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Mesenchymal stem cells protect NOD mice from diabetes by inducing regulatory T cells

Diabetologia, 2009-07, Vol.52 (7), p.1391-1399

2009

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Recombinant Fabs of Human Monoclonal Antibodies Specific to the Middle Epitope of GAD65 Inhibit Type 1 Diabetes–Specific GAD65Abs

Diabetes (New York, N.Y.), 2003-11, Vol.52 (11), p.2689-2695

2003

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Reduction of the Risk of Relapse After Withdrawal of Medical Therapy for Graves' Disease

Thyroid (New York, N.Y.), 2002-10, Vol.12 (10), p.849-853

2002

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Suppressive Effect of Glutamic Acid Decarboxylase 65-Specific Autoimmune B Lymphocytes on Processing of T Cell Determinants Located Within the Antibody Epitope

The Journal of immunology (1950), 2002-07, Vol.169 (2), p.665-672

2002

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High-resolution autoreactive epitope mapping and structural modeling of the 65 kDa form of human glutamic acid decarboxylase

Journal of molecular biology, 1999-04, Vol.287 (5), p.983-999

1999

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Modulation of antigen presentation by autoreactive B cell clones specific for GAD65 from a type I diabetic patient

Clinical and experimental immunology, 2004-01, Vol.135 (1), p.74-84

2004

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Antibody-dependent cell-mediated cytotoxicity in autoimmune thyroid disease : Relationship to antithyroperoxidase antibodies

The journal of clinical endocrinology and metabolism, 1996-07, Vol.81 (7), p.2595-2600

1996

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Epitope analysis of GAD65Ab using fusion proteins and rFab

Journal of immunological methods, 2004-12, Vol.295 (1), p.101-109

2004

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Multiplicity of the antibody response to GAD65 in Type I diabetes

Clinical and experimental immunology, 2004-11, Vol.138 (2), p.337-341

2004

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Management of fetal thyroid goitres: a report of 11 cases in a single perinatal unit

Prenatal diagnosis, 2000-10, Vol.20 (10), p.799-806

2000

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The relationship between peripheral T cell reactivity to insulin, clinical remissions and cytokine production in type 1 (insulin-dependent) diabetes mellitus

The journal of clinical endocrinology and metabolism, 1999-07, Vol.84 (7), p.2419-2424

1999

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Four IgG anti-islet human monoclonal antibodies isolated from a type 1 diabetes patient recognize distinct epitopes of glutamic acid decarboxylase 65 and are somatically mutated

The Journal of immunology (1950), 1996-05, Vol.156 (9), p.3541-3549

1996

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Antithyroid drugs and Graves' disease: a prospective randomized evaluation of the efficacy of treatment duration

The journal of clinical endocrinology and metabolism, 1990-03, Vol.70 (3), p.675-679

1990

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The role of stimulating, function-blocking and growth-blocking anti-TSH receptor antibodies (TRAbs) in GD, Hashimoto's disease and in atrophic thyroiditis

Annales d'endocrinologie, 2003-02, Vol.64 (1), p.31-36

2003

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T cell response pattern to glutamic acid decarboxylase 65 (GAD65) peptides of newly diagnosed type 1 diabetic patients sharing susceptible HLA haplotypes

Clinical and experimental immunology, 1999-07, Vol.117 (1), p.30-37

1999

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Two amino acids in glutamic acid decarboxylase act in concert for maintainance of conformational determinants recognised by Type I diabetic autoantibodies

Diabetologia, 2000-07, Vol.43 (7), p.881-889

2000

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Anti TSH‐receptor antibodies in pregnant patients with autoimmune thyroid disorder

BJOG : an international journal of obstetrics and gynaecology, 1990-11, Vol.97 (11), p.1003-1008

1990

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Peptide specificity of high-titer anti-glutamic acid decarboxylase (GAD)65 autoantibodies

Immunology letters, 1998-07, Vol.62 (3), p.123-130

1998

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Evidence for several cell populations in human thyroid with distinct glycosphingolipid patterns

Biochimica et biophysica acta, 1990-01, Vol.1051 (1), p.1-5

1990

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Blocking anti-thyrotropin receptor antibodies desensitize cultured human thyroid cells

Endocrinology (Philadelphia), 1988-10, Vol.123 (4), p.2062-2066

1988

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Fab of a human monoclonal antibody specifically inhibits GAD65Ab in Type 1 diabetes

DIABETOLOGIA, 2003, Vol.46, p.A15

2003

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THYROID STIMULATING ANTIBODIES: AN AID TO THE STRATEGY OF TREATMENT OF GRAVES' DISEASE?

