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Role of Adipokines in Cardiovascular Disease

Circulation Journal, 2017/06/23, Vol.81(7), pp.920-928

2017

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S100a8/a9 Signaling Causes Mitochondrial Dysfunction and Cardiomyocyte Death in Response to Ischemic/Reperfusion Injury

Circulation (New York, N.Y.), 2019-08, Vol.140 (9), p.751-764

2019

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Small Extracellular Microvesicles Mediated Pathological Communications Between Dysfunctional Adipocytes and Cardiomyocytes as a Novel Mechanism Exacerbating Ischemia/Reperfusion Injury in Diabetic Mice

Circulation (New York, N.Y.), 2020-03, Vol.141 (12), p.968-983

2020

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Cardiac Fibroblast-Specific Activating Transcription Factor 3 Protects Against Heart Failure by Suppressing MAP2K3-p38 Signaling

Circulation (New York, N.Y.), 2017-05, Vol.135 (21), p.2041-2057

2017

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Cardiomyocyte-expressed farnesoid-X-receptor is a novel apoptosis mediator and contributes to myocardial ischaemia/reperfusion injury

European heart journal, 2013-06, Vol.34 (24), p.1834-1845

2013

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S100A8/A9 as a prognostic biomarker with causal effects for post-acute myocardial infarction heart failure

Nature communications, 2024-03, Vol.15 (1), p.2701-2701

2024

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C1q/Tumor Necrosis Factor–Related Protein-9, a Novel Adipocyte-Derived Cytokine, Attenuates Adverse Remodeling in the Ischemic Mouse Heart via Protein Kinase A Activation

Circulation (New York, N.Y.), 2013-09, Vol.128 (26_suppl_1 Suppl 1), p.S113-S120

2013

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C1q/tumor necrosis factor-related protein-3, a newly identified adipokine, is a novel antiapoptotic, proangiogenic, and cardioprotective molecule in the ischemic mouse heart

Circulation (New York, N.Y.), 2012-06, Vol.125 (25), p.3159-3169

2012

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miRNA-Mediated Suppression of a Cardioprotective Cardiokine as a Novel Mechanism Exacerbating Post-MI Remodeling by Sleep Breathing Disorders

Circulation research, 2020-01, Vol.126 (2), p.212-228

2020

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AdipoRon, the first orally active adiponectin receptor activator, attenuates postischemic myocardial apoptosis through both AMPK-mediated and AMPK-independent signalings

American journal of physiology: endocrinology and metabolism, 2015-08, Vol.309 (3), p.E275-E282

2015

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Adiponectin at Physiologically Relevant Concentrations Enhances the Vasorelaxative Effect of Acetylcholine via Cav-1/AdipoR-1 Signaling

PloS one, 2016-03, Vol.11 (3), p.e0152247-e0152247

2016

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Endothelial Autophagy in Coronary Microvascular Dysfunction and Cardiovascular Disease

Cells (Basel, Switzerland), 2022-06, Vol.11 (13), p.2081

2022

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Atg16L1 as a Novel Biomarker and Autophagy Gene for Diabetic Retinopathy

Journal of diabetes research, 2021, Vol.2021, p.5398645-11

2021

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G-Protein–Coupled Receptor Kinase 2–Mediated Desensitization of Adiponectin Receptor 1 in Failing Heart

Circulation (New York, N.Y.), 2015-04, Vol.131 (16), p.1392-1404

2015

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Mechanisms of adiponectin protection against diabetes-induced vascular endothelial injury

Sheng li hsüeh pao, 2019-06, Vol.71 (3), p.485-490

2019

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C1q-TNF-related protein-9, a novel cardioprotetcive cardiokine, requires proteolytic cleavage to generate a biologically active globular domain isoform

American journal of physiology: endocrinology and metabolism, 2015-05, Vol.308 (10), p.E891-E898

2015

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Identification of a CTRP9 C-Terminal polypeptide capable of enhancing bone-derived mesenchymal stem cell cardioprotection through promoting angiogenic exosome production

Redox biology, 2021-05, Vol.41, p.101929-101929, Article 101929

2021

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AMP-Activated Protein Kinase Deficiency Enhances Myocardial Ischemia/Reperfusion Injury but Has Minimal Effect on the Antioxidant/Antinitrative Protection of Adiponectin

Circulation (New York, N.Y.), 2009-02, Vol.119 (6), p.835-844

2009

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Cardiomyocyte-derived adiponectin is biologically active in protecting against myocardial ischemia-reperfusion injury

American journal of physiology: endocrinology and metabolism, 2010-03, Vol.298 (3), p.E663-E670

2010

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Adiponectin inhibits oxidative/nitrative stress during myocardial ischemia and reperfusion via PKA signaling

