UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Transforming growth factor-beta and fibrosis

World journal of gastroenterology : WJG, 2007-06, Vol.13 (22), p.3056

2007

Volltextzugriff (PDF)

Crosstalk mechanisms between the mitogen-activated protein kinase pathways and Smad signaling downstream of TGF-beta: implications for carcinogenesis

Oncogene, 2005-08, Vol.24 (37), p.5742-5750

2005

Volltextzugriff (PDF)

TGF-β-induced SMAD signaling and gene regulation: consequences for extracellular matrix remodeling and wound healing

Journal of dermatological science, 2004-08, Vol.35 (2), p.83-92

2004

Volltextzugriff (PDF)

Pro-Invasive Activity of the Hippo Pathway Effectors YAP and TAZ in Cutaneous Melanoma

Journal of investigative dermatology, 2014-01, Vol.134 (1), p.123-132

2014

Volltextzugriff (PDF)

Large-scale pan-cancer analysis reveals broad prognostic association between TGF-β ligands, not Hedgehog, and GLI1/2 expression in tumors

Scientific reports, 2020-09, Vol.10 (1), p.14491-14491, Article 14491

2020

Volltextzugriff (PDF)

Transcriptional repression of the tyrosinase-related protein 2 gene by transforming growth factor-β and the Kruppel-like transcription factor GLI2

Journal of dermatological science, 2019-06, Vol.94 (3), p.321-329

2019

Volltextzugriff (PDF)

Insights into the Transforming Growth Factor-β Signaling Pathway in Cutaneous Melanoma

Annals of Dermatology, 2013, 25(2), , pp.135-144

2013

Volltextzugriff (PDF)

Systematic classification of melanoma cells by phenotype-specific gene expression mapping

Pigment cell and melanoma research, 2012-05, Vol.25 (3), p.343-353

2012

Volltextzugriff (PDF)

GLI2-Mediated Melanoma Invasion and Metastasis

JNCI : Journal of the National Cancer Institute, 2010-08, Vol.102 (15), p.1148-1159

2010

Volltextzugriff (PDF)

Stable overexpression of Smad7 in human melanoma cells impairs bone metastasis

Cancer research (Chicago, Ill.), 2007-03, Vol.67 (5), p.2317-2324

2007

Volltextzugriff (PDF)

SMAD2/3 mediate oncogenic effects of TGF-β in the absence of SMAD4

Communications biology, 2022-10, Vol.5 (1), p.1068-13, Article 1068

2022

Volltextzugriff (PDF)

Halofuginone Inhibits the Establishment and Progression of Melanoma Bone Metastases

Cancer research (Chicago, Ill.), 2012-12, Vol.72 (23), p.6247-6256

2012

Volltextzugriff (PDF)

In vitro evidence for a direct antifibrotic role of the immunosuppressive drug mycophenolate mofetil

The Journal of pharmacology and experimental therapeutics, 2007-05, Vol.321 (2), p.583-589

2007

Volltextzugriff (PDF)

Cytokine regulation of metalloproteinase gene expression

Journal of cellular biochemistry, 1993-12, Vol.53 (4), p.288-295

Mauviel, Alain

1993

Volltextzugriff (PDF)

JNK supports survival in melanoma cells by controlling cell cycle arrest and apoptosis

Pigment cell and melanoma research, 2008-08, Vol.21 (4), p.429-438

2008

Volltextzugriff (PDF)

Yes-associated protein (YAP65) interacts with Smad7 and potentiates its inhibitory activity against TGF-β/Smad signaling

Oncogene, 2002-07, Vol.21 (32), p.4879-4884

2002

Volltextzugriff (PDF)

Cyclic adenosine 3', 5'-monophosphate-elevating agents inhibit transforming growth factor-β-induced SMAD3/4-dependent transcription via a protein kinase A-dependent mechanism

Oncogene, 2003-12, Vol.22 (55), p.8881-8890

2003

Volltextzugriff (PDF)

Stable overexpression of Smad7 in human melanoma cells inhibits their tumorigenicity in vitro and in vivo

Oncogene, 2005-11, Vol.24 (51), p.7624-7629

2005

Volltextzugriff (PDF)

Efficient TGF-β/SMAD signaling in human melanoma cells associated with high c-SKI/SnoN expression

Molecular cancer, 2011-01, Vol.10 (1), p.2-2, Article 2

2011

Volltextzugriff (PDF)

Disruption of Basal JNK Activity Differentially Affects Key Fibroblast Functions Important for Wound Healing

The Journal of biological chemistry, 2003-07, Vol.278 (27), p.24624-24628

2003

Volltextzugriff (PDF)

Blocking Sp1 Transcription Factor Broadly Inhibits Extracellular Matrix Gene Expression In Vitro and In Vivo: Implications for the Treatment of Tissue Fibrosis

