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Biology and biochemistry of proteinases in tumor invasion

Physiological reviews, 1993, Vol.73 (1), p.161-195

1993

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Membrane-type 1 matrix metalloproteinase controls osteo- and chondrogenesis by a proteolysis-independent mechanism mediated by its cytoplasmic tail

Osteoarthritis and cartilage, 2015-04, Vol.23, p.A64-A64

2015

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Elevated expression of periostin in human osteoarthritis cartilage and its potential role in matrix degradation via MMP-13

Osteoarthritis and cartilage, 2015-04, Vol.23, p.A135-A136

2015

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Extracellular matrix remodeling by metalloproteinases and plasminogen activators

Kidney international. Supplement, 1995-06, Vol.49, p.S12

Mignatti, P

1995

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Thrombin cleaves the high molecular weight forms of basic fibroblast growth factor (FGF-2): a novel mechanism for the control of FGF-2 and thrombin activity

Oncogene, 2008-04, Vol.27 (18), p.2594-2601

2008

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Mitral Valve Prolapse Gene Expression Profiling: A Microarray Study

The Journal of surgical research, 2011, Vol.165 (2), p.171-171

2011

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Basic Fibroblast Growth Factor Released by Single, Isolated Cells Stimulates their Migration in an Autocrine Manner

Proceedings of the National Academy of Sciences - PNAS, 1991-12, Vol.88 (24), p.11007-11011

1991

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Tumor invasion through the human amniotic membrane: Requirement for a proteinase cascade

Cell, 1986-11, Vol.47 (4), p.487-498

1986

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Basic Fibroblast Growth Factor-Induced Activation of Latent Transforming Growth Factor β in Endothelial Cells: Regulation of Plasminogen Activator Activity

The Journal of cell biology, 1992-08, Vol.118 (4), p.901-909

1992

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Activation of progelatinase A (MMP-2) by neutrophil elastase, cathepsin G, and proteinase-3: A role for inflammatory cells in tumor invasion and angiogenesis

Journal of cellular physiology, 2001-11, Vol.189 (2), p.197-206

2001

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Expression of the Urokinase Receptor in Vascular Endothelial Cells Is Stimulated by Basic Fibroblast Growth Factor

The Journal of cell biology, 1991-06, Vol.113 (5), p.1193-1201

1991

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In Vitro Angiogenesis on the Human Amniotic Membrane: Requirement for Basic Fibroblast Growth Factor-Induced Proteinases

The Journal of cell biology, 1989-02, Vol.108 (2), p.671-682

1989

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Fibroblast Growth Factor-2 (FGF-2) Induces Vascular Endothelial Growth Factor (VEGF) Expression in the Endothelial Cells of Forming Capillaries: An Autocrine Mechanism Contributing to Angiogenesis

The Journal of cell biology, 1998-06, Vol.141 (7), p.1659-1673

1998

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Plasmin activates pro-matrix metalloproteinase-2 with a membrane-type 1 matrix metalloproteinase-dependent mechanism

Journal of cellular physiology, 2002-08, Vol.192 (2), p.160-170

2002

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Trophic effects of platelets on cultured endothelial cells are mediated by platelet-associated fibroblast growth factor-2 (FGF-2) and vascular endothelial growth factor (VEGF)

Thrombosis and haemostasis, 2002-11, Vol.88 (5), p.834-842

2002

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Control of type IV collagenase activity by components of the urokinase-plasmin system: a regulatory mechanism with cell-bound reactants

The EMBO journal, 1997-05, Vol.16 (9), p.2319-2332

1997

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Plasminogen activators and angiogenesis

Current topics in microbiology and immunology, 1996, Vol.213 ( Pt 1), p.33

1996

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Shedding of Membrane Vesicles Mediates Fibroblast Growth Factor-2 Release from Cells

The Journal of biological chemistry, 2003-12, Vol.278 (51), p.51911-51919

2003

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Expression of von Willebrand factor, an endothelial cell marker, is up‐regulated by angiogenesis factors: A potential method for objective assessment of tumor angiogenesis

International journal of cancer, 2000-01, Vol.85 (2), p.281-288

2000

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Matrix metalloproteinase expression in vein grafts: role of inflammatory mediators and extracellular signal-regulated kinases-1 and -2

American journal of physiology. Heart and circulatory physiology, 2006-04, Vol.290 (4), p.H1651-H1659

