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From Estrogen-Centric to Aging and Oxidative Stress: A Revised Perspective of the Pathogenesis of Osteoporosis

Endocrine reviews, 2010-06, Vol.31 (3), p.266-300

Manolagas, Stavros C

2010

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Osteocalcin promotes bone mineralization but is not a hormone

PLoS genetics, 2020-06, Vol.16 (6), p.e1008714-e1008714

2020

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The Quest for Osteoporosis Mechanisms and Rational Therapies: How Far We've Come, How Much Further We Need to Go

Journal of bone and mineral research, 2018-03, Vol.33 (3), p.371-385

Manolagas, Stavros C

2018

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The role of estrogen and androgen receptors in bone health and disease

Nature reviews. Endocrinology, 2013-12, Vol.9 (12), p.699-712

2013

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Estrogens and Androgens in Skeletal Physiology and Pathophysiology

Physiological reviews, 2017-01, Vol.97 (1), p.135-187

2017

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Effects of sex steroids on bones and muscles: Similarities, parallels, and putative interactions in health and disease

Bone (New York, N.Y.), 2015-11, Vol.80, p.67-78

2015

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Osteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling Bone

PloS one, 2015-09, Vol.10 (9), p.e0138189-e0138189

2015

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Giant Osteoclast Formation and Long-Term Oral Bisphosphonate Therapy

The New England journal of medicine, 2009-01, Vol.360 (1), p.53-62

2009

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Steap4 Plays a Critical Role in Osteoclastogenesis in Vitro by Regulating Cellular Iron/Reactive Oxygen Species (ROS) Levels and cAMP Response Element-binding Protein (CREB) Activation

The Journal of biological chemistry, 2013-10, Vol.288 (42), p.30064-30074

2013

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FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation

Nature communications, 2014-04, Vol.5 (1), p.3773, Article 3773

2014

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The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct

Journal of bone and mineral research, 2017-03, Vol.32 (3), p.560-574

2017

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Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors

Scientific reports, 2020-07, Vol.10 (1), p.11933, Article 11933

2020

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Suppression of Autophagy in Osteocytes Mimics Skeletal Aging

The Journal of biological chemistry, 2013-06, Vol.288 (24), p.17432-17440

2013

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Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency

Bone (New York, N.Y.), 2014-09, Vol.66, p.146-154

2014

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Oxidation-specific epitopes restrain bone formation

Nature communications, 2018-06, Vol.9 (1), p.2193-11, Article 2193

2018

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Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass

PloS one, 2022-03, Vol.17 (3), p.e0265893-e0265893

2022

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Skeletal Involution by Age-associated Oxidative Stress and Its Acceleration by Loss of Sex Steroids

The Journal of biological chemistry, 2007-09, Vol.282 (37), p.27285-27297

2007

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Control of bone mass and remodeling by PTH receptor signaling in osteocytes

PloS one, 2008-08, Vol.3 (8), p.e2942-e2942

2008

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Intermittent Parathyroid Hormone Administration Counteracts the Adverse Effects of Glucocorticoids on Osteoblast and Osteocyte Viability, Bone Formation, and Strength in Mice

Endocrinology (Philadelphia), 2010-06, Vol.151 (6), p.2641-2649

2010

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Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice

Aging cell, 2010-04, Vol.9 (2), p.147-161

2010

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Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency

JCI insight, 2021-05, Vol.6 (10)

2021

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Connexin 43 Is Required for the Anti‐Apoptotic Effect of Bisphosphonates on Osteocytes and Osteoblasts In Vivo

Journal of bone and mineral research, 2008-11, Vol.23 (11), p.1712-1721

2008

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Osteocyte Apoptosis Is Induced by Weightlessness in Mice and Precedes Osteoclast Recruitment and Bone Loss

Journal of bone and mineral research, 2006-04, Vol.21 (4), p.605-615

2006

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Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency

Scientific reports, 2022-06, Vol.12 (1), p.10257-10257, Article 10257

2022

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Parathyroid Hormone Controls Receptor Activator of NF-κB Ligand Gene Expression via a Distant Transcriptional Enhancer

Molecular and Cellular Biology, 2006-09, Vol.26 (17), p.6453-6468

2006

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From Estrogen-Centric to Aging and Oxidative Stress: A Revised Perspective of the Pathogenesis of Osteoporosis

Osteocalcin promotes bone mineralization but is not a hormone

The Quest for Osteoporosis Mechanisms and Rational Therapies: How Far We've Come, How Much Further We Need to Go

The role of estrogen and androgen receptors in bone health and disease

Estrogens and Androgens in Skeletal Physiology and Pathophysiology

Effects of sex steroids on bones and muscles: Similarities, parallels, and putative interactions in health and disease

Osteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling Bone

Giant Osteoclast Formation and Long-Term Oral Bisphosphonate Therapy

Steap4 Plays a Critical Role in Osteoclastogenesis in Vitro by Regulating Cellular Iron/Reactive Oxygen Species (ROS) Levels and cAMP Response Element-binding Protein (CREB) Activation

FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation

The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct

Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors

Suppression of Autophagy in Osteocytes Mimics Skeletal Aging

Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency

Oxidation-specific epitopes restrain bone formation

Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass

Skeletal Involution by Age-associated Oxidative Stress and Its Acceleration by Loss of Sex Steroids

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Intermittent Parathyroid Hormone Administration Counteracts the Adverse Effects of Glucocorticoids on Osteoblast and Osteocyte Viability, Bone Formation, and Strength in Mice

Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice

Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency

Connexin 43 Is Required for the Anti‐Apoptotic Effect of Bisphosphonates on Osteocytes and Osteoblasts In Vivo

Osteocyte Apoptosis Is Induced by Weightlessness in Mice and Precedes Osteoclast Recruitment and Bone Loss

Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency

Parathyroid Hormone Controls Receptor Activator of NF-κB Ligand Gene Expression via a Distant Transcriptional Enhancer

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