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Clostridium perfringens Enterotoxin: Action, Genetics, and Translational Applications

Toxins, 2016-03, Vol.8 (3), p.73

2016

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Expansion of the Clostridium perfringens toxin-based typing scheme

Anaerobe, 2018-10, Vol.53, p.5-10

2018

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Mechanisms of Action and Cell Death Associated with Clostridium perfringens Toxins

Toxins, 2018-05, Vol.10 (5), p.212

2018

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Pathogenicity and virulence of Clostridium perfringens

Virulence, 2021-12, Vol.12 (1), p.723-753

2021

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NanJ Is the Major Sialidase for Clostridium perfringens Type F Food Poisoning Strain 01E809

Infection and immunity, 2023-06, Vol.91 (6), p.e0005323-e0005323

2023

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Identification of orphan histidine kinases that impact sporulation and enterotoxin production by Clostridium perfringens type F strain SM101 in a pathophysiologically-relevant ex vivo mouse intestinal contents model

PLoS pathogens, 2023-06, Vol.19 (6), p.e1011429-e1011429

2023

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NanI Sialidase Can Support the Growth and Survival of Clostridium perfringens Strain F4969 in the Presence of Sialyated Host Macromolecules (Mucin) or Caco-2 Cells

Infection and immunity, 2018-02, Vol.86 (2)

2018

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Clostridium perfringens Sialidases: Potential Contributors to Intestinal Pathogenesis and Therapeutic Targets

Toxins, 2016-11, Vol.8 (11), p.341

2016

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The Sialidases of Clostridium perfringens Type D Strain CN3718 Differ in Their Properties and Sensitivities to Inhibitors

Applied and Environmental Microbiology, 2014-03, Vol.80 (5), p.1701-1709

2014

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Sialidases affect the host cell adherence and epsilon toxin-induced cytotoxicity of Clostridium perfringens type D strain CN3718

PLoS pathogens, 2011-12, Vol.7 (12), p.e1002429-e1002429

2011

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NanR Regulates Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strain F4969

Infection and immunity, 2018-10, Vol.86 (10)

2018

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Evidence That VirS Is a Receptor for the Signaling Peptide of the Clostridium perfringens Agr-like Quorum Sensing System

mBio, 2020-09, Vol.11 (5)

2020

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Toxin Plasmids of Clostridium perfringens

Microbiology and Molecular Biology Reviews, 2013-06, Vol.77 (2), p.208-233

2013

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The presence of differentiated C2C12 muscle cells enhances toxin production and growth by Clostridium perfringens type A strain ATCC3624

Virulence, 2024-12, Vol.15 (1), p.2388219

2024

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Heat resistance differences are common between both vegetative cells and spores of Clostridium perfringens type F isolates carrying a chromosomal vs plasmid-borne enterotoxin gene

Applied and environmental microbiology, 2024-09

2024

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Contributions of NanI sialidase to Caco-2 cell adherence by Clostridium perfringens type A and C strains causing human intestinal disease

Infection and immunity, 2014-11, Vol.82 (11), p.4620-4630

2014

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Bile acid-independent protection against Clostridioides difficile infection

PLoS pathogens, 2021-10, Vol.17 (10), p.e1010015-e1010015

2021

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A novel small acid soluble protein variant is important for spore resistance of most Clostridium perfringens food poisoning isolates

PLoS pathogens, 2008-05, Vol.4 (5), p.e1000056

2008

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Identifying the Basis for VirS/VirR Two-Component Regulatory System Control of Clostridium perfringens Beta-Toxin Production

Journal of bacteriology, 2021-08, Vol.203 (18), p.e0027921-e0027921

2021

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Role of the Agr-Like Quorum-Sensing System in Regulating Toxin Production by Clostridium perfringens Type B Strains CN1793 and CN1795

Infection and Immunity, 2012-09, Vol.80 (9), p.3008-3017

2012

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Agr-Like Quorum-Sensing System Regulates Sporulation and Production of Enterotoxin and Beta2 Toxin by Clostridium perfringens Type A Non-Food-Borne Human Gastrointestinal Disease Strain F5603

Infection and Immunity, 2011-06, Vol.79 (6), p.2451-2459

2011

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NanH Is Produced by Sporulating Cultures of Clostridium perfringens Type F Food Poisoning Strains and Enhances the Cytotoxicity of C. perfringens Enterotoxin

mSphere, 2021-04, Vol.6 (2)

2021

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The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation

mBio, 2024-04, Vol.15 (4), p.e0224823

2024

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RIP1, RIP3, and MLKL Contribute to Cell Death Caused by Clostridium perfringens Enterotoxin

mBio, 2019-12, Vol.10 (6)

2019

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The multiplicity of thioredoxin systems meets the specific lifestyles of Clostridia

PLoS pathogens, 2024-02, Vol.20 (2), p.e1012001-e1012001

2024

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Clostridium perfringens Enterotoxin: Action, Genetics, and Translational Applications

Expansion of the Clostridium perfringens toxin-based typing scheme

Mechanisms of Action and Cell Death Associated with Clostridium perfringens Toxins

Pathogenicity and virulence of Clostridium perfringens

NanJ Is the Major Sialidase for Clostridium perfringens Type F Food Poisoning Strain 01E809

Identification of orphan histidine kinases that impact sporulation and enterotoxin production by Clostridium perfringens type F strain SM101 in a pathophysiologically-relevant ex vivo mouse intestinal contents model

NanI Sialidase Can Support the Growth and Survival of Clostridium perfringens Strain F4969 in the Presence of Sialyated Host Macromolecules (Mucin) or Caco-2 Cells

Clostridium perfringens Sialidases: Potential Contributors to Intestinal Pathogenesis and Therapeutic Targets

The Sialidases of Clostridium perfringens Type D Strain CN3718 Differ in Their Properties and Sensitivities to Inhibitors

Sialidases affect the host cell adherence and epsilon toxin-induced cytotoxicity of Clostridium perfringens type D strain CN3718

NanR Regulates Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strain F4969

Evidence That VirS Is a Receptor for the Signaling Peptide of the Clostridium perfringens Agr-like Quorum Sensing System

Toxin Plasmids of Clostridium perfringens

The presence of differentiated C2C12 muscle cells enhances toxin production and growth by Clostridium perfringens type A strain ATCC3624

Heat resistance differences are common between both vegetative cells and spores of Clostridium perfringens type F isolates carrying a chromosomal vs plasmid-borne enterotoxin gene

Contributions of NanI sialidase to Caco-2 cell adherence by Clostridium perfringens type A and C strains causing human intestinal disease

Bile acid-independent protection against Clostridioides difficile infection

A novel small acid soluble protein variant is important for spore resistance of most Clostridium perfringens food poisoning isolates

Identifying the Basis for VirS/VirR Two-Component Regulatory System Control of Clostridium perfringens Beta-Toxin Production

Role of the Agr-Like Quorum-Sensing System in Regulating Toxin Production by Clostridium perfringens Type B Strains CN1793 and CN1795

Agr-Like Quorum-Sensing System Regulates Sporulation and Production of Enterotoxin and Beta2 Toxin by Clostridium perfringens Type A Non-Food-Borne Human Gastrointestinal Disease Strain F5603

NanH Is Produced by Sporulating Cultures of Clostridium perfringens Type F Food Poisoning Strains and Enhances the Cytotoxicity of C. perfringens Enterotoxin

The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation

RIP1, RIP3, and MLKL Contribute to Cell Death Caused by Clostridium perfringens Enterotoxin

The multiplicity of thioredoxin systems meets the specific lifestyles of Clostridia

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