Clinical endocrinology (Oxford), 1984-09, Vol.21 (3), p.247-255

1984

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Monoclonal antibody approach to the relationship between immunological structure and biological activity of thyrotropin

Molecular endocrinology (Baltimore, Md.), 1988-07, Vol.2 (7), p.613-618

1988

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Assessment of Antibody Dependent Cell Cytotoxicity in Autoimmune Thyroid Disease Using Porcine Thyroid Cells

Autoimmunity (Chur, Switzerland), 1992, Vol.13 (3), p.177-185

1992

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Thyroid stimulating antibody: an index of thyroid stimulation in Graves' disease?

Acta endocrinologica (Copenhagen), 1987-09, Vol.116 (1), p.7-12

1987

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Mesenchymal stem cells protect NOD mice from diabetes by inducing regulatory T cells

Recombinant Fabs of Human Monoclonal Antibodies Specific to the Middle Epitope of GAD65 Inhibit Type 1 Diabetes–Specific GAD65Abs

Reduction of the Risk of Relapse After Withdrawal of Medical Therapy for Graves' Disease

Suppressive Effect of Glutamic Acid Decarboxylase 65-Specific Autoimmune B Lymphocytes on Processing of T Cell Determinants Located Within the Antibody Epitope

High-resolution autoreactive epitope mapping and structural modeling of the 65 kDa form of human glutamic acid decarboxylase

Modulation of antigen presentation by autoreactive B cell clones specific for GAD65 from a type I diabetic patient

Antibody-dependent cell-mediated cytotoxicity in autoimmune thyroid disease : Relationship to antithyroperoxidase antibodies

Epitope analysis of GAD65Ab using fusion proteins and rFab

Multiplicity of the antibody response to GAD65 in Type I diabetes

Management of fetal thyroid goitres: a report of 11 cases in a single perinatal unit

The relationship between peripheral T cell reactivity to insulin, clinical remissions and cytokine production in type 1 (insulin-dependent) diabetes mellitus

Four IgG anti-islet human monoclonal antibodies isolated from a type 1 diabetes patient recognize distinct epitopes of glutamic acid decarboxylase 65 and are somatically mutated

Antithyroid drugs and Graves' disease: a prospective randomized evaluation of the efficacy of treatment duration

The role of stimulating, function-blocking and growth-blocking anti-TSH receptor antibodies (TRAbs) in GD, Hashimoto's disease and in atrophic thyroiditis

T cell response pattern to glutamic acid decarboxylase 65 (GAD65) peptides of newly diagnosed type 1 diabetic patients sharing susceptible HLA haplotypes

Two amino acids in glutamic acid decarboxylase act in concert for maintainance of conformational determinants recognised by Type I diabetic autoantibodies

Anti TSH‐receptor antibodies in pregnant patients with autoimmune thyroid disorder

Peptide specificity of high-titer anti-glutamic acid decarboxylase (GAD)65 autoantibodies

Evidence for several cell populations in human thyroid with distinct glycosphingolipid patterns

Blocking anti-thyrotropin receptor antibodies desensitize cultured human thyroid cells

Fab of a human monoclonal antibody specifically inhibits GAD65Ab in Type 1 diabetes

THYROID STIMULATING ANTIBODIES: AN AID TO THE STRATEGY OF TREATMENT OF GRAVES' DISEASE?

Monoclonal antibody approach to the relationship between immunological structure and biological activity of thyrotropin

Assessment of Antibody Dependent Cell Cytotoxicity in Autoimmune Thyroid Disease Using Porcine Thyroid Cells

Thyroid stimulating antibody: an index of thyroid stimulation in Graves' disease?

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