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Molecular mechanisms of Saccharomyces cerevisiae stress adaptation and programmed cell death in response to acetic acid

Frontiers in microbiology, 2013, Vol.4, p.33-33

2013

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Production of Reactive Oxygen Species, Alteration of Cytosolic Ascorbate Peroxidase, and Impairment of Mitochondrial Metabolism Are Early Events in Heat Shock-Induced Programmed Cell Death in Tobacco Bright-Yellow 2 Cells

Plant physiology (Bethesda), 2004-03, Vol.134 (3), p.1100-1112

2004

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Yeast growth in raffinose results in resistance to acetic-acid induced programmed cell death mostly due to the activation of the mitochondrial retrograde pathway

Biochimica et biophysica acta, 2013-12, Vol.1833 (12), p.2765-2774

2013

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Cytochrome c Is Released in a Reactive Oxygen Species-Dependent Manner and Is Degraded via Caspase-Like Proteases in Tobacco Bright-Yellow 2 Cells en Route to Heat Shock-Induced Cell Death

Plant physiology (Bethesda), 2006-05, Vol.141 (1), p.208-219

2006

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Glutamate neurotoxicity, oxidative stress and mitochondria

FEBS Letters, 2001-05, Vol.497 (1), p.1-5

2001

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Acid stress adaptation protects Saccharomyces cerevisiae from acetic acid-induced programmed cell death

Gene, 2005-07, Vol.354, p.93-98

2005

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The role of mitochondria in yeast programmed cell death

Frontiers in oncology, 2012, Vol.2, p.70-70

2012

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Loss of BRCA2 promotes prostate cancer cell invasion through up‐regulation of matrix metalloproteinase‐9

Cancer science, 2008-03, Vol.99 (3), p.553-563

2008

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Cytochrome c Is Released from Mitochondria in a Reactive Oxygen Species (ROS)-dependent Fashion and Can Operate as a ROS Scavenger and as a Respiratory Substrate in Cerebellar Neurons Undergoing Excitotoxic Death

The Journal of biological chemistry, 2000-11, Vol.275 (47), p.37159-37166

2000

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Up-regulation of Skp2 after Prostate Cancer Cell Adhesion to Basement Membranes Results in BRCA2 Degradation and Cell Proliferation

The Journal of biological chemistry, 2006-08, Vol.281 (31), p.22100-22107

2006

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The N-acetylcysteine-insensitive acetic acid-induced yeast programmed cell death occurs without macroautophagy

Current pharmaceutical biotechnology, 2012-12, Vol.13 (15), p.2705

2012

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Mitochondrial DNA depletion reduces PARP-1 levels and promotes progression of the neoplastic phenotype in prostate carcinoma

Cellular oncology : the official journal of the International Society for Cellular Oncology, 2008, Vol.30 (4), p.307-322

2008

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Alzheimer's Proteins, Oxidative Stress, and Mitochondrial Dysfunction Interplay in a Neuronal Model of Alzheimer's Disease

International journal of alzheimer's disease, 2010-09, Vol.2010, p.1-11

2010

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An increase in the ATP levels occurs in cerebellar granule cells en route to apoptosis in which ATP derives from both oxidative phosphorylation and anaerobic glycolysis

Biochimica et biophysica acta, 2005-06, Vol.1708 (1), p.50-62

2005

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Proteasome function is required for activation of programmed cell death in heat shocked tobacco Bright-Yellow 2 cells

FEBS letters, 2007-03, Vol.581 (5), p.917-922

2007

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Down-regulation of BRCA2 Expression by Collagen Type I Promotes Prostate Cancer Cell Proliferation

The Journal of biological chemistry, 2005-06, Vol.280 (23), p.22482-22491

2005

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Thyroid Hormone Treatment of Hypothyroid Rats Restores the Regenerative Capacity and the Mitochondrial Membrane Permeability Properties of the Liver after Partial Hepatectomy

Endocrinology (Philadelphia), 2004-11, Vol.145 (11), p.5121-5128

2004

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Deficit of complex I activity in human skin fibroblasts with chromosome 21 trisomy and overproduction of reactive oxygen species by mitochondria: involvement of the cAMP/PKA signalling pathway

Biochemical journal, 2011-05, Vol.435 (3), p.679-688

2011

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Yeast acetic acid-induced programmed cell death can occur without cytochrome c release which requires metacaspase YCA1

FEBS letters, 2010-01, Vol.584 (1), p.224-228

2010

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Knock-out of metacaspase and/or cytochrome c results in the activation of a ROS-independent acetic acid-induced programmed cell death pathway in yeast

