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Post mortem cerebrospinal fluid α-synuclein levels are raised in multiple system atrophy and distinguish this from the other α-synucleinopathies, Parkinson's disease and Dementia with Lewy bodies

Neurobiology of disease, 2012-01, Vol.45 (1), p.188-195

2012

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Huntington's Disease Intranuclear Inclusions Contain Truncated, Ubiquitinated Huntingtin Protein

Experimental neurology, 1999-03, Vol.156 (1), p.92-99

1999

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A SNP in the ACT gene associated with astrocytosis and rapid cognitive decline in AD

Neurobiology of aging, 2008-08, Vol.29 (8), p.1167-1176

2008

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Association between interleukin-1A polymorphism and cerebral amyloid angiopathy-related hemorrhage

Stroke (1970), 2003-10, Vol.34 (10), p.e193-e195

2003

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Cognitive change in motor neurone disease/amyotrophic lateral sclerosis (MND/ALS)

Journal of the Neurological Sciences, 2000-11, Vol.180 (1), p.15-20

2000

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Phosphorylation of Tau proteins: a major event during the process of neurofibrillary degeneration. A comparative study between Alzheimer's disease and Down's syndrome

Brain research, 1990-05, Vol.516 (1), p.15-19

1990

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An integrative hypothesis concerning the pathogenesis and progression of Alzheimer's disease

Neurobiology of Aging, 1986-11, Vol.7 (6), p.489-502

1986

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Negative association between amyloid plaques and cerebral amyloid angiopathy in Alzheimer's disease

Neuroscience letters, 2003-12, Vol.352 (2), p.137-140

2003

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Loss of nerve cells from locus coeruleus in Alzheimer's disease is topographically arranged

Neuroscience letters, 1986-03, Vol.64 (3), p.247-252

1986

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The topography of nerve cell loss from the locus caeruleus in elderly persons

Neurobiology of aging, 1989, Vol.10 (1), p.5-9

1989

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A polymorphism in the angiotensin 1-converting enzyme gene is associated with damage to cerebral cortical white matter in Alzheimer's disease

Neuroscience letters, 2004-01, Vol.354 (2), p.103-106

2004

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The angiotensin 1-converting enzyme insertion (I)/deletion (D) polymorphism does not influence the extent of amyloid or tau pathology in patients with sporadic Alzheimer's disease

Neuroscience letters, 2002-08, Vol.328 (3), p.314-318

2002

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Amyloid β protein deposition in patients with frontotemporal lobar degeneration: relationship to age and apolipoprotein E genotype

Neuroscience letters, 2001-05, Vol.304 (3), p.161-164

2001

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Preferential deposition of amyloid β protein (A β) in the form A β40 in Alzheimer's disease is associated with a gene dosage effect of the apolipoprotein E E4 allele

Neuroscience letters, 1997, Vol.221 (2), p.81-84

1997

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Activated Astrocytes Display Increased 5-HT2a Receptor Expression in Pathological States

Experimental neurology, 1999-08, Vol.158 (2), p.529-533

1999

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Loss of neurones from cortical and subcortical areas in Down's syndrome patients at middle age

Journal of the neurological sciences, 1987-08, Vol.80 (1), p.79-89

1987

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A quantitative morphometric analysis of the neuronal and synaptic content of the frontal and temporal cortex in patients with Alzheimer's disease

Journal of the neurological sciences, 1987-04, Vol.78 (2), p.151-164

1987

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The clinical pathological correlates of lobar atrophy

Dementia (Basel, Switzerland), 1993, Vol.4 (3-4), p.154

1993

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A polymorphism within intron 11 of the tau gene is not increased in frequency in patients with sporadic Alzheimer's disease, nor does it influence the extent of tau pathology in the brain

Neuroscience letters, 2002-05, Vol.324 (2), p.113-116

2002

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The topographic distribution of brain atrophy in frontal lobe dementia

Acta neuropathologica, 1993-02, Vol.85 (3), p.334-340

1993

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A Commentary on the Diagnostic Criteria for the Neuropathological Assessment of Alzheimer’s Disease

