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RNA methyltransferase METTL16: Targets and function

Wiley interdisciplinary reviews. RNA, 2022-03, Vol.13 (2), p.e1681-n/a

2022

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Characterization of METTL16 as a cytoplasmic RNA binding protein

PloS one, 2020-01, Vol.15 (1), p.e0227647-e0227647

2020

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RNA Binding by the m6A Methyltransferases METTL16 and METTL3

Biology (Basel, Switzerland), 2024-05, Vol.13 (6), p.391

Mansfield, Kyle D.

2024

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Neuron-specific ELAV/Hu proteins suppress HuR mRNA during neuronal differentiation by alternative polyadenylation

Nucleic acids research, 2012-03, Vol.40 (6), p.2734

2012

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N 6 -methyladenosine is required for the hypoxic stabilization of specific mRNAs

RNA (Cambridge), 2017-09, Vol.23 (9), p.1444-1455

2017

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Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing

Cell metabolism, 2005-06, Vol.1 (6), p.401-408

2005

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RNA Methyltransferase METTL16's Protein Domains Have Differential Functional Effects on Cell Processes

Current issues in molecular biology, 2023-06, Vol.45 (7), p.5460-5480

2023

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m6A regulates breast cancer proliferation and migration through stage-dependent changes in Epithelial to Mesenchymal Transition gene expression

Frontiers in oncology, 2023-11, Vol.13, p.1268977-1268977

2023

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Multiple Factors Affecting Cellular Redox Status and Energy Metabolism Modulate Hypoxia-Inducible Factor Prolyl Hydroxylase Activity In Vivo and In Vitro

Molecular and Cellular Biology, 2007-02, Vol.27 (3), p.912-925

2007

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Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-α prolyl hydroxylase

Cancer cell, 2005, Vol.7 (1), p.77-85

2005

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Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-α activation

Cell metabolism, 2005-06, Vol.1 (6), p.393-399

2005

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Hypoxic stabilization of mRNA is HIF-independent but requires mtROS

Cellular & molecular biology letters, 2018-10, Vol.23 (1), p.48-48, Article 48

2018

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The ribonome: a dominant force in co-ordinating gene expression

Biology of the cell, 2009-03, Vol.101 (3), p.169-181

2009

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N6-methyladenosine is required for the hypoxic stabilization of specific mRNAs

RNA (Cambridge), 2017-09, Vol.23 (9), p.1444-1455

2017

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Acetylation of p53 Activates Transcription through Recruitment of Coactivators/Histone Acetyltransferases

Molecular cell, 2001-12, Vol.8 (6), p.1243-1254

2001

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Hypoxia and Hypoglycemia synergistically regulate mRNA stability

RNA biology, 2017-07, Vol.14 (7), p.938-951

2017

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N6-methyladenosine contributes to cellular phenotype in a genetically-defined model of breast cancer progression

Oncotarget, 2018-07, Vol.9 (58), p.31231-31243

2018

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Hypoxic reduction in cellular glutathione levels requires mitochondrial reactive oxygen species

Journal of applied physiology (1985), 2004-10, Vol.97 (4), p.1358-1366

2004

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Development of the terminally differentiated state sensitizes epiphyseal chondrocytes to apoptosis through caspase-3 activation

Journal of cellular physiology, 2007-03, Vol.210 (3), p.609-615

2007

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Implementation of a 2D Bond Based Peridynamic Fatigue Model in FEM

Mansfield, Kyle

2020

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ProQuest

Phosphate-induced chondrocyte apoptosis is linked to nitric oxide generation

American Journal of Physiology: Cell Physiology, 2001-09, Vol.281 (3), p.C833-C839

2001

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Matrix Regulation of Skeletal Cell Apoptosis II: Role of Arg‐Gly‐Asp‐Containing Peptides

Journal of bone and mineral research, 2002-01, Vol.17 (1), p.66-76

2002

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N^sup 6^-methyladenosine is required for the hypoxic stabilization of specific mRNAs

RNA (Cambridge), 2017-09, Vol.23 (9), p.1444

2017

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Chondrocyte death is linked to development of a mitochondrial membrane permeability transition in the growth plate

Journal of cellular physiology, 1999-06, Vol.179 (3), p.287-296

1999

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Matrix regulation of skeletal cell apoptosis. Role of calcium and phosphate ions

The Journal of biological chemistry, 2001-06, Vol.276 (23), p.20316-20322

2001

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RNA methyltransferase METTL16: Targets and function

Characterization of METTL16 as a cytoplasmic RNA binding protein

RNA Binding by the m6A Methyltransferases METTL16 and METTL3

Neuron-specific ELAV/Hu proteins suppress HuR mRNA during neuronal differentiation by alternative polyadenylation

N 6 -methyladenosine is required for the hypoxic stabilization of specific mRNAs

Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing

RNA Methyltransferase METTL16's Protein Domains Have Differential Functional Effects on Cell Processes

m6A regulates breast cancer proliferation and migration through stage-dependent changes in Epithelial to Mesenchymal Transition gene expression

Multiple Factors Affecting Cellular Redox Status and Energy Metabolism Modulate Hypoxia-Inducible Factor Prolyl Hydroxylase Activity In Vivo and In Vitro

Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-α prolyl hydroxylase

Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-α activation

Hypoxic stabilization of mRNA is HIF-independent but requires mtROS

The ribonome: a dominant force in co-ordinating gene expression

N6-methyladenosine is required for the hypoxic stabilization of specific mRNAs

Acetylation of p53 Activates Transcription through Recruitment of Coactivators/Histone Acetyltransferases

Hypoxia and Hypoglycemia synergistically regulate mRNA stability

N6-methyladenosine contributes to cellular phenotype in a genetically-defined model of breast cancer progression

Hypoxic reduction in cellular glutathione levels requires mitochondrial reactive oxygen species

Development of the terminally differentiated state sensitizes epiphyseal chondrocytes to apoptosis through caspase-3 activation

Implementation of a 2D Bond Based Peridynamic Fatigue Model in FEM

Phosphate-induced chondrocyte apoptosis is linked to nitric oxide generation

Matrix Regulation of Skeletal Cell Apoptosis II: Role of Arg‐Gly‐Asp‐Containing Peptides

N^sup 6^-methyladenosine is required for the hypoxic stabilization of specific mRNAs

Chondrocyte death is linked to development of a mitochondrial membrane permeability transition in the growth plate

Matrix regulation of skeletal cell apoptosis. Role of calcium and phosphate ions

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