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Impact of the Myosin Modulator Mavacamten on Force Generation and Cross-Bridge Behavior in a Murine Model of Hypercontractility

Journal of the American Heart Association, 2018-09, Vol.7 (17), p.e009627-e009627

2018

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Molecular effects of the myosin activator omecamtiv mecarbil on contractile properties of skinned myocardium lacking cardiac myosin binding protein-C

Journal of molecular and cellular cardiology, 2015-08, Vol.85, p.262-272

2015

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Dose-Dependent Effects of the Myosin Activator Omecamtiv Mecarbil on Cross-Bridge Behavior and Force Generation in Failing Human Myocardium

Circulation. Heart failure, 2017-10, Vol.10 (10)

2017

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AAV9 gene transfer of cMyBPC N-terminal domains ameliorates cardiomyopathy in cMyBPC-deficient mice

JCI insight, 2020-09, Vol.5 (17)

2020

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Divergent effects of α- and β-myosin heavy chain isoforms on the N terminus of rat cardiac troponin T

The Journal of general physiology, 2013-10, Vol.142 (4), p.413-423

2013

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The Hcm-Causing Y235S Cmybpc Mutation Accelerates Contractile Function by Altering C1 Domain Structure

Biophysical journal, 2019-02, Vol.116 (3), p.266a-266a

2019

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Effects of the N-Terminus of Cardiac Troponin T on Contractile Activation Depend on the Type of Myosin Heavy Chain Isoform

Biophysical journal, 2013-01, Vol.104 (2), p.482a-483a

2013

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Divergent effects of [Alpha]- and [Beta]-myosin heavy chain isoforms on the N terminus of rat cardiac troponin T

The Journal of general physiology, 2013-10, Vol.142 (4), p.413

2013

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Nebulin Alters Cross-bridge Cycling Kinetics and Increases Thin Filament Activation

The Journal of biological chemistry, 2009-11, Vol.284 (45), p.30889-30896

2009

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The HCM-causing Y235S cMyBPC mutation accelerates contractile function by altering C1 domain structure

Biochimica et biophysica acta. Molecular basis of disease, 2019-03, Vol.1865 (3), p.661-677

2019

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cMyBPC phosphorylation modulates the effect of omecamtiv mecarbil on myocardial force generation

The Journal of general physiology, 2021-07, Vol.153 (7), p.1

2021

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Lost in translation: Interpreting cardiac muscle mechanics data in clinical practice

Archives of biochemistry and biophysics, 2019-02, Vol.662, p.213-218

2019

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Myosin Activator Omecamtiv Mecarbil Differentially Impacts the Contractile Properties of Skinned Myocardium from Failing and Donor Human Hearts

Biophysical journal, 2017-02, Vol.112 (3), p.556a-556a

2017

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Instability in the Central Region of Tropomyosin Modulates the Function of Its Overlapping Ends

Biophysical journal, 2013-11, Vol.105 (9), p.2104-2113

2013

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The contribution of cardiac myosin binding protein‐c Ser282 phosphorylation to the rate of force generation and in vivo cardiac contractility

The Journal of physiology, 2014-09, Vol.592 (17), p.3747-3765

2014

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Nebulin Alters Cross-bridge Cycling Kinetics and Increases Thin Filament Activation: A NOVEL MECHANISM FOR INCREASING TENSION AND REDUCING TENSION COST

The Journal of biological chemistry, 2009-11, Vol.284 (45), p.30889-30896

2009

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Molecular characterization of linker and loop-mediated structural modulation and hinge motion in the C4-C5 domains of cMyBPC

Journal of structural biology, 2022-06, Vol.214 (2), p.107856-107856, Article 107856

2022

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Interplay between the overlapping ends of tropomyosin and the N terminus of cardiac troponin T affects tropomyosin states on actin

The FASEB journal, 2013-09, Vol.27 (9), p.3848-3859

2013

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cMyBP-C Phosphorylation Modulates Sarcomere Length-Dependent Changes in Cardiac Muscle Contractile Function

Biophysical journal, 2016-02, Vol.110 (3), p.525a-526a

2016

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Sarcomeric protein modification during adrenergic stress enhances cross-bridge kinetics and cardiac output