American journal of physiology: endocrinology and metabolism, 2013-12, Vol.305 (12), p.E1436-E1443

2013

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Hypoadiponectinaemia in diabetes mellitus type 2: molecular mechanisms and clinical significance

Clinical and experimental pharmacology & physiology, 2011-12, Vol.38 (12), p.897-904

2011

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Tumor necrosis factor-α and lymphotoxin-α mediate myocardial ischemic injury via TNF receptor 1, but are cardioprotective when activating TNF receptor 2

PloS one, 2013-05, Vol.8 (5), p.e60227

2013

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High glucose/High Lipids impair vascular adiponectin function via inhibition of caveolin-1/AdipoR1 signalsome formation

Free radical biology & medicine, 2015-12, Vol.89, p.473-485

2015

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Reduced cardioprotective action of adiponectin in high-fat diet-induced type II diabetic mice and its underlying mechanisms

Antioxidants & redox signaling, 2011-10, Vol.15 (7), p.1779

2011

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N-Cadherin Overexpression Mobilizes the Protective Effects of Mesenchymal Stromal Cells Against Ischemic Heart Injury Through a β-Catenin–Dependent Manner

Circulation research, 2020-03, Vol.126 (7), p.857-874

2020

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Role of Adipokines in Cardiovascular Disease

S100a8/a9 Signaling Causes Mitochondrial Dysfunction and Cardiomyocyte Death in Response to Ischemic/Reperfusion Injury

Small Extracellular Microvesicles Mediated Pathological Communications Between Dysfunctional Adipocytes and Cardiomyocytes as a Novel Mechanism Exacerbating Ischemia/Reperfusion Injury in Diabetic Mice

Cardiac Fibroblast-Specific Activating Transcription Factor 3 Protects Against Heart Failure by Suppressing MAP2K3-p38 Signaling

Cardiomyocyte-expressed farnesoid-X-receptor is a novel apoptosis mediator and contributes to myocardial ischaemia/reperfusion injury

S100A8/A9 as a prognostic biomarker with causal effects for post-acute myocardial infarction heart failure

C1q/Tumor Necrosis Factor–Related Protein-9, a Novel Adipocyte-Derived Cytokine, Attenuates Adverse Remodeling in the Ischemic Mouse Heart via Protein Kinase A Activation

C1q/tumor necrosis factor-related protein-3, a newly identified adipokine, is a novel antiapoptotic, proangiogenic, and cardioprotective molecule in the ischemic mouse heart

miRNA-Mediated Suppression of a Cardioprotective Cardiokine as a Novel Mechanism Exacerbating Post-MI Remodeling by Sleep Breathing Disorders

AdipoRon, the first orally active adiponectin receptor activator, attenuates postischemic myocardial apoptosis through both AMPK-mediated and AMPK-independent signalings

Adiponectin at Physiologically Relevant Concentrations Enhances the Vasorelaxative Effect of Acetylcholine via Cav-1/AdipoR-1 Signaling

Endothelial Autophagy in Coronary Microvascular Dysfunction and Cardiovascular Disease

Atg16L1 as a Novel Biomarker and Autophagy Gene for Diabetic Retinopathy

G-Protein–Coupled Receptor Kinase 2–Mediated Desensitization of Adiponectin Receptor 1 in Failing Heart

Mechanisms of adiponectin protection against diabetes-induced vascular endothelial injury

C1q-TNF-related protein-9, a novel cardioprotetcive cardiokine, requires proteolytic cleavage to generate a biologically active globular domain isoform

Identification of a CTRP9 C-Terminal polypeptide capable of enhancing bone-derived mesenchymal stem cell cardioprotection through promoting angiogenic exosome production

AMP-Activated Protein Kinase Deficiency Enhances Myocardial Ischemia/Reperfusion Injury but Has Minimal Effect on the Antioxidant/Antinitrative Protection of Adiponectin

Cardiomyocyte-derived adiponectin is biologically active in protecting against myocardial ischemia-reperfusion injury

Adiponectin inhibits oxidative/nitrative stress during myocardial ischemia and reperfusion via PKA signaling

Hypoadiponectinaemia in diabetes mellitus type 2: molecular mechanisms and clinical significance

Tumor necrosis factor-α and lymphotoxin-α mediate myocardial ischemic injury via TNF receptor 1, but are cardioprotective when activating TNF receptor 2

High glucose/High Lipids impair vascular adiponectin function via inhibition of caveolin-1/AdipoR1 signalsome formation

Reduced cardioprotective action of adiponectin in high-fat diet-induced type II diabetic mice and its underlying mechanisms

N-Cadherin Overexpression Mobilizes the Protective Effects of Mesenchymal Stromal Cells Against Ischemic Heart Injury Through a β-Catenin–Dependent Manner

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