Neurobiology of aging, 1997-07, Vol.18 (4), p.S51-S52

Mann, D.M.A

1997

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Observations on the brains of demented old people. B.E. Tomlinson, G. Blessed and M. Roth, Journal of the Neurological Sciences (1970) 11, 205-242; (1968) 7, 331-356

International journal of geriatric psychiatry, 1997-08, Vol.12 (8), p.785-790

1997

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Deposition of amyloid (A4) protein within the brains of persons with dementing disorders other than Alzheimer's disease and Down's syndrome

Neuroscience letters, 1990-02, Vol.109 (1), p.68-75

1990

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Some morphometric observations on the cerebral cortex and hippocampus in presenile Alzheimer's disease, senile dementia of Alzheimer type and Down's syndrome in middle age

Journal of the neurological sciences, 1985-01, Vol.69 (3), p.139-159

1985

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Apolipoprotein E allelic frequencies in patients with lobar atrophy

Neuroscience letters, 1995-03, Vol.188 (3), p.205-207

1995

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Post mortem cerebrospinal fluid α-synuclein levels are raised in multiple system atrophy and distinguish this from the other α-synucleinopathies, Parkinson's disease and Dementia with Lewy bodies

Huntington's Disease Intranuclear Inclusions Contain Truncated, Ubiquitinated Huntingtin Protein

A SNP in the ACT gene associated with astrocytosis and rapid cognitive decline in AD

Association between interleukin-1A polymorphism and cerebral amyloid angiopathy-related hemorrhage

Cognitive change in motor neurone disease/amyotrophic lateral sclerosis (MND/ALS)

Phosphorylation of Tau proteins: a major event during the process of neurofibrillary degeneration. A comparative study between Alzheimer's disease and Down's syndrome

An integrative hypothesis concerning the pathogenesis and progression of Alzheimer's disease

Negative association between amyloid plaques and cerebral amyloid angiopathy in Alzheimer's disease

Loss of nerve cells from locus coeruleus in Alzheimer's disease is topographically arranged

The topography of nerve cell loss from the locus caeruleus in elderly persons

A polymorphism in the angiotensin 1-converting enzyme gene is associated with damage to cerebral cortical white matter in Alzheimer's disease

The angiotensin 1-converting enzyme insertion (I)/deletion (D) polymorphism does not influence the extent of amyloid or tau pathology in patients with sporadic Alzheimer's disease

Amyloid β protein deposition in patients with frontotemporal lobar degeneration: relationship to age and apolipoprotein E genotype

Preferential deposition of amyloid β protein (A β) in the form A β40 in Alzheimer's disease is associated with a gene dosage effect of the apolipoprotein E E4 allele

Activated Astrocytes Display Increased 5-HT2a Receptor Expression in Pathological States

Loss of neurones from cortical and subcortical areas in Down's syndrome patients at middle age

A quantitative morphometric analysis of the neuronal and synaptic content of the frontal and temporal cortex in patients with Alzheimer's disease

The clinical pathological correlates of lobar atrophy

A polymorphism within intron 11 of the tau gene is not increased in frequency in patients with sporadic Alzheimer's disease, nor does it influence the extent of tau pathology in the brain

The topographic distribution of brain atrophy in frontal lobe dementia

A Commentary on the Diagnostic Criteria for the Neuropathological Assessment of Alzheimer’s Disease

Observations on the brains of demented old people. B.E. Tomlinson, G. Blessed and M. Roth, Journal of the Neurological Sciences (1970) 11, 205-242; (1968) 7, 331-356

Deposition of amyloid (A4) protein within the brains of persons with dementing disorders other than Alzheimer's disease and Down's syndrome

Some morphometric observations on the cerebral cortex and hippocampus in presenile Alzheimer's disease, senile dementia of Alzheimer type and Down's syndrome in middle age

Apolipoprotein E allelic frequencies in patients with lobar atrophy

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