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Role of the coagulation system in acetaminophen‐induced hepatotoxicity in mice

Hepatology (Baltimore, Md.), 2007-10, Vol.46 (4), p.1177-1186

2007

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Modest inflammation enhances diclofenac hepatotoxicity in rats: role of neutrophils and bacterial translocation

The Journal of pharmacology and experimental therapeutics, 2006-12, Vol.319 (3), p.1191-1199

2006

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Microarray analysis of lipopolysaccharide potentiation of trovafloxacin-induced liver injury in rats suggests a role for proinflammatory chemokines and neutrophils

The Journal of pharmacology and experimental therapeutics, 2006-03, Vol.316 (3), p.1080-1087

2006

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Adverse hepatic drug reactions: inflammatory episodes as consequence and contributor

Chemico-biological interactions, 2004-11, Vol.150 (1), p.35-51

2004

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Ranitidine treatment during a modest inflammatory response precipitates idiosyncrasy-like liver injury in rats

The Journal of pharmacology and experimental therapeutics, 2003-10, Vol.307 (1), p.9-16

2003

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Bacterial- and Viral-Induced Inflammation Increases Sensitivity to Acetaminophen Hepatotoxicity

Journal of Toxicology and Environmental Health, Part A, 2010-01, Vol.73 (1), p.58-73

2010

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity

The Journal of pharmacology and experimental therapeutics, 2011-08, Vol.338 (2), p.492-502

2011

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Inflammation and drug idiosyncrasy--is there a connection?

The Journal of pharmacology and experimental therapeutics, 2003-10, Vol.307 (1), p.1-8

2003

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Modeling inflammation–drug interactions in vitro: A rat Kupffer cell-hepatocyte coculture system

Toxicology in vitro, 2006-12, Vol.20 (8), p.1488-1499

2006

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Application of high-throughput Fourier-transform infrared spectroscopy in toxicology studies: contribution to a study on the development of an animal model for idiosyncratic toxicity

Toxicology letters, 2004-02, Vol.146 (3), p.197-205

2004

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Coagulation-mediated hypoxia and neutrophil-dependent hepatic injury in rats given lipopolysaccharide and ranitidine

The Journal of pharmacology and experimental therapeutics, 2005-09, Vol.314 (3), p.1023-1031

2005

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Comparison of TNFα to lipopolysaccharide as an inflammagen to characterize the idiosyncratic hepatotoxicity potential of drugs: Trovafloxacin as an example

International journal of molecular sciences, 2010-11, Vol.11 (11), p.4697-4714

2010

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Acute Exposure to Ozone Exacerbates Acetaminophen-Induced Liver Injury in Mice

Toxicological sciences, 2010-05, Vol.115 (1), p.267-285

2010

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Role of hepatic fibrin in idiosyncrasy‐like liver injury from lipopolysaccharide‐ranitidine coexposure in rats

Hepatology (Baltimore, Md.), 2004-12, Vol.40 (6), p.1342-1351

2004

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Design of Lipoxin A4 Stable Analogs That Block Transmigration and Adhesion of Human Neutrophils

Biochemistry (Easton), 1995-11, Vol.34 (44), p.14609-14615

1995

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Coagulation-dependent gene expression and liver injury in rats given lipopolysaccharide with ranitidine but not with famotidine

The Journal of pharmacology and experimental therapeutics, 2006-05, Vol.317 (2), p.635-643

2006

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Modes of Cell Death in Rat Liver after Monocrotaline Exposure

Toxicological sciences, 2004-01, Vol.77 (1), p.172-182

2004

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Metabonomic evaluation of idiosyncrasy-like liver injury in rats cotreated with ranitidine and lipopolysaccharide

Toxicology and applied pharmacology, 2006-04, Vol.212 (1), p.35-44

2006

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Lipoxin A4 Stable Analogs Are Potent Mimetics That Stimulate Human Monocytes and THP-1 Cells via a G-protein-linked Lipoxin A4 Receptor

The Journal of biological chemistry, 1997-03, Vol.272 (11), p.6972-6978

1997

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Increased neutrophil adherence and adhesion molecule mRNA expression in endothelial cells during selenium deficiency

