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Understanding the broad-spectrum neuroprotective action profile of green tea polyphenols in aging and neurodegenerative diseases

Journal of Alzheimer's disease, 2011-01, Vol.25 (2), p.187

2011

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Cell signaling pathways and iron chelation in the neurorestorative activity of green tea polyphenols: special reference to epigallocatechin gallate (EGCG)

Journal of Alzheimer's disease, 2008-10, Vol.15 (2), p.211

2008

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A molecular signature in blood identifies early Parkinson's disease

Molecular neurodegeneration, 2012-05, Vol.7 (1), p.26-26, Article 26

2012

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Simultaneous Manipulation of Multiple Brain Targets by Green Tea Catechins: A Potential Neuroprotective Strategy for Alzheimer and Parkinson Diseases

CNS drug reviews, 2008, Vol.14 (4), p.352-365

2008

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Green tea polyphenol (-)-epigallocatechin-3-gallate promotes the rapid protein kinase C- and proteasome-mediated degradation of Bad: implications for neuroprotection

Journal of neurochemistry, 2007-02, Vol.100 (4), p.992-1002

2007

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Rasagiline promotes regeneration of substantia nigra dopaminergic neurons in post-MPTP-induced Parkinsonism via activation of tyrosine kinase receptor signaling pathway

Neurochemical research, 2007-10, Vol.32 (10), p.1694-1699

2007

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A Sporadic Parkinson Disease Model via Silencing of the Ubiquitin-Proteasome/E3 Ligase Component SKP1A

The Journal of biological chemistry, 2009-11, Vol.284 (47), p.32835-32845

2009

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DJ‐1 deficiency triggers microglia sensitivity to dopamine toward a pro‐inflammatory phenotype that is attenuated by rasagiline

Journal of neurochemistry, 2014-05, Vol.129 (3), p.434-447

2014

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Low Dosage of Rasagiline and Epigallocatechin Gallate Synergistically Restored the Nigrostriatal Axis in MPTP-Induced Parkinsonism

Neuro-degenerative diseases, 2010-01, Vol.7 (4), p.219-231

2010

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PGC-1α, a potential therapeutic target for early intervention in Parkinson's disease

Science translational medicine, 2010-10, Vol.2 (52), p.52ra73

2010

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Targeting Skp1, an Ubiquitin E3 Ligase Component Found Decreased in Sporadic Parkinson’s Disease

Neuro-degenerative diseases, 2012-01, Vol.10 (1-4), p.220-223

2012

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Differential Alterations in Metabolism and Proteolysis-Related Proteins in Human Parkinson’s Disease Substantia Nigra

Neurotoxicity research, 2018-04, Vol.33 (3), p.560-568

2018

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Targeting Multiple Neurodegenerative Diseases Etiologies with Multimodal-Acting Green Tea Catechins

The Journal of nutrition, 2008-08, Vol.138 (8), p.1578S-1583S

2008

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Biomarkers for Evaluation of Clinical Efficacy of Multipotential Neuroprotective Drugs for Alzheimer's and Parkinson's Diseases

Neurotherapeutics, 2009, Vol.6 (1), p.128-140

2009

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Neuroprotective profile of the multitarget drug rasagiline in Parkinson's disease

International Review of Neurobiology, 2011, Vol.100, p.127-149

2011

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Multifunctional activities of green tea catechins in neuroprotection. Modulation of cell survival genes, iron-dependent oxidative stress and PKC signaling pathway

Neuro-Signals, 2005, Vol.14 (1-2), p.46

2005

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From anti-Parkinson's drug rasagiline to novel multitarget iron chelators with acetylcholinesterase and monoamine oxidase inhibitory and neuroprotective properties for Alzheimer's disease

Journal of Alzheimer's disease, 2012-01, Vol.30 (1), p.1

2012

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The novel multifunctional, iron-chelating drugs M30 and HLA20 protect pancreatic beta-cell lines from oxidative stress damage

The Journal of pharmacology and experimental therapeutics, 2010-06, Vol.333 (3), p.874

2010

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Molecular mechanisms of the neuroprotective/neurorescue action of multi-target green tea polyphenols

Frontiers in bioscience (Scholar edition), 2012-01, Vol.4 (2), p.581

2012

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Effects of novel neuroprotective and neurorestorative multifunctional drugs on iron chelation and glucose metabolism