2006

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Expression of von Willebrand factor, an endothelial cell marker, is up-regulated by angiogenesis factors : A potential method for objective assessment of tumor angiogenesis

International journal of cancer, 2000-01, Vol.85 (2), p.281-288

2000

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Lack of ERK activation and cell migration in FGF‐2‐deficient endothelial cells

The FASEB journal, 2002-04, Vol.16 (6), p.598-600

2002

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Basic fibroblast growth factor, a protein devoid of secretory signal sequence, is released by cells via a pathway independent of the endoplasmic reticulum-Golgi complex

Journal of cellular physiology, 1992-04, Vol.151 (1), p.81-93

1992

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Activation of tumor cell matrix metalloproteinase-2 by neutrophil proteinases requires expression of membrane-type 1 matrix metalloproteinase

Surgery, 1998-08, Vol.124 (2), p.232-238

1998

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Monoclonal antibody to vascular endothelial-cadherin is a potent inhibitor of angiogenesis, tumor growth, and metastasis

Cancer research (Chicago, Ill.), 2000-12, Vol.60 (24), p.6805-6810

2000

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Biology and biochemistry of proteinases in tumor invasion

Membrane-type 1 matrix metalloproteinase controls osteo- and chondrogenesis by a proteolysis-independent mechanism mediated by its cytoplasmic tail

Elevated expression of periostin in human osteoarthritis cartilage and its potential role in matrix degradation via MMP-13

Extracellular matrix remodeling by metalloproteinases and plasminogen activators

Thrombin cleaves the high molecular weight forms of basic fibroblast growth factor (FGF-2): a novel mechanism for the control of FGF-2 and thrombin activity

Mitral Valve Prolapse Gene Expression Profiling: A Microarray Study

Basic Fibroblast Growth Factor Released by Single, Isolated Cells Stimulates their Migration in an Autocrine Manner

Tumor invasion through the human amniotic membrane: Requirement for a proteinase cascade

Basic Fibroblast Growth Factor-Induced Activation of Latent Transforming Growth Factor β in Endothelial Cells: Regulation of Plasminogen Activator Activity

Activation of progelatinase A (MMP-2) by neutrophil elastase, cathepsin G, and proteinase-3: A role for inflammatory cells in tumor invasion and angiogenesis

Expression of the Urokinase Receptor in Vascular Endothelial Cells Is Stimulated by Basic Fibroblast Growth Factor

In Vitro Angiogenesis on the Human Amniotic Membrane: Requirement for Basic Fibroblast Growth Factor-Induced Proteinases

Fibroblast Growth Factor-2 (FGF-2) Induces Vascular Endothelial Growth Factor (VEGF) Expression in the Endothelial Cells of Forming Capillaries: An Autocrine Mechanism Contributing to Angiogenesis

Plasmin activates pro-matrix metalloproteinase-2 with a membrane-type 1 matrix metalloproteinase-dependent mechanism

Trophic effects of platelets on cultured endothelial cells are mediated by platelet-associated fibroblast growth factor-2 (FGF-2) and vascular endothelial growth factor (VEGF)

Control of type IV collagenase activity by components of the urokinase-plasmin system: a regulatory mechanism with cell-bound reactants

Plasminogen activators and angiogenesis

Shedding of Membrane Vesicles Mediates Fibroblast Growth Factor-2 Release from Cells

Expression of von Willebrand factor, an endothelial cell marker, is up‐regulated by angiogenesis factors: A potential method for objective assessment of tumor angiogenesis

Matrix metalloproteinase expression in vein grafts: role of inflammatory mediators and extracellular signal-regulated kinases-1 and -2

Expression of von Willebrand factor, an endothelial cell marker, is up-regulated by angiogenesis factors : A potential method for objective assessment of tumor angiogenesis

Lack of ERK activation and cell migration in FGF‐2‐deficient endothelial cells

Basic fibroblast growth factor, a protein devoid of secretory signal sequence, is released by cells via a pathway independent of the endoplasmic reticulum-Golgi complex

Activation of tumor cell matrix metalloproteinase-2 by neutrophil proteinases requires expression of membrane-type 1 matrix metalloproteinase

Monoclonal antibody to vascular endothelial-cadherin is a potent inhibitor of angiogenesis, tumor growth, and metastasis

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