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Cytoplasmic and nuclear polyglutamine aggregates in SCA6 Purkinje cells

Neurology, 2001-06, Vol.56 (12), p.1753

2001

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Brain hydrogen sulfide is severely decreased in Alzheimer's disease

Biochemical and biophysical research communications, 2002-05, Vol.293 (5), p.1485-1488

2002

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Corticobasal degeneration: etiopathological significance of the cytoskeletal alterations

Acta neuropathologica, 1994-06, Vol.87 (6), p.545-553

1994

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Hereditary spastic paraplegia with frontal lobe dysfunction: a clinicopathologic study

Neurology, 2004-12, Vol.63 (11), p.2149

2004

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A patient with dementia with Lewy bodies and codon 232 mutation of PRNP

Neurology, 2002-11, Vol.59 (10), p.1619

2002

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Sporadic Creutzfeldt-Jakob disease with MM1-type prion protein and plaques

Neurology, 2003-02, Vol.60 (3), p.514

2003

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Prefrontal Abnormality of Schizophrenia Revealed by DNA Microarray: Impact on Glial and Neurotrophic Gene Expression

Annals of the New York Academy of Sciences, 2004-10, Vol.1025 (1), p.84-91

2004

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White matter hyperintensities on MRI in a patient with corticobasal degeneration

Acta neurologica Scandinavica, 1999-03, Vol.99 (3), p.199-201

1999

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Wobble modification deficiency in mutant tRNAs in patients with mitochondrial diseases

FEBS letters, 2005-05, Vol.579 (13), p.2948-2952

2005

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Selective reduction of a PDZ protein, SAP‐97, in the prefrontal cortex of patients with chronic schizophrenia

Journal of neurochemistry, 2002-11, Vol.83 (4), p.797-806

2002

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Development and characterization of a TAPIR-like mouse monoclonal antibody to amyloid-beta

Journal of Alzheimer's disease, 2008-06, Vol.14 (2), p.161

2008

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Aprataxin, the causative protein for EAOH is a nuclear protein with a potential role as a DNA repair protein

Annals of neurology, 2004-02, Vol.55 (2), p.241-249

2004

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Glypican‐1 as an Aβ binding HSPG in the human brain: Its localization in DIG domains and possible roles in the pathogenesis of Alzheimer's disease

The FASEB journal, 2004-06, Vol.18 (9), p.1013-1015

2004

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Increased Production of β-Amyloid and Vulnerability to Endoplasmic Reticulum Stress by an Aberrant Spliced Form of Presenilin 2

The Journal of biological chemistry, 2001-01, Vol.276 (3), p.2108-2114

2001

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Systemic mastocytosis and recurrent anaphylactic shock

The Lancet (British edition), 2002-06, Vol.359 (9323), p.2084-2084

2002

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Ataxin-3 Is Translocated into the Nucleus for the Formation of Intranuclear Inclusions in Normal and Machado–Joseph Disease Brains

Experimental neurology, 2000-10, Vol.165 (2), p.248-256

2000

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A decrease in interleukin-1 receptor antagonist expression in the prefrontal cortex of schizophrenic patients

Neuroscience research, 2003-07, Vol.46 (3), p.299-307

2003

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A quantitative study on the expression of synapsin II and N-ethylmaleimide-sensitive fusion protein in schizophrenic patients

Neuroscience letters, 2001-06, Vol.305 (3), p.185-188

2001

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Intracellular A beta 42 activates p53 promoter: a pathway to neurodegeneration in Alzheimer's disease

The FASEB journal, 2005-02, Vol.19 (2), p.255-257

2005

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Intracellular Ab42 actiavtes p53 promoter: a pathway to neurodegeneration in Alzheimer's disease

The FASEB journal, 2005, Vol.19, p.255-257

2005

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Distinct Pathological Features of the Gallyas- and Tau-positive Glia in the Parkinsonism-Dementia Complex and Amyotrophic Lateral Sclerosis of Guam

Journal of neuropathology and experimental neurology, 1997-03, Vol.56 (3), p.308-316

1997

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Both N-terminal and C-terminal fragments of Presenilin 1 colocalize with neurofibrillary tangles in neurons and dystrophic neurites of senile plaques in Alzheimer's disease

Journal of neuroscience research, 1998-07, Vol.53 (1), p.99-106

1998

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Progressive supranuclear palsy on Guam

Acta neuropathologica, 2001-11, Vol.102 (5), p.510-514

2001

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Fractal analysis of senile plaque observed in various animal species

Neuroscience letters, 2001-01, Vol.297 (3), p.195-198

2001

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A TOPOGRAPHIC AND QUANTITATIVE STUDY OF NEURONS IN HUMAN SPINAL GRAY MATTER, WITH SPECIAL REFERENCE TO THEIR CHANGES IN AMYOTROPHIC LATERAL SCLEROSIS

Biomedical Research, 1983/04/01, Vol.4(2), pp.211-224

1983

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Cytoplasmic and nuclear polyglutamine aggregates in SCA6 Purkinje cells

Brain hydrogen sulfide is severely decreased in Alzheimer's disease

Corticobasal degeneration: etiopathological significance of the cytoskeletal alterations

Hereditary spastic paraplegia with frontal lobe dysfunction: a clinicopathologic study

A patient with dementia with Lewy bodies and codon 232 mutation of PRNP

Sporadic Creutzfeldt-Jakob disease with MM1-type prion protein and plaques

Prefrontal Abnormality of Schizophrenia Revealed by DNA Microarray: Impact on Glial and Neurotrophic Gene Expression

White matter hyperintensities on MRI in a patient with corticobasal degeneration

Wobble modification deficiency in mutant tRNAs in patients with mitochondrial diseases

Selective reduction of a PDZ protein, SAP‐97, in the prefrontal cortex of patients with chronic schizophrenia

Development and characterization of a TAPIR-like mouse monoclonal antibody to amyloid-beta

Aprataxin, the causative protein for EAOH is a nuclear protein with a potential role as a DNA repair protein

Glypican‐1 as an Aβ binding HSPG in the human brain: Its localization in DIG domains and possible roles in the pathogenesis of Alzheimer's disease

Increased Production of β-Amyloid and Vulnerability to Endoplasmic Reticulum Stress by an Aberrant Spliced Form of Presenilin 2

Systemic mastocytosis and recurrent anaphylactic shock

Ataxin-3 Is Translocated into the Nucleus for the Formation of Intranuclear Inclusions in Normal and Machado–Joseph Disease Brains

A decrease in interleukin-1 receptor antagonist expression in the prefrontal cortex of schizophrenic patients

A quantitative study on the expression of synapsin II and N-ethylmaleimide-sensitive fusion protein in schizophrenic patients

Intracellular A beta 42 activates p53 promoter: a pathway to neurodegeneration in Alzheimer's disease

Intracellular Ab42 actiavtes p53 promoter: a pathway to neurodegeneration in Alzheimer's disease

Distinct Pathological Features of the Gallyas- and Tau-positive Glia in the Parkinsonism-Dementia Complex and Amyotrophic Lateral Sclerosis of Guam

Both N-terminal and C-terminal fragments of Presenilin 1 colocalize with neurofibrillary tangles in neurons and dystrophic neurites of senile plaques in Alzheimer's disease

Progressive supranuclear palsy on Guam

Fractal analysis of senile plaque observed in various animal species

A TOPOGRAPHIC AND QUANTITATIVE STUDY OF NEURONS IN HUMAN SPINAL GRAY MATTER, WITH SPECIAL REFERENCE TO THEIR CHANGES IN AMYOTROPHIC LATERAL SCLEROSIS

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