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The Initial Hepatitis B Virus-Hepatocyte Genomic Integrations and Their Role in Hepatocellular Oncogenesis

International journal of molecular sciences, 2023-10, Vol.24 (19), p.14849

Michalak, Tomasz I

2023

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Diverse Virus and Host-Dependent Mechanisms Influence the Systemic and Intrahepatic Immune Responses in the Woodchuck Model of Hepatitis B

Frontiers in immunology, 2020-05, Vol.11, p.853

Michalak, Tomasz I

2020

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Hepadnaviral Lymphotropism and Its Relevance to HBV Persistence and Pathogenesis

Frontiers in microbiology, 2021-08, Vol.12, p.695384-695384

2021

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Differential Expression of Woodchuck Toll-Like Receptors 1-10 in Distinct Forms of Infection and Stages of Hepatitis in Experimental Hepatitis B Virus Infection

Frontiers in microbiology, 2018-12, Vol.9, p.3007-3007

2018

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Primary seronegative but molecularly evident hepadnaviral infection engages liver and induces hepatocarcinoma in the woodchuck model of hepatitis B

PLoS pathogens, 2014-08, Vol.10 (8), p.e1004332-e1004332

2014

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Identification of antibodies cross-reactive with woodchuck immune cells and activation of virus-specific and global cytotoxic T cell responses by anti-PD-1 and anti-PD-L1 in experimental chronic hepatitis B and persistent occult hepadnaviral infection

Frontiers in microbiology, 2022-12, Vol.13, p.1011070-1011070

2022

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Persistent occult hepatitis B virus infection: experimental findings and clinical implications

World journal of gastroenterology : WJG, 2007-11, Vol.13 (43), p.5682

2007

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Reactivation of hepatitis B after liver transplantation: Current knowledge, molecular mechanisms and implications in management

World journal of hepatology, 2018-03, Vol.10 (3), p.352-370

2018

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Hepatitis C Virus Infection of Human T Lymphocytes Is Mediated by CD5

Journal of Virology, 2012-04, Vol.86 (7), p.3723-3735

2012

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Hepatitis C Virus Persistence after Spontaneous or Treatment-Induced Resolution of Hepatitis C

Journal of Virology, 2004-06, Vol.78 (11), p.5867-5874

2004

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Hepatitis C Virus Replicates in the Same Immune Cell Subsets in Chronic Hepatitis C and Occult Infection

Gastroenterology (New York, N.Y. 1943), 2008-03, Vol.134 (3), p.812-822

2008

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Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C

PloS one, 2015-10, Vol.10 (10), p.e0140312-e0140312

2015

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Intrahepatic Expression of Genes Affiliated with Innate and Adaptive Immune Responses Immediately after Invasion and during Acute Infection with Woodchuck Hepadnavirus

Journal of Virology, 2008-09, Vol.82 (17), p.8579-8591

2008

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Differential expression of candidate virus receptors in human T lymphocytes prone or resistant to infection with patient-derived hepatitis C virus

PloS one, 2013-04, Vol.8 (4), p.e62159-e62159

2013

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Hepatocyte cytotoxicity is facilitated by asialoglycoprotein receptor

Hepatology (Baltimore, Md.), 2011-09, Vol.54 (3), p.1043-1050

2011

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Persistence of hepatitis C virus during and after otherwise clinically successful treatment of chronic hepatitis C with standard pegylated interferon α-2b and ribavirin therapy

PloS one, 2013-11, Vol.8 (11), p.e80078

2013

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Hepatitis C virus persisting after clinically apparent sustained virological response to antiviral therapy retains infectivity in vitro

Hepatology (Baltimore, Md.), 2009-05, Vol.49 (5), p.1431-1441

2009

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Repeated Exposure to Trace Amounts of Woodchuck Hepadnavirus Induces Molecularly Evident Infection and Virus-Specific T Cell Response in the Absence of Serological Infection Markers and Hepatitis

Journal of Virology, 2013-01, Vol.87 (2), p.1035-1048

2013

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Primary Occult Hepadnavirus Infection Induces Virus-Specific T-Cell and Aberrant Cytokine Responses in the Absence of Antiviral Antibody Reactivity in the Woodchuck Model of Hepatitis B Virus Infection

Journal of Virology, 2009-04, Vol.83 (8), p.3861-3876

2009

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Occult persistence and lymphotropism of hepatitis C virus infection