Journal of investigative dermatology, 2001-05, Vol.116 (5), p.755-763

2001

Volltextzugriff (PDF)

Molecular mechanisms underlying TGF-ß/Hippo signaling crosstalks – Role of baso-apical epithelial cell polarity

The international journal of biochemistry & cell biology, 2018-05, Vol.98, p.75-81

2018

Volltextzugriff (PDF)

Smad3/AP-1 interactions control transcriptional responses to TGF-β in a promoter-specific manner

Oncogene, 2001-06, Vol.20 (26), p.3332-3340

2001

Volltextzugriff (PDF)

Cytoplasmic SnoN in Normal Tissues and Nonmalignant Cells Antagonizes TGF-β Signaling by Sequestration of the Smad Proteins

Proceedings of the National Academy of Sciences - PNAS, 2005-08, Vol.102 (35), p.12437-12442

2005

Volltextzugriff (PDF)

Retinoic acid receptors interfere with the TGF-β/Smad signaling pathway in a ligand-specific manner

Oncogene, 2003-11, Vol.22 (50), p.8212-8220

2003

Volltextzugriff (PDF)

1 – 25 von 184

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(176)
Mit Volltextzugriff
(179)

Publikationsform

Sprache

Erscheinungsjahr

n.n

Thema

Humans
(104)
Science & Technology
(99)
Life Sciences & Biomedicine
(96)
Animals
(46)
Biological And Medical Sciences
(41)
Biochemistry & Molecular Biology
(38)
Signal Transduction
(37)
Dermatology
(34)
Life Sciences
(33)
Cell Biology
(32)
Mice
(32)
Cells, Cultured
(31)
Oncology
(30)
Cancer
(29)
Gene Expression
(26)
Medical Sciences
(24)
Transforming Growth Factor Beta - Metabolism
(20)
Transforming Growth Factor Beta - Pharmacology
(20)
Fibroblasts - Metabolism
(19)
Fundamental And Applied Biological Sciences. Psychology
(19)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Transforming growth factor-beta and fibrosis

Crosstalk mechanisms between the mitogen-activated protein kinase pathways and Smad signaling downstream of TGF-beta: implications for carcinogenesis

TGF-β-induced SMAD signaling and gene regulation: consequences for extracellular matrix remodeling and wound healing

Pro-Invasive Activity of the Hippo Pathway Effectors YAP and TAZ in Cutaneous Melanoma

Large-scale pan-cancer analysis reveals broad prognostic association between TGF-β ligands, not Hedgehog, and GLI1/2 expression in tumors

Transcriptional repression of the tyrosinase-related protein 2 gene by transforming growth factor-β and the Kruppel-like transcription factor GLI2

Insights into the Transforming Growth Factor-β Signaling Pathway in Cutaneous Melanoma

Systematic classification of melanoma cells by phenotype-specific gene expression mapping

GLI2-Mediated Melanoma Invasion and Metastasis

Stable overexpression of Smad7 in human melanoma cells impairs bone metastasis

SMAD2/3 mediate oncogenic effects of TGF-β in the absence of SMAD4

Halofuginone Inhibits the Establishment and Progression of Melanoma Bone Metastases

In vitro evidence for a direct antifibrotic role of the immunosuppressive drug mycophenolate mofetil

Cytokine regulation of metalloproteinase gene expression

JNK supports survival in melanoma cells by controlling cell cycle arrest and apoptosis

Yes-associated protein (YAP65) interacts with Smad7 and potentiates its inhibitory activity against TGF-β/Smad signaling

Cyclic adenosine 3', 5'-monophosphate-elevating agents inhibit transforming growth factor-β-induced SMAD3/4-dependent transcription via a protein kinase A-dependent mechanism

Stable overexpression of Smad7 in human melanoma cells inhibits their tumorigenicity in vitro and in vivo

Efficient TGF-β/SMAD signaling in human melanoma cells associated with high c-SKI/SnoN expression

Disruption of Basal JNK Activity Differentially Affects Key Fibroblast Functions Important for Wound Healing

Blocking Sp1 Transcription Factor Broadly Inhibits Extracellular Matrix Gene Expression In Vitro and In Vivo: Implications for the Treatment of Tissue Fibrosis

Molecular mechanisms underlying TGF-ß/Hippo signaling crosstalks – Role of baso-apical epithelial cell polarity

Smad3/AP-1 interactions control transcriptional responses to TGF-β in a promoter-specific manner

Cytoplasmic SnoN in Normal Tissues and Nonmalignant Cells Antagonizes TGF-β Signaling by Sequestration of the Smad Proteins

Retinoic acid receptors interfere with the TGF-β/Smad signaling pathway in a ligand-specific manner

Suchergebnisse filtern