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Modifying macrophages at the periphery has the capacity to change microglial reactivity and to extend ALS survival

Nature neuroscience, 2020-11, Vol.23 (11), p.1339

2020

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System xC- is a mediator of microglial function and its deletion slows symptoms in amyotrophic lateral sclerosis mice

Brain (London, England : 1878), 2015-01, Vol.138 (Pt 1), p.53-68

2015

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Microglia Promote the Death of Developing Purkinje Cells

Neuron (Cambridge, Mass.), 2004-02, Vol.41 (4), p.535-547

2004

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Neurotoxic Activation of Microglia Is Promoted by a Nox1-Dependent NADPH Oxidase

The Journal of neuroscience, 2008-11, Vol.28 (46), p.12039-12051

2008

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Exposure to 1800 MHz LTE electromagnetic fields under proinflammatory conditions decreases the response strength and increases the acoustic threshold of auditory cortical neurons

Scientific reports, 2022-03, Vol.12 (1), p.4063-4063, Article 4063

2022

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The NADPH oxidase Nox2 regulates VEGFR1/CSF-1R-mediated microglial chemotaxis and promotes early postnatal infiltration of phagocytes in the subventricular zone of the mouse cerebral cortex

Glia, 2013-09, Vol.61 (9), p.1542-1555

2013

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Phagocytosis in the developing CNS: more than clearing the corpses

Current opinion in neurobiology, 2005-02, Vol.15 (1), p.101-107

2005

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Effect of Exposure to 1,800 MHz Electromagnetic Fields on Heat Shock Proteins and Glial Cells in the Brain of Developing Rats

Neurotoxicity research, 2011-08, Vol.20 (2), p.109-119

2011

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Regulation of Microglial Development: A Novel Role for Thyroid Hormone

The Journal of neuroscience, 2001-03, Vol.21 (6), p.2028-2038

2001

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New insights into the Immune TME of adult-type diffuse gliomas

Current opinion in neurology, 2022-12, Vol.35 (6), p.794

2022

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Cytotoxic Effect of Brain Macrophages on Developing Neurons

The European journal of neuroscience, 1991-11, Vol.3 (11), p.1155-1164

1991

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System Xc- and Apolipoprotein E Expressed by Microglia Have Opposite Effects on the Neurotoxicity of Amyloid-beta Peptide 1-40

The Journal of neuroscience, 2006-03, Vol.26 (12), p.3345-3356

2006

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A Single Exposure to GSM-1800 MHz Signals in the Course of an Acute Neuroinflammatory Reaction can Alter Neuronal Responses and Microglial Morphology in the Rat Primary Auditory Cortex

Neuroscience, 2018-08, Vol.385, p.11-24

2018

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Effects of a Single Head Exposure to GSM-1800 MHz Signals on the Transcriptome Profile in the Rat Cerebral Cortex: Enhanced Gene Responses Under Proinflammatory Conditions

Neurotoxicity research, 2020-06, Vol.38 (1), p.105-123

2020

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LPS-Induced Inflammation Abolishes the Effect of DYRK1A on IkB Stability in the Brain of Mice

Molecular neurobiology, 2019-02, Vol.56 (2), p.963-975

2019

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Acute Neuroinflammation Promotes Cell Responses to 1800 MHz GSM Electromagnetic Fields in the Rat Cerebral Cortex

Neurotoxicity research, 2017-10, Vol.32 (3), p.444-459

2017

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Erratum to: Acute Neuroinflammation Promotes Cell Responses to 1800 MHz GSM Electromagnetic Fields in the rat Cerebral Cortex

Neurotoxicity research, 2017-10, Vol.32 (3), p.530-531

2017

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Remyelination by Resident Oligodendrocyte Precursor Cells in a Xenopus laevis Inducible Model of Demyelination

Developmental neuroscience, 2015-01, Vol.37 (3), p.232-242

2015

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Thyroid hormone actions on the microglial development

Anais da Academia Brasileira de Ciências, 2000-09, Vol.72 (3), p.442-443

2000

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System X c − and Apolipoprotein E Expressed by Microglia Have Opposite Effects on the Neurotoxicity of Amyloid-β Peptide 1–40