Journal of Neural Transmission, 2013, Vol.120 (1), p.37-48

2013

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Biomarkers for prediction and targeted prevention of Alzheimer’s and Parkinson’s diseases: evaluation of drug clinical efficacy

The EPMA journal, 2010-06, Vol.1 (2), p.273-292

2010

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Genetic reduction of the E3 ubiquitin ligase element, SKP1A and environmental manipulation to emulate cardinal features of Parkinson's disease

Parkinsonism & related disorders, 2012, Vol.18, p.S177-S179

2012

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Modeling sporadic Parkinson's disease by silencing the ubiquitin E3 ligase component, SKP1A

Parkinsonism & related disorders, 2009-12, Vol.15, p.S148-S151

2009

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Gene and protein signatures in sporadic Parkinson's disease and a novel genetic model of PD

Parkinsonism & related disorders, 2007, Vol.13, p.S242-S247

2007

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Silencing/Overexpressing Selected Genes as a Model of Sporadic Parkinson’s Disease

Neuro-degenerative diseases, 2010-01, Vol.7 (1-3), p.108-111

2010

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Understanding the broad-spectrum neuroprotective action profile of green tea polyphenols in aging and neurodegenerative diseases

Cell signaling pathways and iron chelation in the neurorestorative activity of green tea polyphenols: special reference to epigallocatechin gallate (EGCG)

A molecular signature in blood identifies early Parkinson's disease

Simultaneous Manipulation of Multiple Brain Targets by Green Tea Catechins: A Potential Neuroprotective Strategy for Alzheimer and Parkinson Diseases

Green tea polyphenol (-)-epigallocatechin-3-gallate promotes the rapid protein kinase C- and proteasome-mediated degradation of Bad: implications for neuroprotection

Rasagiline promotes regeneration of substantia nigra dopaminergic neurons in post-MPTP-induced Parkinsonism via activation of tyrosine kinase receptor signaling pathway

A Sporadic Parkinson Disease Model via Silencing of the Ubiquitin-Proteasome/E3 Ligase Component SKP1A

DJ‐1 deficiency triggers microglia sensitivity to dopamine toward a pro‐inflammatory phenotype that is attenuated by rasagiline

Low Dosage of Rasagiline and Epigallocatechin Gallate Synergistically Restored the Nigrostriatal Axis in MPTP-Induced Parkinsonism

PGC-1α, a potential therapeutic target for early intervention in Parkinson's disease

Targeting Skp1, an Ubiquitin E3 Ligase Component Found Decreased in Sporadic Parkinson’s Disease

Differential Alterations in Metabolism and Proteolysis-Related Proteins in Human Parkinson’s Disease Substantia Nigra

Targeting Multiple Neurodegenerative Diseases Etiologies with Multimodal-Acting Green Tea Catechins

Biomarkers for Evaluation of Clinical Efficacy of Multipotential Neuroprotective Drugs for Alzheimer's and Parkinson's Diseases

Neuroprotective profile of the multitarget drug rasagiline in Parkinson's disease

Multifunctional activities of green tea catechins in neuroprotection. Modulation of cell survival genes, iron-dependent oxidative stress and PKC signaling pathway

From anti-Parkinson's drug rasagiline to novel multitarget iron chelators with acetylcholinesterase and monoamine oxidase inhibitory and neuroprotective properties for Alzheimer's disease

The novel multifunctional, iron-chelating drugs M30 and HLA20 protect pancreatic beta-cell lines from oxidative stress damage

Molecular mechanisms of the neuroprotective/neurorescue action of multi-target green tea polyphenols

Effects of novel neuroprotective and neurorestorative multifunctional drugs on iron chelation and glucose metabolism

Biomarkers for prediction and targeted prevention of Alzheimer’s and Parkinson’s diseases: evaluation of drug clinical efficacy

Genetic reduction of the E3 ubiquitin ligase element, SKP1A and environmental manipulation to emulate cardinal features of Parkinson's disease

Modeling sporadic Parkinson's disease by silencing the ubiquitin E3 ligase component, SKP1A

Gene and protein signatures in sporadic Parkinson's disease and a novel genetic model of PD

Silencing/Overexpressing Selected Genes as a Model of Sporadic Parkinson’s Disease

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