragments Induce Metal Immunogenicity?

Clinical orthopaedics and related research, 2019-09

2019

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CoCrMo alloy vs . UHMWPE Particulate Implant Debris Induces Sex Dependent Aseptic Osteolysis Responses In Vivo using a Murine Model

The open orthopaedics journal, 2018-03, Vol.12 (1), p.115-124

2018

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Methotrexate mitgates UHMWPE-induced inflammatory osteolysis in murine calvaria model, more than anti-TNF or anti-IL-1R treatments: Implications for extending implant lifetimes

Frontiers in bioengineering and biotechnology, 2016, Vol.4

2016

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Activated eosinophils elicit substance P release from cultured dorsal root ganglion neurons

American journal of physiology. Lung cellular and molecular physiology, 1997-11, Vol.273 (5), p.1096-L1102

1997

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Innate inflammatory cytokine response in periprosthetic tissue of failed metal-on-metal is significantly greater than failed metal-on-polyethylene THA implants

Frontiers in bioengineering and biotechnology, 2016, Vol.4

2016

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Augmentation of stimulated eosinophil degranulation by VLA-4 (CD49d)-mediated adhesion to fibronectin

American journal of respiratory cell and molecular biology, 1994-08, Vol.11 (2), p.206-213

1994

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Eosinophil VLA-4 binding to fibronectin augments bronchial narrowing through 5-lipoxygenase activation

American journal of physiology. Lung cellular and molecular physiology, 1996-04, Vol.270 (4), p.587-L594

1996

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Activated eosinophils elicit substance P release from cultured dorsal root ganglion neurons

American journal of physiology. Lung cellular and molecular physiology, 1997-11, Vol.273 (5), p.L1096

1997

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Metal-induced delayed type hypersensitivity responses potentiate particle induced osteolysis in a sex and age dependent manner

Cobalt Alloy Implant Debris Induces Inflammation and Bone Loss Primarily through Danger Signaling, Not TLR4 Activation: Implications for DAMP-ening Implant Related Inflammation

Soluble and particulate Co-Cr-Mo alloy implant metals activate the inflammasome danger signaling pathway in human macrophages: A novel mechanism for implant debris reactivity

Increasing both CoCrMo-alloy particle size and surface irregularity induces increased macrophage inflammasome activation in vitro potentially through lysosomal destabilization mechanisms

Do Battlefield Injury-acquired Indwelling Metal Fragments Induce Metal Immunogenicity?

Orthopedic implant cobalt-alloy particles produce greater toxicity and inflammatory cytokines than titanium alloy and zirconium alloy-based particles in vitro, in human osteoblasts, fibroblasts, and macrophages

Osteoclasts lose innate inflammatory reactivity to metal and polymer implant debris compared to monocytes/macrophages

TLR4 (not TLR2) dominate cognate TLR activity associated with CoCrMo implant particles

Soluble ions more than particulate cobalt-alloy implant debris induce monocyte costimulatory molecule expression and release of proinflammatory cytokines critical to metal-induced lymphocyte reactivity

Macrophage reactivity to different polymers demonstrates particle size- and material-specific reactivity: PEEK-OPTIMA® particles versus UHMWPE particles in the submicron, micron, and 10 micron size ranges

TLR4 (not TLR2) dominate cognate TLR activity associated with CoCrMo implant particles: PATHOGEN RECEPTOR DEPENDENT IMMUNE REACTIVITY TO IMPLANT PARTICLES

In vitro reactivity to implant metals demonstrates a person-dependent association with both T-cell and B-cell activation

Asymptomatic prospective and retrospective cohorts with metal-on-metal hip arthroplasty indicate acquired lymphocyte reactivity varies with metal ion levels on a group basis

Do Battlefield Injury-acquired Indwelling Metal Fragments Induce Metal Immunogenicity?

CoCrMo alloy vs . UHMWPE Particulate Implant Debris Induces Sex Dependent Aseptic Osteolysis Responses In Vivo using a Murine Model

Methotrexate mitgates UHMWPE-induced inflammatory osteolysis in murine calvaria model, more than anti-TNF or anti-IL-1R treatments: Implications for extending implant lifetimes

Activated eosinophils elicit substance P release from cultured dorsal root ganglion neurons

Innate inflammatory cytokine response in periprosthetic tissue of failed metal-on-metal is significantly greater than failed metal-on-polyethylene THA implants

Augmentation of stimulated eosinophil degranulation by VLA-4 (CD49d)-mediated adhesion to fibronectin

Eosinophil VLA-4 binding to fibronectin augments bronchial narrowing through 5-lipoxygenase activation

Activated eosinophils elicit substance P release from cultured dorsal root ganglion neurons

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