FEBS letters, 2010-08, Vol.584 (16), p.3655-3660

2010

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L-lactate metabolism can occur in normal and cancer prostate cells via the novel mitochondrial L-lactate dehydrogenase

International journal of oncology, 2010-12, Vol.37 (6), p.1607-1620

2010

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Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death

Apoptosis (London), 2014-09, Vol.19 (9), p.1330-1341

2014

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Up-regulation of Skp2 after prostate cancer cell adhesion to basement membranes results in BRCA2 degradation and cell proliferation

The Journal of biological chemistry, 2014-06, Vol.289 (25), p.17424-17424

2014

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Down-regulation of BRCA2 expression by collagen type I promotes prostate cancer cell proliferation

The Journal of biological chemistry, 2014-06, Vol.289 (25), p.17423-17423

2014

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Impairment of F1F0-ATPase, adenine nucleotide translocator and adenylate kinase causes mitochondrial energy deficit in human skin fibroblasts with chromosome 21 trisomy

Biochemical journal, 2010-10, Vol.431 (2), p.299-310

2010

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Molecular mechanisms of Saccharomyces cerevisiae stress adaptation and programmed cell death in response to acetic acid

Production of Reactive Oxygen Species, Alteration of Cytosolic Ascorbate Peroxidase, and Impairment of Mitochondrial Metabolism Are Early Events in Heat Shock-Induced Programmed Cell Death in Tobacco Bright-Yellow 2 Cells

Yeast growth in raffinose results in resistance to acetic-acid induced programmed cell death mostly due to the activation of the mitochondrial retrograde pathway

Cytochrome c Is Released in a Reactive Oxygen Species-Dependent Manner and Is Degraded via Caspase-Like Proteases in Tobacco Bright-Yellow 2 Cells en Route to Heat Shock-Induced Cell Death

Glutamate neurotoxicity, oxidative stress and mitochondria

Acid stress adaptation protects Saccharomyces cerevisiae from acetic acid-induced programmed cell death

The role of mitochondria in yeast programmed cell death

Loss of BRCA2 promotes prostate cancer cell invasion through up‐regulation of matrix metalloproteinase‐9

Cytochrome c Is Released from Mitochondria in a Reactive Oxygen Species (ROS)-dependent Fashion and Can Operate as a ROS Scavenger and as a Respiratory Substrate in Cerebellar Neurons Undergoing Excitotoxic Death

Up-regulation of Skp2 after Prostate Cancer Cell Adhesion to Basement Membranes Results in BRCA2 Degradation and Cell Proliferation

The N-acetylcysteine-insensitive acetic acid-induced yeast programmed cell death occurs without macroautophagy

Mitochondrial DNA depletion reduces PARP-1 levels and promotes progression of the neoplastic phenotype in prostate carcinoma

Alzheimer's Proteins, Oxidative Stress, and Mitochondrial Dysfunction Interplay in a Neuronal Model of Alzheimer's Disease

An increase in the ATP levels occurs in cerebellar granule cells en route to apoptosis in which ATP derives from both oxidative phosphorylation and anaerobic glycolysis

Proteasome function is required for activation of programmed cell death in heat shocked tobacco Bright-Yellow 2 cells

Down-regulation of BRCA2 Expression by Collagen Type I Promotes Prostate Cancer Cell Proliferation

Thyroid Hormone Treatment of Hypothyroid Rats Restores the Regenerative Capacity and the Mitochondrial Membrane Permeability Properties of the Liver after Partial Hepatectomy

Deficit of complex I activity in human skin fibroblasts with chromosome 21 trisomy and overproduction of reactive oxygen species by mitochondria: involvement of the cAMP/PKA signalling pathway

Yeast acetic acid-induced programmed cell death can occur without cytochrome c release which requires metacaspase YCA1

Knock-out of metacaspase and/or cytochrome c results in the activation of a ROS-independent acetic acid-induced programmed cell death pathway in yeast

L-lactate metabolism can occur in normal and cancer prostate cells via the novel mitochondrial L-lactate dehydrogenase

Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death

Up-regulation of Skp2 after prostate cancer cell adhesion to basement membranes results in BRCA2 degradation and cell proliferation

Down-regulation of BRCA2 expression by collagen type I promotes prostate cancer cell proliferation

Impairment of F1F0-ATPase, adenine nucleotide translocator and adenylate kinase causes mitochondrial energy deficit in human skin fibroblasts with chromosome 21 trisomy

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