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Characterising effects of impact velocity on brain and behaviour in a model of diffuse traumatic axonal injury

Neuroscience, 2013-09, Vol.248, p.17-29

2013

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Animal models of traumatic brain injury: Is there an optimal model to reproduce human brain injury in the laboratory?

Injury, 2010-07, Vol.41, p.S10-S13

2010

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The duality of the inflammatory response to traumatic brain injury

Molecular neurobiology, 2001-08, Vol.24 (1-3), p.169-181

2001

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Effects of high-intensity strength training on multiple risk factors for osteoporotic fractures. A randomized controlled trial

JAMA : the journal of the American Medical Association, 1994-12, Vol.272 (24), p.1909

1994

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Neurotransmitters in cerebrospinal fluid reflect pathological activity

European journal of clinical investigation, 1997-12, Vol.27 (12), p.1038-1043

1997

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Dose related association of impact activity and bone mineral density in pre-pubertal girls

Calcified tissue international, 2003-01, Vol.72 (1), p.24-31

2003

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Experimental Closed Head Injury: Analysis of Neurological Outcome, Blood-Brain Barrier Dysfunction, Intracranial Neutrophil Infiltration, and Neuronal Cell Death in Mice Deficient in Genes for Pro-Inflammatory Cytokines

Journal of cerebral blood flow and metabolism, 2000-02, Vol.20 (2), p.369-380

2000

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Production of cytokines following brain injury: beneficial and deleterious for the damaged tissue

Molecular psychiatry, 1997-03, Vol.2 (2), p.133-136

1997

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Cell activation and inflammatory response following traumatic axonal injury in the rat

Neuroreport, 2000-08, Vol.11 (11), p.2587-2590

2000

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Strength improvements with 1 yr of progressive resistance training in older women

Medicine and science in sports and exercise, 1995-06, Vol.27 (6), p.906-912

1995

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TGF-β is elevated in the CSF of patients with severe traumatic brain injuries and parallels blood-brain barrier function

Journal of neurotrauma, 1999-07, Vol.16 (7), p.617-628

1999

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Traumatic Brain Injury Increases β‐Amyloid Peptide 1‐42 in Cerebrospinal Fluid

Journal of neurochemistry, 1998-12, Vol.71 (6), p.2505-2509

1998

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Experimental Axonal Injury Triggers Interleukin-6 mRNA, Protein Synthesis and Release Into Cerebrospinal Fluid

Journal of cerebral blood flow and metabolism, 1999-02, Vol.19 (2), p.184-194

1999

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Role of cerebral inflammation after traumatic brain injury : A revisited concept

Shock (Augusta, Ga.), 2001-09, Vol.16 (3), p.165-177

2001

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Interleukin-6 and its soluble receptor in serum and cerebrospinal fluid after cerebral trauma

Neuroreport, 1999-02, Vol.10 (2), p.409-412

1999

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Cytokines and neuropathology

Trends in pharmacological sciences (Regular ed.), 1992, Vol.13 (7), p.286-291

1992

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IL-10 levels in cerebrospinal fluid and serum of patients with severe traumatic brain injury: relationship to IL-6, TNF- alpha , TGF- beta 1 and blood-brain barrier function

Journal of neuroimmunology, 1999-11, Vol.101 (2), p.211-221

1999

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Macrophage- and astrocyte-derived transforming growth factor β as a mediator of central nervous system dysfunction in acquired immune deficiency syndrome

The Journal of experimental medicine, 1991-04, Vol.173 (4), p.981-991

1991

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Soluble ICAM-1 in CSF coincides with the extent of cerebral damage in patients with severe traumatic brain injury

Journal of neurotrauma, 1998-06, Vol.15 (6), p.399-409

1998

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S-100β reflects the extent of injury and outcome, whereas neuronal specific enolase is a better indicator of neuroinflammation in patients with severe traumatic brain injury

Journal of neurotrauma, 2001-05, Vol.18 (5), p.491-498

2001

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Oligodendrocyte-derived J1-160/180 extracellular matrix glycoproteins are adhesive or repulsive depending on the partner cell type and time of interaction

Experimental neurology, 1990-07, Vol.109 (1), p.98-110

1990

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Early management of severe traumatic brain injury

The Lancet (British edition), 2012-09, Vol.380 (9847), p.1088-1098

2012

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Involvement of Pro- and Anti-Inflammatory Cytokines and Chemokines in the Pathophysiology of Traumatic Brain Injury

Neurotherapeutics, 2010-01, Vol.7 (1), p.22-30

2010

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The role of the complement system in traumatic brain injury

Brain Research Reviews, 1998-08, Vol.27 (3), p.243-256

1998

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The role of markers of inflammation in traumatic brain injury

Frontiers in neurology, 2013-01, Vol.4, p.18-18

2013

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Characterising effects of impact velocity on brain and behaviour in a model of diffuse traumatic axonal injury

Animal models of traumatic brain injury: Is there an optimal model to reproduce human brain injury in the laboratory?

The duality of the inflammatory response to traumatic brain injury

Effects of high-intensity strength training on multiple risk factors for osteoporotic fractures. A randomized controlled trial

Neurotransmitters in cerebrospinal fluid reflect pathological activity

Dose related association of impact activity and bone mineral density in pre-pubertal girls

Experimental Closed Head Injury: Analysis of Neurological Outcome, Blood-Brain Barrier Dysfunction, Intracranial Neutrophil Infiltration, and Neuronal Cell Death in Mice Deficient in Genes for Pro-Inflammatory Cytokines

Production of cytokines following brain injury: beneficial and deleterious for the damaged tissue

Cell activation and inflammatory response following traumatic axonal injury in the rat

Strength improvements with 1 yr of progressive resistance training in older women

TGF-β is elevated in the CSF of patients with severe traumatic brain injuries and parallels blood-brain barrier function

Traumatic Brain Injury Increases β‐Amyloid Peptide 1‐42 in Cerebrospinal Fluid

Experimental Axonal Injury Triggers Interleukin-6 mRNA, Protein Synthesis and Release Into Cerebrospinal Fluid

Role of cerebral inflammation after traumatic brain injury : A revisited concept

Interleukin-6 and its soluble receptor in serum and cerebrospinal fluid after cerebral trauma

Cytokines and neuropathology

IL-10 levels in cerebrospinal fluid and serum of patients with severe traumatic brain injury: relationship to IL-6, TNF- alpha , TGF- beta 1 and blood-brain barrier function

Macrophage- and astrocyte-derived transforming growth factor β as a mediator of central nervous system dysfunction in acquired immune deficiency syndrome

Soluble ICAM-1 in CSF coincides with the extent of cerebral damage in patients with severe traumatic brain injury

S-100β reflects the extent of injury and outcome, whereas neuronal specific enolase is a better indicator of neuroinflammation in patients with severe traumatic brain injury

Oligodendrocyte-derived J1-160/180 extracellular matrix glycoproteins are adhesive or repulsive depending on the partner cell type and time of interaction

Early management of severe traumatic brain injury

Involvement of Pro- and Anti-Inflammatory Cytokines and Chemokines in the Pathophysiology of Traumatic Brain Injury

The role of the complement system in traumatic brain injury

The role of markers of inflammation in traumatic brain injury

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