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Osteocalcin promotes bone mineralization but is not a hormone

PLoS genetics, 2020-06, Vol.16 (6), p.e1008714-e1008714

2020

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From Estrogen-Centric to Aging and Oxidative Stress: A Revised Perspective of the Pathogenesis of Osteoporosis

Endocrine reviews, 2010-06, Vol.31 (3), p.266-300

Manolagas, Stavros C

2010

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The Quest for Osteoporosis Mechanisms and Rational Therapies: How Far We've Come, How Much Further We Need to Go

Journal of bone and mineral research, 2018-03, Vol.33 (3), p.371-385

Manolagas, Stavros C

2018

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Osteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling Bone

PloS one, 2015-09, Vol.10 (9), p.e0138189-e0138189

2015

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The role of estrogen and androgen receptors in bone health and disease

Nature reviews. Endocrinology, 2013-12, Vol.9 (12), p.699-712

2013

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Estrogens and Androgens in Skeletal Physiology and Pathophysiology

Physiological reviews, 2017-01, Vol.97 (1), p.135-187

2017

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Wnt signaling and osteoporosis

Maturitas, 2014-07, Vol.78 (3), p.233-237

Manolagas, Stavros C

2014

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Effects of sex steroids on bones and muscles: Similarities, parallels, and putative interactions in health and disease

Bone (New York, N.Y.), 2015-11, Vol.80, p.67-78

2015

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Gone with the Wnts: β-Catenin, T-Cell Factor, Forkhead Box O, and Oxidative Stress in Age-Dependent Diseases of Bone, Lipid, and Glucose Metabolism

Molecular endocrinology (Baltimore, Md.), 2007-11, Vol.21 (11), p.2605-2614

2007

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What old means to bone

Trends in endocrinology and metabolism, 2010-06, Vol.21 (6), p.369-374

2010

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Giant Osteoclast Formation and Long-Term Oral Bisphosphonate Therapy

The New England journal of medicine, 2009-01, Vol.360 (1), p.53-62

2009

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Glucocorticoids and Tumor Necrosis Factor α Increase Oxidative Stress and Suppress Wnt Protein Signaling in Osteoblasts

The Journal of biological chemistry, 2011-12, Vol.286 (52), p.44326-44335

2011

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Matrix-embedded cells control osteoclast formation

Nature medicine, 2011-10, Vol.17 (10), p.1235-1241

2011

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Steroids and osteoporosis: the quest for mechanisms

The Journal of clinical investigation, 2013-05, Vol.123 (5), p.1919-1921

Manolagas, Stavros C

2013

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Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass

PloS one, 2022-03, Vol.17 (3), p.e0265893-e0265893

2022

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DNA damage and senescence in osteoprogenitors expressing Osx1 may cause their decrease with age

Aging cell, 2017-08, Vol.16 (4), p.693-703

2017

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Steap4 Plays a Critical Role in Osteoclastogenesis in Vitro by Regulating Cellular Iron/Reactive Oxygen Species (ROS) Levels and cAMP Response Element-binding Protein (CREB) Activation

The Journal of biological chemistry, 2013-10, Vol.288 (42), p.30064-30074

2013

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Receptor Activator of Nuclear Factor κB Ligand (RANKL) Protein Expression by B Lymphocytes Contributes to Ovariectomy-induced Bone Loss

The Journal of biological chemistry, 2012-08, Vol.287 (35), p.29851-29860

2012

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FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation

Nature communications, 2014-04, Vol.5 (1), p.3773, Article 3773

2014

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Retraction: Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass

PloS one, 2023, Vol.18 (8), p.e0290458-e0290458

2023

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The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct

Journal of bone and mineral research, 2017-03, Vol.32 (3), p.560-574

2017

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Control of bone mass and remodeling by PTH receptor signaling in osteocytes

PloS one, 2008-08, Vol.3 (8), p.e2942-e2942

2008

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Increased Lipid Oxidation Causes Oxidative Stress, Increased Peroxisome Proliferator-activated Receptor-γ Expression, and Diminished Pro-osteogenic Wnt Signaling in the Skeleton

The Journal of biological chemistry, 2009-10, Vol.284 (40), p.27438-27448

2009

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Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors

Scientific reports, 2020-07, Vol.10 (1), p.11933, Article 11933

2020

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Suppression of Autophagy in Osteocytes Mimics Skeletal Aging

The Journal of biological chemistry, 2013-06, Vol.288 (24), p.17432-17440

2013

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Osteocalcin promotes bone mineralization but is not a hormone

From Estrogen-Centric to Aging and Oxidative Stress: A Revised Perspective of the Pathogenesis of Osteoporosis

The Quest for Osteoporosis Mechanisms and Rational Therapies: How Far We've Come, How Much Further We Need to Go

Osteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling Bone

The role of estrogen and androgen receptors in bone health and disease

Estrogens and Androgens in Skeletal Physiology and Pathophysiology

Wnt signaling and osteoporosis

Effects of sex steroids on bones and muscles: Similarities, parallels, and putative interactions in health and disease

Gone with the Wnts: β-Catenin, T-Cell Factor, Forkhead Box O, and Oxidative Stress in Age-Dependent Diseases of Bone, Lipid, and Glucose Metabolism

What old means to bone

Giant Osteoclast Formation and Long-Term Oral Bisphosphonate Therapy

Glucocorticoids and Tumor Necrosis Factor α Increase Oxidative Stress and Suppress Wnt Protein Signaling in Osteoblasts

Matrix-embedded cells control osteoclast formation

Steroids and osteoporosis: the quest for mechanisms

Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass

DNA damage and senescence in osteoprogenitors expressing Osx1 may cause their decrease with age

Steap4 Plays a Critical Role in Osteoclastogenesis in Vitro by Regulating Cellular Iron/Reactive Oxygen Species (ROS) Levels and cAMP Response Element-binding Protein (CREB) Activation

Receptor Activator of Nuclear Factor κB Ligand (RANKL) Protein Expression by B Lymphocytes Contributes to Ovariectomy-induced Bone Loss

FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation

Retraction: Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass

The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Increased Lipid Oxidation Causes Oxidative Stress, Increased Peroxisome Proliferator-activated Receptor-γ Expression, and Diminished Pro-osteogenic Wnt Signaling in the Skeleton

Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors

Suppression of Autophagy in Osteocytes Mimics Skeletal Aging

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