Journal of applied physiology (1985), 2017-03, Vol.122 (3), p.520-530

2017

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Cardiac Myosin Binding Protein-C Phosphorylation Modulates Myofilament Length-Dependent Activation

Frontiers in physiology, 2016-02, Vol.7, p.38-38

2016

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Length-Dependent Contractile Dynamics are Blunted Upon Ablation of Cardiac Myosin Binding Protein-C

Biophysical journal, 2015-01, Vol.108 (2), p.597a-597a

2015

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Sarcomere-based genetic enhancement of systolic cardiac function in a murine model of dilated cardiomyopathy

International journal of cardiology, 2018-12, Vol.273, p.168-176

2018

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Identification of two new regions in the N‐terminus of cardiac troponin T that have divergent effects on cardiac contractile function

The Journal of physiology, 2013-03, Vol.591 (5), p.1217-1234

2013

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Strategies for targeting the cardiac sarcomere: avenues for novel drug discovery

Expert opinion on drug discovery, 2020-04, Vol.15 (4), p.457-469

2020

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Impact of the Myosin Modulator Mavacamten on Force Generation and Cross-Bridge Behavior in a Murine Model of Hypercontractility

Molecular effects of the myosin activator omecamtiv mecarbil on contractile properties of skinned myocardium lacking cardiac myosin binding protein-C

Dose-Dependent Effects of the Myosin Activator Omecamtiv Mecarbil on Cross-Bridge Behavior and Force Generation in Failing Human Myocardium

AAV9 gene transfer of cMyBPC N-terminal domains ameliorates cardiomyopathy in cMyBPC-deficient mice

Divergent effects of α- and β-myosin heavy chain isoforms on the N terminus of rat cardiac troponin T

The Hcm-Causing Y235S Cmybpc Mutation Accelerates Contractile Function by Altering C1 Domain Structure

Effects of the N-Terminus of Cardiac Troponin T on Contractile Activation Depend on the Type of Myosin Heavy Chain Isoform

Divergent effects of [Alpha]- and [Beta]-myosin heavy chain isoforms on the N terminus of rat cardiac troponin T

Nebulin Alters Cross-bridge Cycling Kinetics and Increases Thin Filament Activation

The HCM-causing Y235S cMyBPC mutation accelerates contractile function by altering C1 domain structure

cMyBPC phosphorylation modulates the effect of omecamtiv mecarbil on myocardial force generation

Lost in translation: Interpreting cardiac muscle mechanics data in clinical practice

Myosin Activator Omecamtiv Mecarbil Differentially Impacts the Contractile Properties of Skinned Myocardium from Failing and Donor Human Hearts

Instability in the Central Region of Tropomyosin Modulates the Function of Its Overlapping Ends

The contribution of cardiac myosin binding protein‐c Ser282 phosphorylation to the rate of force generation and in vivo cardiac contractility

Nebulin Alters Cross-bridge Cycling Kinetics and Increases Thin Filament Activation: A NOVEL MECHANISM FOR INCREASING TENSION AND REDUCING TENSION COST

Molecular characterization of linker and loop-mediated structural modulation and hinge motion in the C4-C5 domains of cMyBPC

Interplay between the overlapping ends of tropomyosin and the N terminus of cardiac troponin T affects tropomyosin states on actin

cMyBP-C Phosphorylation Modulates Sarcomere Length-Dependent Changes in Cardiac Muscle Contractile Function

Sarcomeric protein modification during adrenergic stress enhances cross-bridge kinetics and cardiac output

Cardiac Myosin Binding Protein-C Phosphorylation Modulates Myofilament Length-Dependent Activation

Length-Dependent Contractile Dynamics are Blunted Upon Ablation of Cardiac Myosin Binding Protein-C

Sarcomere-based genetic enhancement of systolic cardiac function in a murine model of dilated cardiomyopathy

Identification of two new regions in the N‐terminus of cardiac troponin T that have divergent effects on cardiac contractile function

Strategies for targeting the cardiac sarcomere: avenues for novel drug discovery

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