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The roles of starvation and selective substrates in the emergence of araB‐lacZ fusion clones

The EMBO journal, 1994-11, Vol.13 (21), p.5229-5239

1994

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Overproduction of the Lon protease triggers inhibition of translation in Escherichia coli: involvement of the yefM‐yoeB toxin‐antitoxin system

Molecular microbiology, 2004-03, Vol.51 (6), p.1705-1717

2004

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Role of RecA Protein in Untargeted UV Mutagenesis of Bacteriophage λ : Evidence for the Requirement for the dinB Gene

Proceedings of the National Academy of Sciences - PNAS, 1986-06, Vol.83 (11), p.3904-3908

1986

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Multiple Pathways for SOS-Induced Mutagenesis in Escherichia coli: An Overexpression of dinB / dinP Results in Strongly Enhancing Mutagenesis in the Absence of any Exogenous Treatment to Damage DNA

Proceedings of the National Academy of Sciences - PNAS, 1997-12, Vol.94 (25), p.13792-13797

1997

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GATC sequences, DNA nicks and the MutH function in Escherichia coli mismatch repair

The EMBO journal, 1987-04, Vol.6 (4), p.1121-1127

1987

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GATC sequence and mismatch repair in Escherichia coli

The EMBO journal, 1986-08, Vol.5 (8), p.2009-2013

1986

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Control of Bacteriophage Mu Lysogenic Repression

Journal of molecular biology, 2005-10, Vol.353 (1), p.186-195

2005

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SOS mutator effect in E. coli mutants deficient in mismatch correction

The EMBO journal, 1984-04, Vol.3 (4), p.707-712

1984

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Starvation‐induced Mucts62‐mediated coding sequence fusion: a role for ClpXP, Lon, RpoS and Crp

Molecular microbiology, 1999-04, Vol.32 (2), p.327-343

1999

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Mechanism of SOS-induced targeted and untargeted mutagenesis in E. coli

Biochimie, 1985-03, Vol.67 (3), p.365-369

Maenhaut-Michel, G.

1985

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Effect of umuC mutations on targeted and untargeted ultraviolet mutagenesis in bacteriophage λ

Journal of molecular biology, 1984-07, Vol.177 (1), p.181-187

1984

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Nature of the SOS mutator activity: genetic characterization of untargeted mutagenesis in Escherichia coli

Molecular & general genetics, 1988-08, Vol.213 (2-3), p.491-498

1988

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Induction of frameshift mutations at hotspot sequences by carcinogen adducts

Recent results in cancer research, 1997, Vol.143, p.1

1997

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A umuDC-independent SOS pathway for frameshift mutagenesis

Molecular & general genetics, 1992-11, Vol.235 (2-3), p.373-380

1992

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Roles of recA mutant allele ( recA495) in frameshift mutagenesis

Biochimie, 1991-04, Vol.73 (4), p.491-495

1991

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Different structures of selected and unselected araB‐lacZ fusions

Molecular microbiology, 1997-03, Vol.23 (6), p.1133-1145

1997

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MucAB but not UmuDC proteins enhance -2 frameshift mutagenesis induced by N-2-acetylaminofluorene at alternating GC sequences

Molecular & general genetics, 1994-11, Vol.245 (3), p.279-285

1994

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Increased detection of the SOS mutator effect in Escherichia coli mutants deficient in mismatch correction

Biochimie, 1982-01, Vol.64 (8), p.661-663

1982

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Effects of Escherichia coli mutator genes mutH, mutL and mutS on 2-aminopurine induced DNA repair

Biochimie, 1982-01, Vol.64 (8), p.739-742

1982

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Specific strand loss in N-2-acetylaminofluorene-modified DNA

Journal of molecular biology, 1987-02, Vol.193 (4), p.651-659

1987

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Processing of mispaired and unpaired bases in heteroduplex DNA in E. coli

Biochimie, 1985-07, Vol.67 (7), p.745-752

1985

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DNA damage and repair in mutagenesis and carcinogenesis: implications of structure-activity relationships for cross-species extrapolation

Mutation research, 1996-06, Vol.353 (1-2), p.177

1996

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Regulation of bacteriophage Mu transposition

Genetica, 1994-01, Vol.93 (1-3), p.27-39

1994

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Adducts formed by the food mutagen 2-amino-3-methylimidazo(4, 5-f) quinoline induce frameshift mutations at hot spots through an SOS-independent pathway

Molecular & general genetics, 1997-02, Vol.253 (5), p.634-641

1997

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Conservation and diversification of genes by mismatch correction and SOS induction

Biochimie, 1982-01, Vol.64 (8), p.559-564

1982

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The roles of starvation and selective substrates in the emergence of araB‐lacZ fusion clones

Overproduction of the Lon protease triggers inhibition of translation in Escherichia coli: involvement of the yefM‐yoeB toxin‐antitoxin system

Role of RecA Protein in Untargeted UV Mutagenesis of Bacteriophage λ : Evidence for the Requirement for the dinB Gene

Multiple Pathways for SOS-Induced Mutagenesis in Escherichia coli: An Overexpression of dinB / dinP Results in Strongly Enhancing Mutagenesis in the Absence of any Exogenous Treatment to Damage DNA

GATC sequences, DNA nicks and the MutH function in Escherichia coli mismatch repair

GATC sequence and mismatch repair in Escherichia coli

Control of Bacteriophage Mu Lysogenic Repression

SOS mutator effect in E. coli mutants deficient in mismatch correction

Starvation‐induced Mucts62‐mediated coding sequence fusion: a role for ClpXP, Lon, RpoS and Crp

Mechanism of SOS-induced targeted and untargeted mutagenesis in E. coli

Effect of umuC mutations on targeted and untargeted ultraviolet mutagenesis in bacteriophage λ

Nature of the SOS mutator activity: genetic characterization of untargeted mutagenesis in Escherichia coli

Induction of frameshift mutations at hotspot sequences by carcinogen adducts

A umuDC-independent SOS pathway for frameshift mutagenesis

Roles of recA mutant allele ( recA495) in frameshift mutagenesis

Different structures of selected and unselected araB‐lacZ fusions

MucAB but not UmuDC proteins enhance -2 frameshift mutagenesis induced by N-2-acetylaminofluorene at alternating GC sequences

Increased detection of the SOS mutator effect in Escherichia coli mutants deficient in mismatch correction

Effects of Escherichia coli mutator genes mutH, mutL and mutS on 2-aminopurine induced DNA repair

Specific strand loss in N-2-acetylaminofluorene-modified DNA

Processing of mispaired and unpaired bases in heteroduplex DNA in E. coli

DNA damage and repair in mutagenesis and carcinogenesis: implications of structure-activity relationships for cross-species extrapolation

Regulation of bacteriophage Mu transposition

Adducts formed by the food mutagen 2-amino-3-methylimidazo(4, 5-f) quinoline induce frameshift mutations at hot spots through an SOS-independent pathway

Conservation and diversification of genes by mismatch correction and SOS induction

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