World journal of gastroenterology : WJG, 2008-05, Vol.14 (18), p.2789-2793

2008

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Immune cell subsets and their gene expression profiles from human PBMC isolated by Vacutainer Cell Preparation Tube (CPT™) and standard density gradient

BMC immunology, 2015-08, Vol.16 (1), p.48-48, Article 48

2015

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Kinetics of DNA damage repair response accompanying initial hepadnavirus-host genomic integration in woodchuck hepatitis virus infection of hepatocyte

Cancer genetics, 2020-06, Vol.244, p.1-10

2020

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Authentic Patient-Derived Hepatitis C Virus Infects and Productively Replicates in Primary CD4 + and CD8 + T Lymphocytes In Vitro

Journal of virology, 2018-02, Vol.92 (3)

2018

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Low Doses of Hepadnavirus Induce Infection of the Lymphatic System That Does Not Engage the Liver

Journal of Virology, 2004-02, Vol.78 (4), p.1730-1738

2004

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Repeated Passage of Wild-Type Woodchuck Hepatitis Virus in Lymphoid Cells Does Not Generate Cell Type-Specific Variants or Alter Virus Infectivity

Journal of Virology, 2008-08, Vol.82 (15), p.7540-7550

2008

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The Initial Hepatitis B Virus-Hepatocyte Genomic Integrations and Their Role in Hepatocellular Oncogenesis

Diverse Virus and Host-Dependent Mechanisms Influence the Systemic and Intrahepatic Immune Responses in the Woodchuck Model of Hepatitis B

Hepadnaviral Lymphotropism and Its Relevance to HBV Persistence and Pathogenesis

Differential Expression of Woodchuck Toll-Like Receptors 1-10 in Distinct Forms of Infection and Stages of Hepatitis in Experimental Hepatitis B Virus Infection

Primary seronegative but molecularly evident hepadnaviral infection engages liver and induces hepatocarcinoma in the woodchuck model of hepatitis B

Identification of antibodies cross-reactive with woodchuck immune cells and activation of virus-specific and global cytotoxic T cell responses by anti-PD-1 and anti-PD-L1 in experimental chronic hepatitis B and persistent occult hepadnaviral infection

Persistent occult hepatitis B virus infection: experimental findings and clinical implications

Reactivation of hepatitis B after liver transplantation: Current knowledge, molecular mechanisms and implications in management

Hepatitis C Virus Infection of Human T Lymphocytes Is Mediated by CD5

Hepatitis C Virus Persistence after Spontaneous or Treatment-Induced Resolution of Hepatitis C

Hepatitis C Virus Replicates in the Same Immune Cell Subsets in Chronic Hepatitis C and Occult Infection

Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C

Intrahepatic Expression of Genes Affiliated with Innate and Adaptive Immune Responses Immediately after Invasion and during Acute Infection with Woodchuck Hepadnavirus

Differential expression of candidate virus receptors in human T lymphocytes prone or resistant to infection with patient-derived hepatitis C virus

Hepatocyte cytotoxicity is facilitated by asialoglycoprotein receptor

Persistence of hepatitis C virus during and after otherwise clinically successful treatment of chronic hepatitis C with standard pegylated interferon α-2b and ribavirin therapy

Hepatitis C virus persisting after clinically apparent sustained virological response to antiviral therapy retains infectivity in vitro

Repeated Exposure to Trace Amounts of Woodchuck Hepadnavirus Induces Molecularly Evident Infection and Virus-Specific T Cell Response in the Absence of Serological Infection Markers and Hepatitis

Primary Occult Hepadnavirus Infection Induces Virus-Specific T-Cell and Aberrant Cytokine Responses in the Absence of Antiviral Antibody Reactivity in the Woodchuck Model of Hepatitis B Virus Infection

Occult persistence and lymphotropism of hepatitis C virus infection

Immune cell subsets and their gene expression profiles from human PBMC isolated by Vacutainer Cell Preparation Tube (CPT™) and standard density gradient

Kinetics of DNA damage repair response accompanying initial hepadnavirus-host genomic integration in woodchuck hepatitis virus infection of hepatocyte

Authentic Patient-Derived Hepatitis C Virus Infects and Productively Replicates in Primary CD4 + and CD8 + T Lymphocytes In Vitro

Low Doses of Hepadnavirus Induce Infection of the Lymphatic System That Does Not Engage the Liver

Repeated Passage of Wild-Type Woodchuck Hepatitis Virus in Lymphoid Cells Does Not Generate Cell Type-Specific Variants or Alter Virus Infectivity

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