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Vpu enhances HIV-1 virus release in the absence of Bst-2 cell surface down-modulation and intracellular depletion

Proceedings of the National Academy of Sciences - PNAS, 2009-02, Vol.106 (8), p.2868-2873

2009

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Mannose Receptor 1 Restricts HIV Particle Release from Infected Macrophages

Cell reports (Cambridge), 2018-01, Vol.22 (3), p.786-795

2018

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HIV-1 Vpr Induces Degradation of Gelsolin, a Myeloid Cell-Specific Host Factor That Reduces Viral Infectivity by Inhibiting the Expression and Packaging of the HIV-1 Env Glycoprotein

mBio, 2023-02, Vol.14 (1), p.e0297322

2023

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The formation of cysteine-linked dimers of BST-2/tetherin is important for inhibition of HIV-1 virus release but not for sensitivity to Vpu

Retrovirology, 2009-09, Vol.6 (1), p.80-80, Article 80

2009

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Acetylation of SAMHD1 at lysine 580 is crucial for blocking HIV-1 infection

mBio, 2024-09, Vol.15 (9), p.e0195824

2024

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Enzymatically Active APOBEC3G Is Required for Efficient Inhibition of Human Immunodeficiency Virus Type 1

Journal of Virology, 2007-12, Vol.81 (24), p.13346-13353

2007

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Differential Effects of Human Immunodeficiency Virus Type 1 Vpu on the Stability of BST-2/Tetherin

Journal of Virology, 2011-03, Vol.85 (6), p.2611-2619

2011

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Apolipoprotein E is an HIV-1-inducible inhibitor of viral production and infectivity in macrophages

PLoS pathogens, 2018-11, Vol.14 (11), p.e1007372-e1007372

2018

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The Human Immunodeficiency Virus Type 1 Vif Protein Reduces Intracellular Expression and Inhibits Packaging of APOBEC3G (CEM15), a Cellular Inhibitor of Virus Infectivity

Journal of Virology, 2003-11, Vol.77 (21), p.11398-11407

2003

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Identification and characterization of naturally occurring splice variants of SAMHD1

Retrovirology, 2012-10, Vol.9 (1), p.86-86, Article 86

2012

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Human Immunodeficiency Virus Type 1 Vif Inhibits Packaging and Antiviral Activity of a Degradation-Resistant APOBEC3G Variant

Journal of Virology, 2007-08, Vol.81 (15), p.8236-8246

2007

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The Expression of Functional Vpx during Pathogenic SIVmac Infections of Rhesus Macaques Suppresses SAMHD1 in CD4+ Memory T Cells

PLoS pathogens, 2015-05, Vol.11 (5), p.e1004928-e1004928

2015

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Viral RNA Is Required for the Association of APOBEC3G with Human Immunodeficiency Virus Type 1 Nucleoprotein Complexes

Journal of Virology, 2005-05, Vol.79 (9), p.5870-5874

2005

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Targeting APOBEC3A to the viral nucleoprotein complex confers antiviral activity

Retrovirology, 2007-08, Vol.4 (1), p.61-61, Article 61

2007

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Antibody-Mediated Enhancement of HIV-1 and HIV-2 Production from BST-2/Tetherin-Positive Cells

Journal of Virology, 2011-11, Vol.85 (22), p.11981-11994

2011

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Stably Expressed APOBEC3F Has Negligible Antiviral Activity

Journal of Virology, 2010-11, Vol.84 (21), p.11067-11075

2010

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Identification of Dominant Negative Human Immunodeficiency Virus Type 1 Vif Mutants That Interfere with the Functional Inactivation of APOBEC3G by Virus-Encoded Vif

Journal of Virology, 2010-05, Vol.84 (10), p.5201-5211

2010

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Analysis of the contribution of cellular and viral RNA to the packaging of APOBEC3G into HIV-1 virions

Retrovirology, 2007-07, Vol.4 (1), p.48-48, Article 48

2007

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Expression of Thyroid Transcription Factor-1 in Normal and Neoplastic Lung Tissues

Modern pathology, 2002-10, Vol.15 (10), p.1058-1067

2002

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Monomeric APOBEC3G Is Catalytically Active and Has Antiviral Activity