Journal of leukocyte biology, 1999-05, Vol.65 (5), p.658-664

1999

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Lipoxin B4 regulates human monocyte/neutrophil adherence and motility: design of stable lipoxin B4 analogs with increased biologic activity

The FASEB journal, 1998-04, Vol.12 (6), p.487-494

1998

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15-Deoxy Prostaglandin J2 Enhances Allyl Alcohol–Induced Toxicity in Rat Hepatocytes

Toxicological sciences, 2004-02, Vol.77 (2), p.290-298

2004

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Unique Gene Expression and Hepatocellular Injury in the Lipopolysaccharide-Ranitidine Drug Idiosyncrasy Rat Model: Comparison with Famotidine

Toxicological sciences, 2006-04, Vol.90 (2), p.569-585

2006

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Gene Expression Analysis Points to Hemostasis in Livers of Rats Cotreated with Lipopolysaccharide and Ranitidine

Toxicological sciences, 2004-07, Vol.80 (1), p.203-213

2004

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Allyl Alcohol Activation of Protein Kinase C δ Leads to Cytotoxicity of Rat Hepatocytes

Chemical research in toxicology, 2003-05, Vol.16 (5), p.609-615

2003

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Role of the coagulation system in acetaminophen‐induced hepatotoxicity in mice

Modest inflammation enhances diclofenac hepatotoxicity in rats: role of neutrophils and bacterial translocation

Microarray analysis of lipopolysaccharide potentiation of trovafloxacin-induced liver injury in rats suggests a role for proinflammatory chemokines and neutrophils

Adverse hepatic drug reactions: inflammatory episodes as consequence and contributor

Ranitidine treatment during a modest inflammatory response precipitates idiosyncrasy-like liver injury in rats

Bacterial- and Viral-Induced Inflammation Increases Sensitivity to Acetaminophen Hepatotoxicity

The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity

Inflammation and drug idiosyncrasy--is there a connection?

Modeling inflammation–drug interactions in vitro: A rat Kupffer cell-hepatocyte coculture system

Application of high-throughput Fourier-transform infrared spectroscopy in toxicology studies: contribution to a study on the development of an animal model for idiosyncratic toxicity

Coagulation-mediated hypoxia and neutrophil-dependent hepatic injury in rats given lipopolysaccharide and ranitidine

Comparison of TNFα to lipopolysaccharide as an inflammagen to characterize the idiosyncratic hepatotoxicity potential of drugs: Trovafloxacin as an example

Acute Exposure to Ozone Exacerbates Acetaminophen-Induced Liver Injury in Mice

Role of hepatic fibrin in idiosyncrasy‐like liver injury from lipopolysaccharide‐ranitidine coexposure in rats

Design of Lipoxin A4 Stable Analogs That Block Transmigration and Adhesion of Human Neutrophils

Coagulation-dependent gene expression and liver injury in rats given lipopolysaccharide with ranitidine but not with famotidine

Modes of Cell Death in Rat Liver after Monocrotaline Exposure

Metabonomic evaluation of idiosyncrasy-like liver injury in rats cotreated with ranitidine and lipopolysaccharide

Lipoxin A4 Stable Analogs Are Potent Mimetics That Stimulate Human Monocytes and THP-1 Cells via a G-protein-linked Lipoxin A4 Receptor

Increased neutrophil adherence and adhesion molecule mRNA expression in endothelial cells during selenium deficiency

Lipoxin B4 regulates human monocyte/neutrophil adherence and motility: design of stable lipoxin B4 analogs with increased biologic activity

15-Deoxy Prostaglandin J2 Enhances Allyl Alcohol–Induced Toxicity in Rat Hepatocytes

Unique Gene Expression and Hepatocellular Injury in the Lipopolysaccharide-Ranitidine Drug Idiosyncrasy Rat Model: Comparison with Famotidine

Gene Expression Analysis Points to Hemostasis in Livers of Rats Cotreated with Lipopolysaccharide and Ranitidine

Allyl Alcohol Activation of Protein Kinase C δ Leads to Cytotoxicity of Rat Hepatocytes

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