The Journal of neuroscience, 2006-03, Vol.26 (12), p.3345-3356

2006

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System X sub(c) super(-) and Apolipoprotein E Expressed by Microglia Have Opposite Effects on the Neurotoxicity of Amyloid- beta Peptide 1-40

The Journal of neuroscience, 2006-03, Vol.26 (12), p.3345-3356

2006

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Production of Monocyte Chemotactic Protein-1 By Rat Brain Macrophages

The European journal of neuroscience, 1996-08, Vol.8 (8), p.1725-1734

1996

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Neurons Promote Macrophage Proliferation by Producing Transforming Growth Factor-β2

The Journal of neuroscience, 1997-07, Vol.17 (14), p.5305-5315

1997

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Downregulation of in vitro neurotoxicity of brain macrophages by prostaglandin E2 and a β-adrenergic agonist

Glia, 1994-08, Vol.11 (4), p.383-386

1994

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Neurons Promote Macrophage Proliferation by Producing Transforming Growth Factor-beta 2

The Journal of neuroscience, 1997-07, Vol.17 (14), p.5305-5315

1997

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Modifying macrophages at the periphery has the capacity to change microglial reactivity and to extend ALS survival

System xC- is a mediator of microglial function and its deletion slows symptoms in amyotrophic lateral sclerosis mice

Microglia Promote the Death of Developing Purkinje Cells

Neurotoxic Activation of Microglia Is Promoted by a Nox1-Dependent NADPH Oxidase

Exposure to 1800 MHz LTE electromagnetic fields under proinflammatory conditions decreases the response strength and increases the acoustic threshold of auditory cortical neurons

The NADPH oxidase Nox2 regulates VEGFR1/CSF-1R-mediated microglial chemotaxis and promotes early postnatal infiltration of phagocytes in the subventricular zone of the mouse cerebral cortex

Phagocytosis in the developing CNS: more than clearing the corpses

Effect of Exposure to 1,800 MHz Electromagnetic Fields on Heat Shock Proteins and Glial Cells in the Brain of Developing Rats

Regulation of Microglial Development: A Novel Role for Thyroid Hormone

New insights into the Immune TME of adult-type diffuse gliomas

Cytotoxic Effect of Brain Macrophages on Developing Neurons

System Xc- and Apolipoprotein E Expressed by Microglia Have Opposite Effects on the Neurotoxicity of Amyloid-beta Peptide 1-40

A Single Exposure to GSM-1800 MHz Signals in the Course of an Acute Neuroinflammatory Reaction can Alter Neuronal Responses and Microglial Morphology in the Rat Primary Auditory Cortex

Effects of a Single Head Exposure to GSM-1800 MHz Signals on the Transcriptome Profile in the Rat Cerebral Cortex: Enhanced Gene Responses Under Proinflammatory Conditions

LPS-Induced Inflammation Abolishes the Effect of DYRK1A on IkB Stability in the Brain of Mice

Acute Neuroinflammation Promotes Cell Responses to 1800 MHz GSM Electromagnetic Fields in the Rat Cerebral Cortex

Erratum to: Acute Neuroinflammation Promotes Cell Responses to 1800 MHz GSM Electromagnetic Fields in the rat Cerebral Cortex

Remyelination by Resident Oligodendrocyte Precursor Cells in a Xenopus laevis Inducible Model of Demyelination

Thyroid hormone actions on the microglial development

System X c − and Apolipoprotein E Expressed by Microglia Have Opposite Effects on the Neurotoxicity of Amyloid-β Peptide 1–40

System X sub(c) super(-) and Apolipoprotein E Expressed by Microglia Have Opposite Effects on the Neurotoxicity of Amyloid- beta Peptide 1-40

Production of Monocyte Chemotactic Protein-1 By Rat Brain Macrophages

Neurons Promote Macrophage Proliferation by Producing Transforming Growth Factor-β2

Downregulation of in vitro neurotoxicity of brain macrophages by prostaglandin E2 and a β-adrenergic agonist

Neurons Promote Macrophage Proliferation by Producing Transforming Growth Factor-beta 2

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