Journal of Virology, 2006-05, Vol.80 (10), p.4673-4682

2006

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Differential Sensitivity of “Old” versus “New” APOBEC3G to Human Immunodeficiency Virus Type 1 Vif

Journal of Virology, 2009-01, Vol.83 (2), p.1156-1160

2009

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Production of infectious virus and degradation of APOBEC3G are separable functional properties of human immunodeficiency virus type 1 Vif

Virology (New York, N.Y.), 2007-12, Vol.369 (2), p.329-339

2007

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Production of infectious human immunodeficiency virus type 1 does not require depletion of APOBEC3G from virus-producing cells

Retrovirology, 2004-09, Vol.1 (1), p.27-27

2004

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APOBEC3B Potently Restricts HIV-2 but Not HIV-1 in a Vif-Dependent Manner

Journal of virology, 2021-11, Vol.95 (23), p.e0117021-e0117021

2021

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Encapsidation of APOBEC3G into HIV-1 virions involves lipid raft association and does not correlate with APOBEC3G oligomerization

Retrovirology, 2009-11, Vol.6 (1), p.99-99, Article 99

2009

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Vpu enhances HIV-1 virus release in the absence of Bst-2 cell surface down-modulation and intracellular depletion

Mannose Receptor 1 Restricts HIV Particle Release from Infected Macrophages

HIV-1 Vpr Induces Degradation of Gelsolin, a Myeloid Cell-Specific Host Factor That Reduces Viral Infectivity by Inhibiting the Expression and Packaging of the HIV-1 Env Glycoprotein

The formation of cysteine-linked dimers of BST-2/tetherin is important for inhibition of HIV-1 virus release but not for sensitivity to Vpu

Acetylation of SAMHD1 at lysine 580 is crucial for blocking HIV-1 infection

Enzymatically Active APOBEC3G Is Required for Efficient Inhibition of Human Immunodeficiency Virus Type 1

Differential Effects of Human Immunodeficiency Virus Type 1 Vpu on the Stability of BST-2/Tetherin

Apolipoprotein E is an HIV-1-inducible inhibitor of viral production and infectivity in macrophages

The Human Immunodeficiency Virus Type 1 Vif Protein Reduces Intracellular Expression and Inhibits Packaging of APOBEC3G (CEM15), a Cellular Inhibitor of Virus Infectivity

Identification and characterization of naturally occurring splice variants of SAMHD1

Human Immunodeficiency Virus Type 1 Vif Inhibits Packaging and Antiviral Activity of a Degradation-Resistant APOBEC3G Variant

The Expression of Functional Vpx during Pathogenic SIVmac Infections of Rhesus Macaques Suppresses SAMHD1 in CD4+ Memory T Cells

Viral RNA Is Required for the Association of APOBEC3G with Human Immunodeficiency Virus Type 1 Nucleoprotein Complexes

Targeting APOBEC3A to the viral nucleoprotein complex confers antiviral activity

Antibody-Mediated Enhancement of HIV-1 and HIV-2 Production from BST-2/Tetherin-Positive Cells

Stably Expressed APOBEC3F Has Negligible Antiviral Activity

Identification of Dominant Negative Human Immunodeficiency Virus Type 1 Vif Mutants That Interfere with the Functional Inactivation of APOBEC3G by Virus-Encoded Vif

Analysis of the contribution of cellular and viral RNA to the packaging of APOBEC3G into HIV-1 virions

Expression of Thyroid Transcription Factor-1 in Normal and Neoplastic Lung Tissues

Monomeric APOBEC3G Is Catalytically Active and Has Antiviral Activity

Differential Sensitivity of “Old” versus “New” APOBEC3G to Human Immunodeficiency Virus Type 1 Vif

Production of infectious virus and degradation of APOBEC3G are separable functional properties of human immunodeficiency virus type 1 Vif

Production of infectious human immunodeficiency virus type 1 does not require depletion of APOBEC3G from virus-producing cells

APOBEC3B Potently Restricts HIV-2 but Not HIV-1 in a Vif-Dependent Manner

Encapsidation of APOBEC3G into HIV-1 virions involves lipid raft association and does not correlate with APOBEC3G oligomerization

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