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Biological Properties of Vitamins of the B-Complex, Part 1: Vitamins B1, B2, B3, and B5

Nutrients, 2022-01, Vol.14 (3), p.484

2022

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Sphingosine‐1‐Phosphate Receptor 1 Regulates Cardiac Function by Modulating Ca2+ Sensitivity and Na+/H+ Exchange and Mediates Protection by Ischemic Preconditioning

Journal of the American Heart Association, 2016-05, Vol.5 (5), p.n/a

2016

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Survivin Determines Cardiac Function by Controlling Total Cardiomyocyte Number

Circulation (New York, N.Y.), 2008-03, Vol.117 (12), p.1583-1593

2008

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Overexpression of the Catalytic Subunit of Protein Phosphatase 2A Impairs Cardiac Function

The Journal of biological chemistry, 2004-09, Vol.279 (39), p.40827-40834

2004

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Upregulation of SERCA2a following short-term ACE inhibition (by enalaprilat) alters contractile performance and arrhythmogenicity of healthy myocardium in rat

Molecular and cellular biochemistry, 2015-05, Vol.403 (1-2), p.199-208

2015

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TRP Channels Expression Profile in Human End-Stage Heart Failure

Medicina (Kaunas, Lithuania), 2019-07, Vol.55 (7), p.380

2019

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Critical Role of Transcription Factor Cyclic AMP Response Element Modulator in β1-Adrenoceptor-Mediated Cardiac Dysfunction

Circulation (New York, N.Y.), 2009-01, Vol.119 (1), p.79-88

2009

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Heart-directed Expression of a Human Cardiac Isoform of cAMP-Response Element Modulator in Transgenic Mice

The Journal of biological chemistry, 2005-02, Vol.280 (8), p.6906-6914

2005

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Cardiomyocyte-specific inactivation of transcription factor CREB in mice

The FASEB journal, 2007-06, Vol.21 (8), p.1884-1892

2007

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Altered Expression of ORAI and STIM Isoforms in Activated Human Cardiac Fibroblasts

Physiological research, 2021-11, Vol.70 (Suppl 1), p.S21-S30

2021

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Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2012-05, Vol.302 (10), p.H2008-H2017

2012

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Modulation of cardiac contractility by serine/threonine protein phosphatase type 5

International journal of cardiology, 2012-01, Vol.154 (2), p.116-121

2012

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Inhibition of protein phosphatase 1 by inhibitor-2 exacerbates progression of cardiac failure in a model with pressure overload

Cardiovascular research, 2008-08, Vol.79 (3), p.464-471

2008

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The Cyclic AMP Response Element Modulator α Suppresses CD86 Expression and APC Function

The Journal of immunology (1950), 2009-04, Vol.182 (7), p.4167-4174

2009

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Inhibitor-2 prevents protein phosphatase 1-induced cardiac hypertrophy and mortality

American journal of physiology. Heart and circulatory physiology, 2008-10, Vol.295 (4), p.H1539-H1546

2008

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Enhanced cardiac function in mice overexpressing protein phosphatase Inhibitor-2

Cardiovascular research, 2005-10, Vol.68 (1), p.98-108

2005

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Knockout of ATF1 leads to enhanced cardiac contractility and output

The FASEB journal, 2008-03, Vol.22 (S1)

2008

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Impaired cardiac contraction and relaxation and decreased expression of sarcoplasmic Ca2+‐ATPase in mice lacking the CREM gene

The FASEB journal, 2003-01, Vol.17 (1), p.103-105

2003

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The cyclic AMP response element modulator α suppresses CD86 expression and APC function

The Journal of immunology (1950), 2009-07, Vol.183 (1), p.770-770

2009

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The Cyclic AMP Response Element Modulator {alpha} Suppresses CD86 Expression and APC Function

The Journal of immunology (1950), 2009-04, Vol.182 (7), p.4167-4174

2009

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The Cyclic AMP Response Element Modulator α Suppresses CD86 Expression and APC Function1

The Journal of immunology (1950), 2009-04, Vol.182 (7), p.4167-4174

2009

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Critical Role of Transcription Factor Cyclic AMP Response Element Modulator in β 1 -Adrenoceptor–Mediated Cardiac Dysfunction

Circulation (New York, N.Y.), 2009-01, Vol.119 (1), p.79-88

2009

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Inhibitor-2 prevents protein phosphatase 1-induced cardiac hypertrophy and mortality

American journal of physiology. Heart and circulatory physiology, 2008, Vol.64 (4)

2008

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Triadin is a critical determinant of cellular Ca cycling and contractility in the heart

American journal of physiology. Heart and circulatory physiology, 2007-11, Vol.293 (5), p.H3165-H3174

2007

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Biological Properties of Vitamins of the B-Complex, Part 1: Vitamins B1, B2, B3, and B5

Sphingosine‐1‐Phosphate Receptor 1 Regulates Cardiac Function by Modulating Ca2+ Sensitivity and Na+/H+ Exchange and Mediates Protection by Ischemic Preconditioning

Survivin Determines Cardiac Function by Controlling Total Cardiomyocyte Number

Overexpression of the Catalytic Subunit of Protein Phosphatase 2A Impairs Cardiac Function

Upregulation of SERCA2a following short-term ACE inhibition (by enalaprilat) alters contractile performance and arrhythmogenicity of healthy myocardium in rat

TRP Channels Expression Profile in Human End-Stage Heart Failure

Critical Role of Transcription Factor Cyclic AMP Response Element Modulator in β1-Adrenoceptor-Mediated Cardiac Dysfunction

Heart-directed Expression of a Human Cardiac Isoform of cAMP-Response Element Modulator in Transgenic Mice

Cardiomyocyte-specific inactivation of transcription factor CREB in mice

Altered Expression of ORAI and STIM Isoforms in Activated Human Cardiac Fibroblasts

Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytes

Modulation of cardiac contractility by serine/threonine protein phosphatase type 5

Inhibition of protein phosphatase 1 by inhibitor-2 exacerbates progression of cardiac failure in a model with pressure overload

The Cyclic AMP Response Element Modulator α Suppresses CD86 Expression and APC Function

Inhibitor-2 prevents protein phosphatase 1-induced cardiac hypertrophy and mortality

Enhanced cardiac function in mice overexpressing protein phosphatase Inhibitor-2

Sphingosine‐1‐Phosphate Receptor 1 Regulates Cardiac Function by Modulating Ca 2+ Sensitivity and Na + /H + Exchange and Mediates Protection by Ischemic Preconditioning

Knockout of ATF1 leads to enhanced cardiac contractility and output

Impaired cardiac contraction and relaxation and decreased expression of sarcoplasmic Ca2+‐ATPase in mice lacking the CREM gene

The cyclic AMP response element modulator α suppresses CD86 expression and APC function

The Cyclic AMP Response Element Modulator {alpha} Suppresses CD86 Expression and APC Function

The Cyclic AMP Response Element Modulator α Suppresses CD86 Expression and APC Function1

Critical Role of Transcription Factor Cyclic AMP Response Element Modulator in β 1 -Adrenoceptor–Mediated Cardiac Dysfunction

Inhibitor-2 prevents protein phosphatase 1-induced cardiac hypertrophy and mortality

Triadin is a critical determinant of cellular Ca cycling and contractility in the heart

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