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Lipopolysaccharide‐primed heterotolerant dendritic cells suppress experimental autoimmune uveoretinitis by multiple mechanisms

Immunology, 2017-03, Vol.150 (3), p.364-377

2017

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Ppm1E is an in cellulo AMP-activated protein kinase phosphatase

Cellular signalling, 2011, Vol.23 (1), p.114-124

2011

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Targeted delivery of autoantigen to dendritic cells prevents development of spontaneous uveitis

Frontiers in immunology, 2023-09, Vol.14, p.1227633-1227633

2023

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Myeloid-Cell Protein Tyrosine Phosphatase-1B Deficiency in Mice Protects Against High-Fat Diet and Lipopolysaccharide-Induced Inflammation, Hyperinsulinemia, and Endotoxemia Through an IL-10 STAT3-Dependent Mechanism

Diabetes (New York, N.Y.), 2014-02, Vol.63 (2), p.456-470

2014

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A role for PP1/NIPP1 in steering migration of human cancer cells

PloS one, 2012-07, Vol.7 (7), p.e40769-e40769

2012

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Depletion of protein phosphatase 4 in human cells reveals essential roles in centrosome maturation, cell migration and the regulation of Rho GTPases

The international journal of biochemistry & cell biology, 2008, Vol.40 (10), p.2315-2332

2008

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Recurrent deletions of ULK4 in schizophrenia: a novel gene crucial for neuritogenesis and neuronal motility

Journal of cell science, 2013-01

2013

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Absence of See1p, a widely conserved Saccharomyces cerevisiae protein, confers both deficient heterologous protein production and endocytosis

Yeast (Chichester, England), 2008-12, Vol.25 (12), p.871-877

2008

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Recurrent deletions of ULK4 in schizophrenia: a gene crucial for neuritogenesis and neuronal motility

Journal of cell science, 2014-02, Vol.127 (Pt 3), p.630-640

2014

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Harnessing the Electric Spark of Life to Cure Skin Wounds

Advances in wound care (New Rochelle, N.Y.), 2014-02, Vol.3 (2), p.127-138

2014

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Deletion of myeloid-PTP1B decreases MHC Class I expression and peptide presentation through an IL-10 dependent mechanism in response to LPS challenge

Journal of inflammation (London, England), 2015-04, Vol.12 (Suppl 1), p.P1-P1

2015

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A key role for PTP1B in dendritic cell maturation, migration, and T cell activation

Journal of molecular cell biology, 2015-12, Vol.7 (6), p.517-528

2015

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Protein phosphatase 4 is phosphorylated and inactivated by Cdk in response to spindle toxins and interacts with γ-tubulin

Cell cycle (Georgetown, Tex.), 2013-09, Vol.12 (17), p.2876-2887

2013

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Cell-based therapies for ocular inflammation

Progress in retinal and eye research, 2013-07, Vol.35, p.82-101

2013

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Erratum to “Ppm1E is an in cellulo AMP-activated protein kinase phosphatase” [Cell. Signalling 23 (2011) 114–124]

Cellular signalling, 2011-05, Vol.23 (5), p.947-947

2011

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Lipopolysaccharide‐primed heterotolerant dendritic cells suppress experimental autoimmune uveoretinitis by multiple mechanisms

Ppm1E is an in cellulo AMP-activated protein kinase phosphatase

Targeted delivery of autoantigen to dendritic cells prevents development of spontaneous uveitis

Myeloid-Cell Protein Tyrosine Phosphatase-1B Deficiency in Mice Protects Against High-Fat Diet and Lipopolysaccharide-Induced Inflammation, Hyperinsulinemia, and Endotoxemia Through an IL-10 STAT3-Dependent Mechanism

A role for PP1/NIPP1 in steering migration of human cancer cells

Depletion of protein phosphatase 4 in human cells reveals essential roles in centrosome maturation, cell migration and the regulation of Rho GTPases

Recurrent deletions of ULK4 in schizophrenia: a novel gene crucial for neuritogenesis and neuronal motility

Absence of See1p, a widely conserved Saccharomyces cerevisiae protein, confers both deficient heterologous protein production and endocytosis

Recurrent deletions of ULK4 in schizophrenia: a gene crucial for neuritogenesis and neuronal motility

Harnessing the Electric Spark of Life to Cure Skin Wounds

Deletion of myeloid-PTP1B decreases MHC Class I expression and peptide presentation through an IL-10 dependent mechanism in response to LPS challenge

A key role for PTP1B in dendritic cell maturation, migration, and T cell activation

Protein phosphatase 4 is phosphorylated and inactivated by Cdk in response to spindle toxins and interacts with γ-tubulin

Cell-based therapies for ocular inflammation

Erratum to “Ppm1E is an in cellulo AMP-activated protein kinase phosphatase” [Cell. Signalling 23 (2011) 114–124]

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