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Activation of NF-kappa B signaling promotes growth of prostate cancer cells in bone

PloS one, 2013-04, Vol.8 (4), p.e60983

2013

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KDM5B Is Essential for the Hyperactivation of PI3K/AKT Signaling in Prostate Tumorigenesis

Cancer research (Chicago, Ill.), 2020-11, Vol.80 (21), p.4633-4643

2020

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Mouse models of prostate cancer: picking the best model for the question

Cancer and metastasis reviews, 2014-09, Vol.33 (2-3), p.377-397

2014

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MicroRNA-21 deficiency suppresses prostate cancer progression through downregulation of the IRS1-SREBP-1 signaling pathway

Cancer letters, 2022-01, Vol.525, p.46-54

2022

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Loss of the urothelial differentiation marker FOXA1 is associated with high grade, late stage bladder cancer and increased tumor proliferation

PloS one, 2012-05, Vol.7 (5), p.e36669-e36669

2012

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Fetuin-A Promotes 3-Dimensional Growth in LNCaP Prostate Cancer Cells by Sequestering Extracellular Vesicles to Their Surfaces to Act as Signaling Platforms

International journal of molecular sciences, 2022-04, Vol.23 (7), p.4031

2022

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Role of Androgen Receptor Variants in Prostate Cancer: Report from the 2017 Mission Androgen Receptor Variants Meeting

European urology, 2018-05, Vol.73 (5), p.715-723

2018

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Hepsin promotes prostate cancer progression and metastasis

Cancer cell, 2004-08, Vol.6 (2), p.185-195

2004

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NF-κB Gene Signature Predicts Prostate Cancer Progression

Cancer research (Chicago, Ill.), 2014-05, Vol.74 (10), p.2763-2772

2014

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Identification of Genes Required for Enzalutamide Resistance in Castration-Resistant Prostate Cancer Cells In Vitro

Molecular cancer therapeutics, 2021-02, Vol.20 (2), p.398-409

2021

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Skp2 regulates androgen receptor through ubiquitin-mediated degradation independent of Akt/mTOR pathways in prostate cancer

The Prostate, 2014-04, Vol.74 (4), p.421-432

2014

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Transcriptomic analysis of benign prostatic hyperplasia identifies critical pathways in prostatic overgrowth and 5-alpha reductase inhibitor resistance

The Prostate, 2024-04, Vol.84 (5), p.441-459

2024

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Lysosome-dependent FOXA1 ubiquitination contributes to luminal lineage of advanced prostate cancer

Molecular oncology, 2023-10, Vol.17 (10), p.2126-2146

2023

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Bone Metastasis of Prostate Cancer Can Be Therapeutically Targeted at the TBX2-WNT Signaling Axis

Cancer research (Chicago, Ill.), 2017-03, Vol.77 (6), p.1331-1344

2017

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Loss of FOXA1 Drives Sexually Dimorphic Changes in Urothelial Differentiation and Is an Independent Predictor of Poor Prognosis in Bladder Cancer

The American journal of pathology, 2015-05, Vol.185 (5), p.1385-1395

2015

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Glucocorticoids are induced while dihydrotestosterone levels are suppressed in 5‐alpha reductase inhibitor treated human benign prostate hyperplasia patients

The Prostate, 2022-10, Vol.82 (14), p.1378-1388

2022

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ARF Represses Androgen Receptor Transactivation in Prostate Cancer

Molecular endocrinology (Baltimore, Md.), 2013-04, Vol.27 (4), p.635-648

2013

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Slug regulates E-cadherin repression via p19Arf in prostate tumorigenesis

Molecular oncology, 2014-10, Vol.8 (7), p.1355-1364

2014

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Therapy-induced small-cell disease: from mouse to man and back

Nature reviews. Urology, 2018-11, Vol.15 (11), p.662-663

2018

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When urothelial differentiation pathways go wrong: Implications for bladder cancer development and progression

Urologic oncology, 2013-08, Vol.31 (6), p.802-811

2013

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Prostatic osteopontin expression is associated with symptomatic benign prostatic hyperplasia

The Prostate, 2020-07, Vol.80 (10), p.731-741

2020

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NFI Transcription Factors Interact with FOXA1 to Regulate Prostate-Specific Gene Expression

Molecular endocrinology (Baltimore, Md.), 2014-06, Vol.28 (6), p.949-964

2014

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The prostaglandin pathway is activated in patients who fail medical therapy for benign prostatic hyperplasia with lower urinary tract symptoms

The Prostate, 2021-09, Vol.81 (13), p.944-955

2021

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NF-κB and androgen receptor variant expression correlate with human BPH progression

The Prostate, 2016-04, Vol.76 (5), p.491-511

2016

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FOXA1 deletion in luminal epithelium causes prostatic hyperplasia and alteration of differentiated phenotype

Laboratory investigation, 2014-07, Vol.94 (7), p.726-739

2014

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Activation of NF-kappa B signaling promotes growth of prostate cancer cells in bone

KDM5B Is Essential for the Hyperactivation of PI3K/AKT Signaling in Prostate Tumorigenesis

Mouse models of prostate cancer: picking the best model for the question

MicroRNA-21 deficiency suppresses prostate cancer progression through downregulation of the IRS1-SREBP-1 signaling pathway

Loss of the urothelial differentiation marker FOXA1 is associated with high grade, late stage bladder cancer and increased tumor proliferation

Fetuin-A Promotes 3-Dimensional Growth in LNCaP Prostate Cancer Cells by Sequestering Extracellular Vesicles to Their Surfaces to Act as Signaling Platforms

Role of Androgen Receptor Variants in Prostate Cancer: Report from the 2017 Mission Androgen Receptor Variants Meeting

Hepsin promotes prostate cancer progression and metastasis

NF-κB Gene Signature Predicts Prostate Cancer Progression

Identification of Genes Required for Enzalutamide Resistance in Castration-Resistant Prostate Cancer Cells In Vitro

Skp2 regulates androgen receptor through ubiquitin-mediated degradation independent of Akt/mTOR pathways in prostate cancer

Transcriptomic analysis of benign prostatic hyperplasia identifies critical pathways in prostatic overgrowth and 5-alpha reductase inhibitor resistance

Lysosome-dependent FOXA1 ubiquitination contributes to luminal lineage of advanced prostate cancer

Bone Metastasis of Prostate Cancer Can Be Therapeutically Targeted at the TBX2-WNT Signaling Axis

Loss of FOXA1 Drives Sexually Dimorphic Changes in Urothelial Differentiation and Is an Independent Predictor of Poor Prognosis in Bladder Cancer

Glucocorticoids are induced while dihydrotestosterone levels are suppressed in 5‐alpha reductase inhibitor treated human benign prostate hyperplasia patients

ARF Represses Androgen Receptor Transactivation in Prostate Cancer

Slug regulates E-cadherin repression via p19Arf in prostate tumorigenesis

Therapy-induced small-cell disease: from mouse to man and back

When urothelial differentiation pathways go wrong: Implications for bladder cancer development and progression

Prostatic osteopontin expression is associated with symptomatic benign prostatic hyperplasia

NFI Transcription Factors Interact with FOXA1 to Regulate Prostate-Specific Gene Expression

The prostaglandin pathway is activated in patients who fail medical therapy for benign prostatic hyperplasia with lower urinary tract symptoms

NF-κB and androgen receptor variant expression correlate with human BPH progression

FOXA1 deletion in luminal epithelium causes prostatic hyperplasia and alteration of differentiated phenotype

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