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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

International journal of molecular sciences, 2020-02, Vol.21 (3), p.977

2020

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The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

Molecular medicine (Cambridge, Mass.), 2020-06, Vol.26 (1), p.63-63, Article 63

2020

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A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

Molecules (Basel, Switzerland), 2023-09, Vol.28 (18), p.6560

2023

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.1-98

2020

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Carbon monoxide provides protection against hyperoxic lung injury

American journal of physiology. Lung cellular and molecular physiology, 1999-04, Vol.276 (4), p.688-L694

1999

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Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-08, Vol.27 (1), p.93-93, Article 93

2021

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HMGB-1 as a therapeutic target for infectious and inflammatory disorders

Shock (Augusta, Ga.), 2006, Vol.25 (1), p.4-11

2006

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From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Journal of immunotoxicology, 2021-01, Vol.18 (1), p.23-29

2021

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Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury

The Journal of clinical investigation, 1999-04, Vol.103 (7), p.1047-1054

1999

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2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-07, Vol.27 (1), p.79-79, Article 79

2021

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Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.132-132, Article 132

2020

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Hyperoxia sensing: From molecular mechanisms to significance in disease

Journal of immunotoxicology, 2010-12, Vol.7 (4), p.239-254

2010

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Inhibition of extracellular HMGB1 attenuates hyperoxia-induced inflammatory acute lung injury

Redox biology, 2014-01, Vol.2 (C), p.314-322

2014

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Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-09, Vol.27 (1), p.98-98, Article 98

2021

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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-02, Vol.26 (1), p.16-16, Article 16

2020

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2014-06, Vol.20 (1), p.238-247

2014

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Interleukin-6-Induced Protection in Hyperoxic Acute Lung Injury

American journal of respiratory cell and molecular biology, 2000-05, Vol.22 (5), p.535-542

2000

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Our Fragile Water Planet: An Introduction to the Earth Sciences

Mantell, C

1, 1976

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The Positive Allosteric Modulation of alpha7-Nicotinic Cholinergic Receptors by GAT107 Increases Bacterial Lung Clearance in Hyperoxic Mice by Decreasing Oxidative Stress in Macrophages

Antioxidants, 2021-01, Vol.10 (1), p.135

2021

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Signal Transduction Pathways in Hyperoxia-Induced Lung Cell Death

Molecular Genetics and Metabolism, 2000-09, Vol.71 (1-2), p.359-370

2000

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Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Toxicological sciences, 2019-12, Vol.172 (2), p.398-410

2019

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MAPK pathways mediate hyperoxia-induced oncotic cell death in lung epithelial cells

Free radical biology & medicine, 2003-10, Vol.35 (8), p.978-993

2003

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Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

Free radical biology & medicine, 2022-09, Vol.190, p.247-263

2022

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Antioxidants preserve macrophage phagocytosis of Pseudomonas aeruginosa during hyperoxia

Free radical biology & medicine, 2007-05, Vol.42 (9), p.1338-1349

2007

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Mitogen-activated protein kinase pathway mediates hyperoxia-induced apoptosis in cultured macrophage cells

American journal of physiology. Lung cellular and molecular physiology, 1999-09, Vol.277 (3), p.589-L595

1999

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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Carbon monoxide provides protection against hyperoxic lung injury

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

HMGB-1 as a therapeutic target for infectious and inflammatory disorders

From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury

2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Hyperoxia sensing: From molecular mechanisms to significance in disease

Inhibition of extracellular HMGB1 attenuates hyperoxia-induced inflammatory acute lung injury

Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Interleukin-6-Induced Protection in Hyperoxic Acute Lung Injury

Our Fragile Water Planet: An Introduction to the Earth Sciences

The Positive Allosteric Modulation of alpha7-Nicotinic Cholinergic Receptors by GAT107 Increases Bacterial Lung Clearance in Hyperoxic Mice by Decreasing Oxidative Stress in Macrophages

Signal Transduction Pathways in Hyperoxia-Induced Lung Cell Death

Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

MAPK pathways mediate hyperoxia-induced oncotic cell death in lung epithelial cells

Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

Antioxidants preserve macrophage phagocytosis of Pseudomonas aeruginosa during hyperoxia

Mitogen-activated protein kinase pathway mediates hyperoxia-induced apoptosis in cultured macrophage cells

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