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Requirement for Atm in Ionizing Radiation-Induced Cell Death in the Developing Central Nervous System

Science (American Association for the Advancement of Science), 1998-05, Vol.280 (5366), p.1089-1091

1998

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Responding to DNA double strand breaks in the nervous system

Neuroscience, 2007-04, Vol.145 (4), p.1365-1374

2007

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Puma is an essential mediator of p53-dependent and -independent apoptotic pathways

Cancer cell, 2003-10, Vol.4 (4), p.321-328

2003

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Phosphorylation of SMC1 is a critical downstream event in the ATM-NBS1-BRCA1 pathway

Genes & development, 2004-06, Vol.18 (12), p.1423-1438

2004

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Rint1 inactivation triggers genomic instability, ER stress and autophagy inhibition in the brain

Cell death and differentiation, 2016-03, Vol.23 (3), p.454-468

2016

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Gustducin is a taste-cell-specific G protein closely related to the transducins

Nature (London), 1992-06, Vol.357 (6379), p.563-569

1992

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Cross-species epigenetics identifies a critical role for VAV1 in SHH subgroup medulloblastoma maintenance

Oncogene, 2015-09, Vol.34 (36), p.4746-4757

2015

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DNA double-strand break repair and development

Oncogene, 2007-12, Vol.26 (56), p.7799-7808

2007

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Loss of the ARF tumor suppressor reverses premature replicative arrest but not radiation hypersensitivity arising from disabled atm function

Cancer research (Chicago, Ill.), 1999-05, Vol.59 (10), p.2464-2469

1999

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Six3 repression of Wnt signaling in the anterior neuroectoderm is essential for vertebrate forebrain development

Genes & development, 2003-02, Vol.17 (3), p.368-379

2003

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ATM dependent apoptosis in the nervous system

Apoptosis (London), 2000-12, Vol.5 (6), p.523-529

2000

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DNA ligase IV suppresses medulloblastoma formation

Cancer research (Chicago, Ill.), 2002-11, Vol.62 (22), p.6395-6399

2002

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Atm expression patterns suggest a contribution from the peripheral nervous system to the phenotype of ataxia–telangiectasia

Neuroscience, 1998-10, Vol.86 (4), p.1045-1054

1998

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Pineal Opsin: A Nonvisual Opsin Expressed in Chick Pineal

Science (American Association for the Advancement of Science), 1995-03, Vol.267 (5203), p.1502-1506

1995

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Linking DNA damage and neurodegeneration

Trends in Neurosciences, 2000-09, Vol.23 (9), p.417-424

2000

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Ataxia-telangiectasia: an inherited disorder of ionizing-radiation sensitivity in man: progress in the elucidation of the underlying biochemical defect

Human genetics, 1987-03, Vol.75 (3), p.197-208

MCKINNON, P. J

1987

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Ataxia Telangiectasia Mutated-Dependent Apoptosis after Genotoxic Stress in the Developing Nervous System Is Determined by Cellular Differentiation Status

The Journal of neuroscience, 2001-09, Vol.21 (17), p.6687-6693

2001

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Mice heterozygous for mutation in Atm , the gene involved in ataxia-telangiectasia, have heightened susceptibility to cancer

Nature genetics, 2002-09, Vol.32 (1), p.185-190

2002

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Ataxia telangiectasia mutated deficiency affects astrocyte growth but not radiosensitivity

Cancer research (Chicago, Ill.), 1999-10, Vol.59 (20), p.5294-5298

1999

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Defective neurogenesis resulting from DNA ligase IV deficiency requires Atm

Genes & development, 2000-10, Vol.14 (20), p.2576-2580

2000

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The DNA double-strand break response in the nervous system

DNA Repair, 2004-08, Vol.3 (8), p.1141-1147

2004

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SC1: a marker for astrocytes in the adult rodent brain is upregulated during reactive astrocytosis

Brain research, 1996-02, Vol.709 (1), p.27-36

1996

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Atm and Bax Cooperate in Ionizing Radiation-Induced Apoptosis in the Central Nervous System

Proceedings of the National Academy of Sciences - PNAS, 2000-01, Vol.97 (2), p.889-894

2000

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ATM and ataxia telangiectasia: Second in Molecular Medicine Review Series

EMBO reports, 2004-08, Vol.5 (8), p.772-776

McKinnon, Peter J

2004

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The Centrosomal, Putative Tumor Suppressor Protein TACC2 Is Dispensable for Normal Development, and Deficiency Does Not Lead to Cancer

Molecular and Cellular Biology, 2004-07, Vol.24 (14), p.6403-6409

2004

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Requirement for Atm in Ionizing Radiation-Induced Cell Death in the Developing Central Nervous System

Responding to DNA double strand breaks in the nervous system

Puma is an essential mediator of p53-dependent and -independent apoptotic pathways

Phosphorylation of SMC1 is a critical downstream event in the ATM-NBS1-BRCA1 pathway

Rint1 inactivation triggers genomic instability, ER stress and autophagy inhibition in the brain

Gustducin is a taste-cell-specific G protein closely related to the transducins

Cross-species epigenetics identifies a critical role for VAV1 in SHH subgroup medulloblastoma maintenance

DNA double-strand break repair and development

Loss of the ARF tumor suppressor reverses premature replicative arrest but not radiation hypersensitivity arising from disabled atm function

Six3 repression of Wnt signaling in the anterior neuroectoderm is essential for vertebrate forebrain development

ATM dependent apoptosis in the nervous system

DNA ligase IV suppresses medulloblastoma formation

Atm expression patterns suggest a contribution from the peripheral nervous system to the phenotype of ataxia–telangiectasia

Pineal Opsin: A Nonvisual Opsin Expressed in Chick Pineal

Linking DNA damage and neurodegeneration

Ataxia-telangiectasia: an inherited disorder of ionizing-radiation sensitivity in man: progress in the elucidation of the underlying biochemical defect

Ataxia Telangiectasia Mutated-Dependent Apoptosis after Genotoxic Stress in the Developing Nervous System Is Determined by Cellular Differentiation Status

Mice heterozygous for mutation in Atm , the gene involved in ataxia-telangiectasia, have heightened susceptibility to cancer

Ataxia telangiectasia mutated deficiency affects astrocyte growth but not radiosensitivity

Defective neurogenesis resulting from DNA ligase IV deficiency requires Atm

The DNA double-strand break response in the nervous system

SC1: a marker for astrocytes in the adult rodent brain is upregulated during reactive astrocytosis

Atm and Bax Cooperate in Ionizing Radiation-Induced Apoptosis in the Central Nervous System

ATM and ataxia telangiectasia: Second in Molecular Medicine Review Series

The Centrosomal, Putative Tumor Suppressor Protein TACC2 Is Dispensable for Normal Development, and Deficiency Does Not Lead to Cancer

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